micromaxtm: human cdna microarray system i (NEN Life Science)
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NEN Life Science
micromaxtm: human cdna microarray system i
Micromaxtm: Human Cdna Microarray System I, supplied by NEN Life Science, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/human+cdna+microarray/human+cdna+microarray+system+i/pmc00116586-484-6-14
Average 90 stars, based on 1 article reviews
Micromaxtm: Human Cdna Microarray System I, supplied by NEN Life Science, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/human+cdna+microarray/human+cdna+microarray+system+i/pmc00116586-484-6-14
Average 90 stars, based on 1 article reviews
micromaxtm: human cdna microarray system i - by Bioz Stars,
2026-09
90/100 stars
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Microarray:Article Title: Unique dietary-related mouse model of colitis Article Snippet: Results: Mice fed the DOC-supplemented diet developed focal areas of colonic inflammation associated with increases in angiogenesis, nitrosative stress, DNA/RNA damage, and proliferation.. Genes that play a central role in inflammation and angiogenesis and other related processes such as epithelial barrier function, oxidative stress, apoptosis, cell proliferation/cell cycle/DNA repair, membrane transport, and the ubiquitin-proteasome pathway showed altered expression in the DOC-fed mice compared with the control mice.. Changes in expression of individual genes (increases or reductions) correlated over time. Article Title: The chemopreventive agent alpha-difluoromethylornithine blocks Ki-ras-dependent tumor formation and specific gene expression in Caco-2 cells. Article Snippet: Natalia A. Ignatenko, Hui Zhang, George S. Watts, Bethany A. Skovan, David E. Stringer, and Eugene W. Gerner* Department of Cell Biology and Anatomy, Arizona Cancer Center, The University of Arizona, Tucson, Arizona Cancer Biology Division, Arizona Cancer Center, The University of Arizona, Tucson, Arizona Department of Pharmacology, College of Medicine, Arizona Cancer Center, The University of Arizona, Tucson, Arizona Department of Biochemistry and Molecular Biophysics, Arizona Cancer Center, The University of Arizona, Tucson, Arizona Article Title: Mutant p53 and aberrant cytosine methylation cooperate to silence gene expression. Article Snippet: .. Slides were then washed in 1% SDS for 2min, rinsed in double-distilled water three times, spun dry, and stored at room temperature (RT) in a desiccator until use. cDNA microarray analysis Total RNA (40 mg) was converted to fluorescent first-strand cDNA using the Micromax Article Title: Deoxycholate-induced colitis is markedly attenuated in Nos2 knockout mice in association with modulation of gene expression profiles. Article Snippet: Nos2 knockout mice were compared to wild-type mice for susceptibility to colitis in response to a diet supplemented with deoxycholate, a bile acid increased in the colon of individuals on a high-fat diet.. Wild-type mice fed a fat-related diet, supplemented with 0.2% DOC, develop colonic inflammation associated with increases in nitrosative stress, proliferation, oxidative DNA/RNA damage, and angiogenesis, as well as altered expression of numerous genes.. However, Nos2 knockout mice fed a diet supplemented with deoxycholate were resistant to these alterations. Article Title: Clinical applications of microarray technology: creatine kinase B is an up-regulated gene in epithelial ovarian cancer and shows promise as a serum marker. Article Snippet: Objective. (1) To identify and (2) validate genes that are up-regulated in ovarian cancer, and (3) to investigate whether the activity of a candidate gene, creatine kinase B (CKB) is elevated in pre-operative sera from ovarian cancer patients compared to patients with benign pelvic masses and normal controls.. Methods.. MICROMAX cDNA microarray system and RNA derived from pooled ovarian cancer cell lines and normal ovary surface epithelial cells (HOSE) were used to identify differentially expressed genes. Membrane:Article Title: Biochemical assessment of intracellular signal transduction pathways in eosinophils: implications for pharmacotherapy. Article Snippet: Allergic asthma and allergic rhinitis are inflammatory diseases of the airway.. Cytokines and chemokines produced by T helper (Th) type 2 cells (GM-CSF, IL-4, IL-5, IL-6, IL-9, IL-10 and IL-13), eotaxin, transforming growth factor-β, and IL-11 orchestrate most pathophysiological processes of the late-phase allergic reaction, including the recruitment, activation, and delayed apoptosis of eosinophils, as well as eosinophilic degranulation to release eosinophilic cationic protein, major basic protein, and eosinophil-derived neurotoxin.. These processes are regulated through an extensive network of interactive intracellular signal transduction pathways that have been intensively investigated recently. Expressing:Article Title: Biochemical assessment of intracellular signal transduction pathways in eosinophils: implications for pharmacotherapy. Article Snippet: Allergic asthma and allergic rhinitis are inflammatory diseases of the airway.. Cytokines and chemokines produced by T helper (Th) type 2 cells (GM-CSF, IL-4, IL-5, IL-6, IL-9, IL-10 and IL-13), eotaxin, transforming growth factor-β, and IL-11 orchestrate most pathophysiological processes of the late-phase allergic reaction, including