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ke00092  (Proteintech)


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    Proteintech ke00092
    Ke00092, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 2 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/ke00092/product/Proteintech
    Average 93 stars, based on 2 article reviews
    ke00092 - by Bioz Stars, 2026-03
    93/100 stars

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    ke00092  (Proteintech)
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    Fig. 3 Macrophage-mediated inflammation in myocardium near the fibro-fatty tissues of ACM patients. A The mRNA expression of <t>CCL3</t> in RV myocardium from ACM patients and NC; (B) The IHC staining of CCL3 in RV myocardium from ACM patients and NC; (C) Opal multicolor IHC staining of CCL3, CD68, and DAPI in RV myocardium from ACM patients and NC; (D) The correlation between the mRNA level of CCL3 in RV myocardium and the level of saturated TGs in RV fibro-fatty tissue from ACM patients; (E) The correlation between the mRNA level of CCL3 and duration from initial symptom to HTx; (F) The correlation between the mRNA level of CCL3 and duration from heart failure to HTx. RV, right ventricle; ACM, arrhythmogenic cardiomyopathy; NC, normal control; HTx, heart transplantation
    Ccl3, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    ccl3 ke00092  (Proteintech)
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    Proteintech ccl3 ke00092
    BYF inhibits CSE-induced cellular senescence. The protein levels of senescence markers p16, p21 and p27 were detected by Western blot ( A and B ). The secretion of IL-6 ( C ), IL-8 ( D ), and <t>CCL3</t> ( E ) into the medium was measured by ELISA. *Compared into NC group, P<0.05; **Compared into NC group, P<0.01; ***Compared into NC group, P<0.001; # Compared into CSE group, P<0.05; ## Compared into CSE group, P<0.01.
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    Fig. 3 Macrophage-mediated inflammation in myocardium near the fibro-fatty tissues of ACM patients. A The mRNA expression of CCL3 in RV myocardium from ACM patients and NC; (B) The IHC staining of CCL3 in RV myocardium from ACM patients and NC; (C) Opal multicolor IHC staining of CCL3, CD68, and DAPI in RV myocardium from ACM patients and NC; (D) The correlation between the mRNA level of CCL3 in RV myocardium and the level of saturated TGs in RV fibro-fatty tissue from ACM patients; (E) The correlation between the mRNA level of CCL3 and duration from initial symptom to HTx; (F) The correlation between the mRNA level of CCL3 and duration from heart failure to HTx. RV, right ventricle; ACM, arrhythmogenic cardiomyopathy; NC, normal control; HTx, heart transplantation

    Journal: BMC medicine

    Article Title: Plasma CCL3 predicts adverse heart failure outcomes in patients with arrhythmogenic cardiomyopathy.

    doi: 10.1186/s12916-025-04024-y

    Figure Lengend Snippet: Fig. 3 Macrophage-mediated inflammation in myocardium near the fibro-fatty tissues of ACM patients. A The mRNA expression of CCL3 in RV myocardium from ACM patients and NC; (B) The IHC staining of CCL3 in RV myocardium from ACM patients and NC; (C) Opal multicolor IHC staining of CCL3, CD68, and DAPI in RV myocardium from ACM patients and NC; (D) The correlation between the mRNA level of CCL3 in RV myocardium and the level of saturated TGs in RV fibro-fatty tissue from ACM patients; (E) The correlation between the mRNA level of CCL3 and duration from initial symptom to HTx; (F) The correlation between the mRNA level of CCL3 and duration from heart failure to HTx. RV, right ventricle; ACM, arrhythmogenic cardiomyopathy; NC, normal control; HTx, heart transplantation

    Article Snippet: The concentrations of CCL3 in plasma were quantified using a commercial kit (Proteintech, KE00092).

