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189 ythdf2  (Proteintech)


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    Proteintech 189 ythdf2
    189 Ythdf2, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 470 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/ythdf2/YTHDF2+Antibody/pm41917949-100-13-16
    Average 96 stars, based on 470 article reviews
    189 ythdf2 - by Bioz Stars, 2026-09
    96/100 stars

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    other:

    Article Title: A novel METTL3 inhibitor Nimbolide ameliorates osteoporosis via orchestrating osteoclastogenesis in an m6A-dependent manner
    Article Snippet: Please cite this article as: Chenhui Cai , Zaoqing Zhang , Xu Zhao , Chuan Yang , Xianming Huang , Chao Tang , Hao Qiu , Sizhen Yang , Ying Zhang , Xu Hu , Rui Zuo , Chao Zhang , Yueqi Chen , Tongwei Chu , A novel METTL3 inhibitor Nimbolide ameliorates osteoporosis via orchestrating osteoclastogenesis in an m6A-dependent manner, Phytomedicine (2026), doi: https://doi.org/10.1016/j.phymed.2026.158048

    Article Title: Zinc finger protein ZNF33B facilitates Japanese encephalitis virus replication by regulating m 6 A modification on Ifih1 and Irf3 transcripts to antagonize antiviral response.
    Article Snippet: The antibodies probed by GAPDH (Cat. # 60004-1-Ig), IgG (Cat. # 30000-0-AP), FLAG (Cat. # 20543-1-AP), HA (Cat. # 51064-2-AP, 66006-2-Ig), RIG-I (Cat. # 20566-1-AP), MDA5 (Cat. # 21775-1-AP), TBK1 (Cat. # 283971-1-AP), METTL14 (Cat. # 26158-1-AP), YTHDC1 (Cat. # 14392-1-AP), YTHDF2 (Cat. # 24744-1- AP) and m6A (Cat. # 68055-1-Ig) were obtained from Proteintech (USA).

    Article Title: Zinc finger protein ZNF33B facilitates Japanese encephalitis virus replication by regulating m 6 A modification on Ifih1 and Irf3 transcripts to antagonize antiviral response
    Article Snippet: The antibodies probed by GAPDH (Cat. # 60004-1-Ig), IgG (Cat. # 30000-0-AP), FLAG (Cat. # 20543-1-AP), HA (Cat. # 51064-2-AP, 66006-2-Ig), RIG-I (Cat. # 20566-1-AP), MDA5 (Cat. # 21775-1-AP), TBK1 (Cat. # 283971-1-AP), METTL14 (Cat. # 26158-1-AP), YTHDC1 (Cat. # 14392-1-AP), YTHDF2 (Cat. # 24744-1-AP) and m6A (Cat. # 68055-1-Ig) were obtained from Proteintech (USA).

    Incubation:

    Article Title: Bisphenol A disrupts circadian locomotor rhythms via m 6 A-dependent nr1d1 destabilization in zebrafish larvae.
    Article Snippet: .. Then incubated overnight at 4 ◦C with primary antibodies against Mettl14 (ab220030, Abcam, NW, U.S.), Mettl3 (ab195352, Abcam), Fto (ab124892, Abcam), Alkbh5 (ab195377, Abcam), Ythdf2 (24744–1-AP, Proteintech, Wuhan, China) and β-actin (66009–1-Ig, Proteintech). .. After washing with TBST, membranes were incubated with HRP-conjugated secondary antibody (SA00001–2, Proteintech) for 1 h at room temperature.