the recruitment, activation, and delayed apoptosis of eosinophils, as well as eosinophilic degranulation to release eosinophilic cationic protein, major basic protein, and eosinophil-derived neurotoxin.. These processes are regulated through an extensive network of interactive intracellular signal transduction pathways that have been intensively investigated recently. RNA Extraction:Article Title: Biochemical assessment of intracellular signal transduction pathways in eosinophils: implications for pharmacotherapy. Article Snippet: Allergic asthma and allergic rhinitis are inflammatory diseases of the airway.. Cytokines and chemokines produced by T helper (Th) type 2 cells (GM-CSF, IL-4, IL-5, IL-6, IL-9, IL-10 and IL-13), eotaxin, transforming growth factor-β, and IL-11 orchestrate most pathophysiological processes of the late-phase allergic reaction, including the recruitment, activation, and delayed apoptosis of eosinophils, as well as eosinophilic degranulation to release eosinophilic cationic protein, major basic protein, and eosinophil-derived neurotoxin.. These processes are regulated through an extensive network of interactive intracellular signal transduction pathways that have been intensively investigated recently. Reverse Transcription:Article Title: Biochemical assessment of intracellular signal transduction pathways in eosinophils: implications for pharmacotherapy. Article Snippet: Allergic asthma and allergic rhinitis are inflammatory diseases of the airway.. Cytokines and chemokines produced by T helper (Th) type 2 cells (GM-CSF, IL-4, IL-5, IL-6, IL-9, IL-10 and IL-13), eotaxin, transforming growth factor-β, and IL-11 orchestrate most pathophysiological processes of the late-phase allergic reaction, including the recruitment, activation, and delayed apoptosis of eosinophils, as well as eosinophilic degranulation to release eosinophilic cationic protein, major basic protein, and eosinophil-derived neurotoxin.. These processes are regulated through an extensive network of interactive intracellular signal transduction pathways that have been intensively investigated recently. Hybridization:Article Title: Biochemical assessment of intracellular signal transduction pathways in eosinophils: implications for pharmacotherapy. Article Snippet: Allergic asthma and allergic rhinitis are inflammatory diseases of the airway.. Cytokines and chemokines produced by T helper (Th) type 2 cells (GM-CSF, IL-4, IL-5, IL-6, IL-9, IL-10 and IL-13), eotaxin, transforming growth factor-β, and IL-11 orchestrate most pathophysiological processes of the late-phase allergic reaction, including the recruitment, activation, and delayed apoptosis of eosinophils, as well as eosinophilic degranulation to release eosinophilic cationic protein, major basic protein, and eosinophil-derived neurotoxin.. These processes are regulated through an extensive network of interactive intracellular signal transduction pathways that have been intensively investigated recently. Fluorescence:Article Title: Biochemical assessment of intracellular signal transduction pathways in eosinophils: implications for pharmacotherapy. Article Snippet: Allergic asthma and allergic rhinitis are inflammatory diseases of the airway.. Cytokines and chemokines produced by T helper (Th) type 2 cells (GM-CSF, IL-4, IL-5, IL-6, IL-9, IL-10 and IL-13), eotaxin, transforming growth factor-β, and IL-11 orchestrate most pathophysiological processes of the late-phase allergic reaction, including the recruitment, activation, and delayed apoptosis of eosinophils, as well as eosinophilic degranulation to release eosinophilic cationic protein, major basic protein, and eosinophil-derived neurotoxin.. These processes are regulated through an extensive network of interactive intracellular signal transduction pathways that have been intensively investigated recently. Control:Article Title: Biochemical assessment of intracellular signal transduction pathways in eosinophils: implications for pharmacotherapy. Article Snippet: Allergic asthma and allergic rhinitis are inflammatory diseases of the airway.. Cytokines and chemokines produced by T helper (Th) type 2 cells (GM-CSF, IL-4, IL-5, IL-6, IL-9, IL-10 and IL-13), eotaxin, transforming growth factor-β, and IL-11 orchestrate most pathophysiological processes of the late-phase allergic reaction, including the recruitment, activation, and delayed apoptosis of eosinophils, as well as eosinophilic degranulation to release eosinophilic cationic protein, major basic protein, and eosinophil-derived neurotoxin.. These processes are regulated through an extensive network of interactive intracellular signal transduction pathways that have been intensively investigated recently. other:Article Title: Glucocorticoid regulation of human eosinophil gene expression. Article Snippet: Molecular analysis of steroid-regulated gene expression in freshly isolated human eosinophils is difficult due to the inherent high rate of spontaneous apoptosis and elevated levels of endogenous ribonucleases.. To circumvent these limitations, we determined if the human eosinophilic cell line EoL-1 could serve as an in vitro model of glucocorticoid signaling.. We found by optimizing growth conditions in low serum-containing media that dexamethasone (Dex) treatment of EoL-1 cells induced an apoptotic pathway that was inhibited by interleukin-5 (IL-5). |