    Techniques: Expressing, Immunohistochemistry, Control, Transplantation Assay

    BYF inhibits CSE-induced cellular senescence. The protein levels of senescence markers p16, p21 and p27 were detected by Western blot ( A and B ). The secretion of IL-6 ( C ), IL-8 ( D ), and CCL3 ( E ) into the medium was measured by ELISA. *Compared into NC group, P<0.05; **Compared into NC group, P<0.01; ***Compared into NC group, P<0.001; # Compared into CSE group, P<0.05; ## Compared into CSE group, P<0.01.

    Journal: International Journal of Chronic Obstructive Pulmonary Disease

    Article Title: Bufei Yishen Formula Inhibits the Cell Senescence in COPD by Up-Regulating the ZNF263 and Klotho Expression

    doi: 10.2147/COPD.S383295

    Figure Lengend Snippet: BYF inhibits CSE-induced cellular senescence. The protein levels of senescence markers p16, p21 and p27 were detected by Western blot ( A and B ). The secretion of IL-6 ( C ), IL-8 ( D ), and CCL3 ( E ) into the medium was measured by ELISA. *Compared into NC group, P<0.05; **Compared into NC group, P<0.01; ***Compared into NC group, P<0.001; # Compared into CSE group, P<0.05; ## Compared into CSE group, P<0.01.

    Article Snippet: Concentrations of IL-6 (ab178013, Abcam), IL-8 (b214030, Abcam) and CCL3 (KE00092, Proteintech) in the culture medium were measured using Human ELISA Kits according to the manufacturer’s recommendations.

    Techniques: Western Blot, Enzyme-linked Immunosorbent Assay

    BYF activates klotho transcription by upregulating the expression of transcription factor ZNF263. ZNF263 binding klotho promoter sequence with JASPAR and USCS databases ( A ). The protein level of ZNF263 in BEAS-2B cells with CSE exposure and BYF treatment was detected by Western blot ( B and C ). The protein levels of ZNF263, klotho and senescence markers in BEAS-2B cells with CSE exposure and ZNF263-expression plasmid transfection were detected by Western blot ( D – F ). The secretion of IL-6 ( G ), IL-8 ( H ), and CCL3 ( I ) into the medium was measured by ELISA. *Compared into NC group, P<0.05; **Compared into NC group, P<0.01; ***Compared into NC group, P<0.001; # Compared into CSE group, P<0.05; ## Compared into CSE group, P<0.01; ### Compared into CSE group, P<0.001.

    Journal: International Journal of Chronic Obstructive Pulmonary Disease

    Article Title: Bufei Yishen Formula Inhibits the Cell Senescence in COPD by Up-Regulating the ZNF263 and Klotho Expression

    doi: 10.2147/COPD.S383295

    Figure Lengend Snippet: BYF activates klotho transcription by upregulating the expression of transcription factor ZNF263. ZNF263 binding klotho promoter sequence with JASPAR and USCS databases ( A ). The protein level of ZNF263 in BEAS-2B cells with CSE exposure and BYF treatment was detected by Western blot ( B and C ). The protein levels of ZNF263, klotho and senescence markers in BEAS-2B cells with CSE exposure and ZNF263-expression plasmid transfection were detected by Western blot ( D – F ). The secretion of IL-6 ( G ), IL-8 ( H ), and CCL3 ( I ) into the medium was measured by ELISA. *Compared into NC group, P<0.05; **Compared into NC group, P<0.01; ***Compared into NC group, P<0.001; # Compared into CSE group, P<0.05; ## Compared into CSE group, P<0.01; ### Compared into CSE group, P<0.001.

    Article Snippet: Concentrations of IL-6 (ab178013, Abcam), IL-8 (b214030, Abcam) and CCL3 (KE00092, Proteintech) in the culture medium were measured using Human ELISA Kits according to the manufacturer’s recommendations.

    Techniques: Expressing, Binding Assay, Sequencing, Western Blot, Plasmid Preparation, Transfection, Enzyme-linked Immunosorbent Assay