empty vector (nc-oe (Obio Technology Corp Ltd)
90
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Obio Technology Corp Ltd
empty vector (nc-oe
Empty Vector (Nc Oe, supplied by Obio Technology Corp Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/empty+vector+(nc-oe/empty+vector++lv+control/pmc09309501-70-9-14
Average 90 stars, based on 1 article reviews
Empty Vector (Nc Oe, supplied by Obio Technology Corp Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/empty+vector+(nc-oe/empty+vector++lv+control/pmc09309501-70-9-14
Average 90 stars, based on 1 article reviews
empty vector (nc-oe - by Bioz Stars,
2026-09
90/100 stars
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Infection:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. shRNA:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Negative Control:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Over Expression:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Plasmid Preparation:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Construct:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Produced:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Binding Assay:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Transfection:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Quantitative RT-PCR:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Reverse Transcription Polymerase Chain Reaction:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Luciferase:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Reporter Assay:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Expressing:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Real-time Polymerase Chain Reaction:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Mutagenesis:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Enzyme-linked Immunosorbent Assay:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Flow Cytometry:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. Western Blot:Article Title: Restoration of p53 acetylation by HDAC inhibition permits the necrosis/apoptosis switch of pancreatic ainar cell during experimental pancreatitis in mice. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 81870437 Abstract The severity of acute pancreatitis (AP) is greatly attributed to the pancreatic acinar cell (PAC) death response.. It has been established that the apoptosis‐inducing therapy can protect against experimental pancreatitis and have great clinical therapeutic potential.. However, current pharmacologic agents that target apoptosis during AP largely lack specificity. Article Title: Enterotoxigenic Bacteroidesfragilis Promotes Intestinal Inflammation and Malignancy by Inhibiting Exosome-Packaged miR-149-3p. Article Snippet: State Key Laboratory for Oncogenes and Related Genes; Key Laboratory of Gastroenterology and Hepatology, Ministry of Health; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastrointestinal Surgery, Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Department of Gastroenterology and Guangzhou Key Laboratory of Digestive Disease, Guangzhou Digestive Disease Center, Guangzhou First People’s Hospital, Guangzhou Medical University, Guangzhou, China; and State Key Laboratory for Oncogenes and Related Genes; Shanghai Cancer Institute; Ren Ji Hospital, School of Medicine, Shanghai Jiao Tong University Article Title: CEACAM5 targeted by miR-498 promotes cell proliferation, migration and epithelial to mesenchymal transition in gastric cancer Article Snippet: CEACAM5 overexpression (OE) plasmid (CEACAM5-OE) and its corresponding empty vector (NC-OE) were purchased from OBIO Technology (Shanghai, China). Article Title: ZNF692 promotes osteosarcoma cell proliferation, migration, and invasion through TNK2-mediated activation of the MEK/ERK pathway. Article Snippet: For Article Title: Knockdown of lncRNA TTTY15 alleviates ischemia/reperfusion-induced inflammation and apoptosis of PC12 cells by targeting miR-766-5p Article Snippet: The TTTY15 overexpression plasmid (pcDNA3.1-TTTY15) and empty vector (pcDNA-NC) were produced by OBiO Technology (Shanghai) Corp., Ltd. miR-766-5p mimics and miR-NC were purchased from Guangzhou RiboBio Co., Ltd. |