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    Role of m 6 A in adipogenesis. Insufficient adipogenesis in adipose tissue leads to persistent, chronic inflammation. m 6 A modification plays a crucial role in all stages of adipogenesis, from commitment to terminal differentiation. During commitment, METTL3 promotes lipogenic differentiation in BMSCs by regulating the m 6 A levels of PTH1R and JAK1, whereas silencing METTL14 reduces the expression of SMAD1, inhibiting BMSC proliferation. During terminal differentiation, m 6 A regulates MCE and the transition to mature adipocytes. FTO influences key genes such as ATG5, ATG7 and JAK2, affecting autophagy, STAT3 phosphorylation and adipogenesis. FTO knockout increases the m 6 A levels of CCND1 and CDK2, blocking MCE. m 6 A, N6-methyladenine; METTL, methyltransferase-like; PTH1R, parathyroid hormone 1 receptor; JAK, Janus kinase; BMSC, bone marrow mesenchymal stem cell; MCE, mitotic clone amplification; FTO, Fat mass and obesity-associated protein; ATG, autophagy-related; STAT3, signal transducer and activator of transcription 3; CCND1, cyclin D1; CDK2, cyclin-dependent kinase 2; IGF2BP1, insulin-like growth factor 2 mRNA-binding protein 1; <t>YTHDF2,</t> YTH domain family 2.
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    Role of m 6 A in adipogenesis. Insufficient adipogenesis in adipose tissue leads to persistent, chronic inflammation. m 6 A modification plays a crucial role in all stages of adipogenesis, from commitment to terminal differentiation. During commitment, METTL3 promotes lipogenic differentiation in BMSCs by regulating the m 6 A levels of PTH1R and JAK1, whereas silencing METTL14 reduces the expression of SMAD1, inhibiting BMSC proliferation. During terminal differentiation, m 6 A regulates MCE and the transition to mature adipocytes. FTO influences key genes such as ATG5, ATG7 and JAK2, affecting autophagy, STAT3 phosphorylation and adipogenesis. FTO knockout increases the m 6 A levels of CCND1 and CDK2, blocking MCE. m 6 A, N6-methyladenine; METTL, methyltransferase-like; PTH1R, parathyroid hormone 1 receptor; JAK, Janus kinase; BMSC, bone marrow mesenchymal stem cell; MCE, mitotic clone amplification; FTO, Fat mass and obesity-associated protein; ATG, autophagy-related; STAT3, signal transducer and activator of transcription 3; CCND1, cyclin D1; CDK2, cyclin-dependent kinase 2; IGF2BP1, insulin-like growth factor 2 mRNA-binding protein 1; YTHDF2, YTH domain family 2.

    Journal: International Journal of Molecular Medicine

    Article Title: m 6 A in adipose tissue inflammation: A novel regulator of obesity and metabolic diseases (Review)

    doi: 10.3892/ijmm.2026.5795

    Figure Lengend Snippet: Role of m 6 A in adipogenesis. Insufficient adipogenesis in adipose tissue leads to persistent, chronic inflammation. m 6 A modification plays a crucial role in all stages of adipogenesis, from commitment to terminal differentiation. During commitment, METTL3 promotes lipogenic differentiation in BMSCs by regulating the m 6 A levels of PTH1R and JAK1, whereas silencing METTL14 reduces the expression of SMAD1, inhibiting BMSC proliferation. During terminal differentiation, m 6 A regulates MCE and the transition to mature adipocytes. FTO influences key genes such as ATG5, ATG7 and JAK2, affecting autophagy, STAT3 phosphorylation and adipogenesis. FTO knockout increases the m 6 A levels of CCND1 and CDK2, blocking MCE. m 6 A, N6-methyladenine; METTL, methyltransferase-like; PTH1R, parathyroid hormone 1 receptor; JAK, Janus kinase; BMSC, bone marrow mesenchymal stem cell; MCE, mitotic clone amplification; FTO, Fat mass and obesity-associated protein; ATG, autophagy-related; STAT3, signal transducer and activator of transcription 3; CCND1, cyclin D1; CDK2, cyclin-dependent kinase 2; IGF2BP1, insulin-like growth factor 2 mRNA-binding protein 1; YTHDF2, YTH domain family 2.

    Article Snippet: In addition, for mitotic clone amplification (MCE) in the early stage of terminal differentiation, the inhibition of FTO expression in 3T3-L1 cells leads to increased m 6 A methylation levels of cyclin D1 (CCND1) and cyclin-dependent kinase 2, the protein expression of which is reduced after recognition by YTHDF2, resulting in blockade of the MCE process and in turn the inhibition of lipogenesis ( ) ( ).

    Techniques: Modification, Expressing, Phospho-proteomics, Knock-Out, Blocking Assay, Amplification, Binding Assay