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pde4b kd rs09 group  (MedChemExpress)


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    MedChemExpress pde4b kd rs09 group
    Pde4b Kd Rs09 Group, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 94/100, based on 9 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Average 94 stars, based on 9 article reviews
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    94/100 stars

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    In young males, dAIH exposure significantly reduced Q pathway associated 5HT2A receptor expression (Panel A). In young females, dAIH significantly reduced S to Q cross talk molecules – p38Map kinase β ( Mapk11 ) and phosphodiesterase ( <t>Pde4b</t> ) expression. Reduced phosphodiesterase expression could indirectly increase PKA or EPAC activity which could in turn increase S to Q inhibition (Panel B). In middle aged males, no significant effect of dAIH exposure was observed (Panel C). In contrast widespread gene expression changes with dAIH exposure were observed in middle aged females. dAIH reduced expression of 5HT2B and A2A receptor, Q to S cross talk molecules–PKCδ ( Prkcd ) and NADPH-p47 ( Ncf1 ), S to Q cross-talk molecules phosphodiesterase 4b, and fractalkine ( Cx3cl1 ) (Panel D).
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    In young males, dAIH exposure significantly reduced Q pathway associated 5HT2A receptor expression (Panel A). In young females, dAIH significantly reduced S to Q cross talk molecules – p38Map kinase β ( Mapk11 ) and phosphodiesterase ( <t>Pde4b</t> ) expression. Reduced phosphodiesterase expression could indirectly increase PKA or EPAC activity which could in turn increase S to Q inhibition (Panel B). In middle aged males, no significant effect of dAIH exposure was observed (Panel C). In contrast widespread gene expression changes with dAIH exposure were observed in middle aged females. dAIH reduced expression of 5HT2B and A2A receptor, Q to S cross talk molecules–PKCδ ( Prkcd ) and NADPH-p47 ( Ncf1 ), S to Q cross-talk molecules phosphodiesterase 4b, and fractalkine ( Cx3cl1 ) (Panel D).
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    In young males, dAIH exposure significantly reduced Q pathway associated 5HT2A receptor expression (Panel A). In young females, dAIH significantly reduced S to Q cross talk molecules – p38Map kinase β ( Mapk11 ) and phosphodiesterase ( <t>Pde4b</t> ) expression. Reduced phosphodiesterase expression could indirectly increase PKA or EPAC activity which could in turn increase S to Q inhibition (Panel B). In middle aged males, no significant effect of dAIH exposure was observed (Panel C). In contrast widespread gene expression changes with dAIH exposure were observed in middle aged females. dAIH reduced expression of 5HT2B and A2A receptor, Q to S cross talk molecules–PKCδ ( Prkcd ) and NADPH-p47 ( Ncf1 ), S to Q cross-talk molecules phosphodiesterase 4b, and fractalkine ( Cx3cl1 ) (Panel D).
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    In young males, dAIH exposure significantly reduced Q pathway associated 5HT2A receptor expression (Panel A). In young females, dAIH significantly reduced S to Q cross talk molecules – p38Map kinase β ( Mapk11 ) and phosphodiesterase ( <t>Pde4b</t> ) expression. Reduced phosphodiesterase expression could indirectly increase PKA or EPAC activity which could in turn increase S to Q inhibition (Panel B). In middle aged males, no significant effect of dAIH exposure was observed (Panel C). In contrast widespread gene expression changes with dAIH exposure were observed in middle aged females. dAIH reduced expression of 5HT2B and A2A receptor, Q to S cross talk molecules–PKCδ ( Prkcd ) and NADPH-p47 ( Ncf1 ), S to Q cross-talk molecules phosphodiesterase 4b, and fractalkine ( Cx3cl1 ) (Panel D).
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    In young males, dAIH exposure significantly reduced Q pathway associated 5HT2A receptor expression (Panel A). In young females, dAIH significantly reduced S to Q cross talk molecules – p38Map kinase β ( Mapk11 ) and phosphodiesterase ( Pde4b ) expression. Reduced phosphodiesterase expression could indirectly increase PKA or EPAC activity which could in turn increase S to Q inhibition (Panel B). In middle aged males, no significant effect of dAIH exposure was observed (Panel C). In contrast widespread gene expression changes with dAIH exposure were observed in middle aged females. dAIH reduced expression of 5HT2B and A2A receptor, Q to S cross talk molecules–PKCδ ( Prkcd ) and NADPH-p47 ( Ncf1 ), S to Q cross-talk molecules phosphodiesterase 4b, and fractalkine ( Cx3cl1 ) (Panel D).

    Journal: Experimental neurology

    Article Title: Daily acute intermittent hypoxia elicits age & sex-dependent changes in molecules regulating phrenic motor plasticity

    doi: 10.1016/j.expneurol.2025.115240

    Figure Lengend Snippet: In young males, dAIH exposure significantly reduced Q pathway associated 5HT2A receptor expression (Panel A). In young females, dAIH significantly reduced S to Q cross talk molecules – p38Map kinase β ( Mapk11 ) and phosphodiesterase ( Pde4b ) expression. Reduced phosphodiesterase expression could indirectly increase PKA or EPAC activity which could in turn increase S to Q inhibition (Panel B). In middle aged males, no significant effect of dAIH exposure was observed (Panel C). In contrast widespread gene expression changes with dAIH exposure were observed in middle aged females. dAIH reduced expression of 5HT2B and A2A receptor, Q to S cross talk molecules–PKCδ ( Prkcd ) and NADPH-p47 ( Ncf1 ), S to Q cross-talk molecules phosphodiesterase 4b, and fractalkine ( Cx3cl1 ) (Panel D).

    Article Snippet: phosphodiesterase 4B , pde4b , S to Q crosstalk , Rn00566785_m1 , 70.

    Techniques: Expressing, Activity Assay, Inhibition, Gene Expression