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scd1  (Proteintech)


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    Structured Review

    Proteintech scd1
    (A): (left) Representative immunohistochemical staining of the skin using an anti -ABCB1 antibody. (center) Comparison of PPARg and <t>SCD1</t> gene expression. (right) Representative immunohistochemical staining of the skin using an anti -SCD1 antibody. ∗∗ no significant difference; ∗P < 0.05. (B): Representative immunohistochemical staining of the skin using an anti -ki67 antibody and Nuclear Ki67 IHC score. Expression of ki67 was significantly reduced in Tg compared to wild type. ∗P < 0.05. (C): Representative immunohistochemical staining of the skin using an anti -Loricrin antibody. (D): Representative immunohistochemical staining of the skin using an anti-YAP antibody.
    Scd1, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 144 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/scd1/product/Proteintech
    Average 96 stars, based on 144 article reviews
    scd1 - by Bioz Stars, 2026-03
    96/100 stars

    Images

    1) Product Images from "TMEM207-mediated the impairment of skin regeneration through YAP sequestration in an allergic contact dermatitis model"

    Article Title: TMEM207-mediated the impairment of skin regeneration through YAP sequestration in an allergic contact dermatitis model

    Journal: Biochemistry and Biophysics Reports

    doi: 10.1016/j.bbrep.2025.102409

    (A): (left) Representative immunohistochemical staining of the skin using an anti -ABCB1 antibody. (center) Comparison of PPARg and SCD1 gene expression. (right) Representative immunohistochemical staining of the skin using an anti -SCD1 antibody. ∗∗ no significant difference; ∗P < 0.05. (B): Representative immunohistochemical staining of the skin using an anti -ki67 antibody and Nuclear Ki67 IHC score. Expression of ki67 was significantly reduced in Tg compared to wild type. ∗P < 0.05. (C): Representative immunohistochemical staining of the skin using an anti -Loricrin antibody. (D): Representative immunohistochemical staining of the skin using an anti-YAP antibody.
    Figure Legend Snippet: (A): (left) Representative immunohistochemical staining of the skin using an anti -ABCB1 antibody. (center) Comparison of PPARg and SCD1 gene expression. (right) Representative immunohistochemical staining of the skin using an anti -SCD1 antibody. ∗∗ no significant difference; ∗P < 0.05. (B): Representative immunohistochemical staining of the skin using an anti -ki67 antibody and Nuclear Ki67 IHC score. Expression of ki67 was significantly reduced in Tg compared to wild type. ∗P < 0.05. (C): Representative immunohistochemical staining of the skin using an anti -Loricrin antibody. (D): Representative immunohistochemical staining of the skin using an anti-YAP antibody.

    Techniques Used: Immunohistochemical staining, Staining, Comparison, Gene Expression, Expressing



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    (A): (left) Representative immunohistochemical staining of the skin using an anti -ABCB1 antibody. (center) Comparison of PPARg and <t>SCD1</t> gene expression. (right) Representative immunohistochemical staining of the skin using an anti -SCD1 antibody. ∗∗ no significant difference; ∗P < 0.05. (B): Representative immunohistochemical staining of the skin using an anti -ki67 antibody and Nuclear Ki67 IHC score. Expression of ki67 was significantly reduced in Tg compared to wild type. ∗P < 0.05. (C): Representative immunohistochemical staining of the skin using an anti -Loricrin antibody. (D): Representative immunohistochemical staining of the skin using an anti-YAP antibody.
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    Image Search Results


    (A): (left) Representative immunohistochemical staining of the skin using an anti -ABCB1 antibody. (center) Comparison of PPARg and SCD1 gene expression. (right) Representative immunohistochemical staining of the skin using an anti -SCD1 antibody. ∗∗ no significant difference; ∗P < 0.05. (B): Representative immunohistochemical staining of the skin using an anti -ki67 antibody and Nuclear Ki67 IHC score. Expression of ki67 was significantly reduced in Tg compared to wild type. ∗P < 0.05. (C): Representative immunohistochemical staining of the skin using an anti -Loricrin antibody. (D): Representative immunohistochemical staining of the skin using an anti-YAP antibody.

    Journal: Biochemistry and Biophysics Reports

    Article Title: TMEM207-mediated the impairment of skin regeneration through YAP sequestration in an allergic contact dermatitis model

    doi: 10.1016/j.bbrep.2025.102409

    Figure Lengend Snippet: (A): (left) Representative immunohistochemical staining of the skin using an anti -ABCB1 antibody. (center) Comparison of PPARg and SCD1 gene expression. (right) Representative immunohistochemical staining of the skin using an anti -SCD1 antibody. ∗∗ no significant difference; ∗P < 0.05. (B): Representative immunohistochemical staining of the skin using an anti -ki67 antibody and Nuclear Ki67 IHC score. Expression of ki67 was significantly reduced in Tg compared to wild type. ∗P < 0.05. (C): Representative immunohistochemical staining of the skin using an anti -Loricrin antibody. (D): Representative immunohistochemical staining of the skin using an anti-YAP antibody.

    Article Snippet: Rabbit anti -GAPDH antibodies were obtained from Sigma-Aldrich (St. Louis, MO, USA), and rabbit anti -ABCB1, anti -SCD1, anti -NEDD4, anti -Ki67, anti -Loricrin, and anti-YAP antibodies were purchased from ProteinTech (Proteintech, Inc., USA).

    Techniques: Immunohistochemical staining, Staining, Comparison, Gene Expression, Expressing

    ACSL3 deficiency abrogates PA-induced activation of lipogenic programs without enhancing β-oxidation. (A–C) mRNA levels by RT-qPCR normalized to ACTB: CPT1, CPT2, and ACOX (A); SCD, ChREBP, and SREBP1 (B); PPARG and LXRA (C). Abbreviations: KO, knockout; WT, wild type.

    Journal: Hepatology Communications

    Article Title: Genome-wide CRISPR screen identifies ACSL3 as a regulator of lipotoxicity and progression of MASLD

    doi: 10.1097/HC9.0000000000000884

    Figure Lengend Snippet: ACSL3 deficiency abrogates PA-induced activation of lipogenic programs without enhancing β-oxidation. (A–C) mRNA levels by RT-qPCR normalized to ACTB: CPT1, CPT2, and ACOX (A); SCD, ChREBP, and SREBP1 (B); PPARG and LXRA (C). Abbreviations: KO, knockout; WT, wild type.

    Article Snippet: Primers used in the TaqMan assays (ThermoFisher Scientific) for ACSL3 (Hs00244853_m1), CPT1A ( Hs00912671_m1), CPT2 ( Hs00988962_m1), ACOX1 ( Hs01074241_m1), SCD ( Hs01682761_m1), ChREBP (MLXIPL; Hs00975714_m1), SREBP1 (SREBF1; Hs02561944_s1), PPARγ (Hs01115513_m1), and LXRα (Nr1h3; Hs00172885_m1 ) were also used.

    Techniques: Activation Assay, Quantitative RT-PCR, Knock-Out