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cd45 30 f11 89y surface 100 fluidigm 3089005c cd3 145 2c11 152sm surface 100 fluidigm 3152004c cd4 rm4 5 145nd surface 100 fluidigm 3145002c cd8  (fluidigm)


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    fluidigm cd45 30 f11 89y surface 100 fluidigm 3089005c cd3 145 2c11 152sm surface 100 fluidigm 3152004c cd4 rm4 5 145nd surface 100 fluidigm 3145002c cd8
    Cd45 30 F11 89y Surface 100 Fluidigm 3089005c Cd3 145 2c11 152sm Surface 100 Fluidigm 3152004c Cd4 Rm4 5 145nd Surface 100 Fluidigm 3145002c Cd8, supplied by fluidigm, used in various techniques. Bioz Stars score: 93/100, based on 26 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/rm4-5/pmc12506966__sciadv%2Eadv9161_sm-45-8-13?v=fluidigm
    Average 93 stars, based on 26 article reviews
    cd45 30 f11 89y surface 100 fluidigm 3089005c cd3 145 2c11 152sm surface 100 fluidigm 3152004c cd4 rm4 5 145nd surface 100 fluidigm 3145002c cd8 - by Bioz Stars, 2026-07
    93/100 stars

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    Image Search Results


    Effects of quinic acid on hepatic GLUT2 protein expression and GLP-1 levels in sodium arsenite (SA)-induced hepatotoxicity. (A) Hepatic GLUT2 protein expression. (B) Representative Western blot analysis of liver tissues for GLUT2 and GAPDH. (C) Hepatic GLP-1 levels. GLUT2 protein levels were quantified relative to the loading control GAPDH. The results are presented as mean ± SEM. *Significant difference with the control group (*** P < 0.001). #Significant difference with the SA group (# P < 0.05, ## P < 0.01).

    Journal: Scientific Reports

    Article Title: Quinic acid attenuates arsenic-induced hepatic injury and hyperglycemia in mice via GLUT2 upregulation and suppression of oxidative stress and inflammation

    doi: 10.1038/s41598-025-33451-3

    Figure Lengend Snippet: Effects of quinic acid on hepatic GLUT2 protein expression and GLP-1 levels in sodium arsenite (SA)-induced hepatotoxicity. (A) Hepatic GLUT2 protein expression. (B) Representative Western blot analysis of liver tissues for GLUT2 and GAPDH. (C) Hepatic GLP-1 levels. GLUT2 protein levels were quantified relative to the loading control GAPDH. The results are presented as mean ± SEM. *Significant difference with the control group (*** P < 0.001). #Significant difference with the SA group (# P < 0.05, ## P < 0.01).

    Article Snippet: Primary antibodies against GLUT2 (1:500, EPR16550 , cat. no. ab192599; RabMAb Technology, China) and the enzyme glyceraldehyde 3-phosphate dehydrogenase (GAPDH) (1:500, 14C10, cat. no. 2118; Cell Signaling Technology, USA) were incubated overnight at 4 °C.

    Techniques: Expressing, Western Blot, Control

    Graphical representation of the proposed protective mechanism of quinic acid against arsenic-induced hepatotoxicity and glucose intolerance. Quinic acid (QA) reduces fasting blood sugar and improves glucose intolerance. Its mechanism involves enhancing the cellular antioxidant defense system, reducing inflammation, and upregulating GLP-1 and GLUT2.Portions of the graphical abstract were drawn using images from Servier Medical Art ( https://smart.servier.com/ ). Servier Medical Art content is licensed under the Creative Commons Attribution 4.0 International License (CC BY 4.0). ( https://creativecommons.org/licenses/by/4.0/ ).

    Journal: Scientific Reports

    Article Title: Quinic acid attenuates arsenic-induced hepatic injury and hyperglycemia in mice via GLUT2 upregulation and suppression of oxidative stress and inflammation

    doi: 10.1038/s41598-025-33451-3

    Figure Lengend Snippet: Graphical representation of the proposed protective mechanism of quinic acid against arsenic-induced hepatotoxicity and glucose intolerance. Quinic acid (QA) reduces fasting blood sugar and improves glucose intolerance. Its mechanism involves enhancing the cellular antioxidant defense system, reducing inflammation, and upregulating GLP-1 and GLUT2.Portions of the graphical abstract were drawn using images from Servier Medical Art ( https://smart.servier.com/ ). Servier Medical Art content is licensed under the Creative Commons Attribution 4.0 International License (CC BY 4.0). ( https://creativecommons.org/licenses/by/4.0/ ).

    Article Snippet: Primary antibodies against GLUT2 (1:500, EPR16550 , cat. no. ab192599; RabMAb Technology, China) and the enzyme glyceraldehyde 3-phosphate dehydrogenase (GAPDH) (1:500, 14C10, cat. no. 2118; Cell Signaling Technology, USA) were incubated overnight at 4 °C.

    Techniques: