syntaxin1a (Cell Signaling Technology Inc)
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Cell Signaling Technology Inc manufactures this product 
93
Structured Review
Cell Signaling Technology Inc
syntaxin1a
Syntaxin1a, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 93/100, based on 25 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/syntaxin1a/Syntaxin+1A+Rabbit+mAb/pm39993668-110-33-34
Average 93 stars, based on 25 article reviews
Syntaxin1a, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 93/100, based on 25 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/syntaxin1a/Syntaxin+1A+Rabbit+mAb/pm39993668-110-33-34
Average 93 stars, based on 25 article reviews
syntaxin1a - by Bioz Stars,
2026-09
93/100 stars
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Related Articles
Membrane:Article Title: Electroacupuncture restores maternal separation-induced glutamatergic presynaptic deficits of the medial prefrontal cortex in adulthood. Article Snippet: Maternal separation (MS) serves as a critical model of early life stress (ELS) that can lead to mood disorders, such as depression.. Our previous studies suggest that MS may disrupt synaptic transmission in adulthood.. While electroacupuncture (EA) has demonstrated antidepressant effects in several animal models of stress-induced depression, it remains unclear whether EA can reverse synaptic transmission deficits caused by ELS. Incubation:Article Title: Electroacupuncture restores maternal separation-induced glutamatergic presynaptic deficits of the medial prefrontal cortex in adulthood. Article Snippet: Maternal separation (MS) serves as a critical model of early life stress (ELS) that can lead to mood disorders, such as depression.. Our previous studies suggest that MS may disrupt synaptic transmission in adulthood.. While electroacupuncture (EA) has demonstrated antidepressant effects in several animal models of stress-induced depression, it remains unclear whether EA can reverse synaptic transmission deficits caused by ELS. |
