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m1523-5mg  (Millipore)


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    Millipore m1523-5mg
    M1523 5mg, supplied by Millipore, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/m1523/muscimol/pm38733991-245-14-11
    Average 90 stars, based on 1 article reviews
    m1523-5mg - by Bioz Stars, 2026-09
    90/100 stars

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    Software:

    Article Title: Reversible Silencing of the Frontopolar Cortex Selectively Impairs Metacognitive Judgment on Non-experience in Primates.
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    Magnetic Resonance Imaging:

    Article Title: Reversible Silencing of the Frontopolar Cortex Selectively Impairs Metacognitive Judgment on Non-experience in Primates.
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    Injection:

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    Article Title: Conversion of concept-specific decision confidence into integrative introspection in primates.
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    Altered vascular dynamics in chronically stressed mice, as measured in acute brain slices. A , The schematic setup showing the experiment. B , Representative images of baseline and postelectrical 20 Hz stimulation arterioles in the somatosensory cortex of control and stressed mice. Red vertical lines were traced along vascular walls for visualization of penetrating arterioles. C , D , Time course traces and maximal relative amplitudes of arteriolar diameter changes following electrical stimulation for control (black, n = 10) and stressed brain (red, n = 11). Clearly, stimulation induces dilation for the control, but constriction for the stressed brain slice. E , F , Time course traces and maximal relative amplitudes of vascular diameter changes after stimulation in the presence (green, n = 6) or absence of TTX (gray, n = 5) in the control mouse brain slice. In the control brain slice, stimulation induces vascular dilation (1.08 ± 0.01), which is abolished by TTX application (0.98 ± 0.01, U (6,5) = 0 , p = 0.004, U test). G–J , Time course traces and maximal relative amplitudes of vessel diameter change induced by 2 min application (horizontal black line) of 30 μ <t>m</t> <t>NMDA</t> ( G , H ; control, n = 9; stressed, n = 7) and 10 μ m AMPA ( I , J ; control, n = 5; stressed, n = 5) for control (black) and stressed brain slice (red). Both NMDA and AMPA mimic the action of glutamate. Neither compounds produce any significant difference in vascular diameter for control versus stressed slice. K–M , Time course traces ( K , M ) and maximal relative amplitudes ( L , N ) of vessel diameter change induced by 2 min treatment of GABA receptor agonists (100 μ m <t>muscimol</t> + 50 μ m baclofen) in the absence ( K , M ) and presence of TTX ( L , N ) for control (black, n = 13 without and 8 with TTX) and stressed brain slices (red, n = 11 without and 11 with TTX). Data are mean ± SEM. ** p < 0.01, *** p < 0.001, n.s., not significant.
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    Image Search Results


    Altered vascular dynamics in chronically stressed mice, as measured in acute brain slices. A , The schematic setup showing the experiment. B , Representative images of baseline and postelectrical 20 Hz stimulation arterioles in the somatosensory cortex of control and stressed mice. Red vertical lines were traced along vascular walls for visualization of penetrating arterioles. C , D , Time course traces and maximal relative amplitudes of arteriolar diameter changes following electrical stimulation for control (black, n = 10) and stressed brain (red, n = 11). Clearly, stimulation induces dilation for the control, but constriction for the stressed brain slice. E , F , Time course traces and maximal relative amplitudes of vascular diameter changes after stimulation in the presence (green, n = 6) or absence of TTX (gray, n = 5) in the control mouse brain slice. In the control brain slice, stimulation induces vascular dilation (1.08 ± 0.01), which is abolished by TTX application (0.98 ± 0.01, U (6,5) = 0 , p = 0.004, U test). G–J , Time course traces and maximal relative amplitudes of vessel diameter change induced by 2 min application (horizontal black line) of 30 μ m NMDA ( G , H ; control, n = 9; stressed, n = 7) and 10 μ m AMPA ( I , J ; control, n = 5; stressed, n = 5) for control (black) and stressed brain slice (red). Both NMDA and AMPA mimic the action of glutamate. Neither compounds produce any significant difference in vascular diameter for control versus stressed slice. K–M , Time course traces ( K , M ) and maximal relative amplitudes ( L , N ) of vessel diameter change induced by 2 min treatment of GABA receptor agonists (100 μ m muscimol + 50 μ m baclofen) in the absence ( K , M ) and presence of TTX ( L , N ) for control (black, n = 13 without and 8 with TTX) and stressed brain slices (red, n = 11 without and 11 with TTX). Data are mean ± SEM. ** p < 0.01, *** p < 0.001, n.s., not significant.

    Journal: The Journal of Neuroscience

    Article Title: Neurovascular Coupling under Chronic Stress Is Modified by Altered GABAergic Interneuron Activity

    doi: 10.1523/JNEUROSCI.1357-19.2019

    Figure Lengend Snippet: Altered vascular dynamics in chronically stressed mice, as measured in acute brain slices. A , The schematic setup showing the experiment. B , Representative images of baseline and postelectrical 20 Hz stimulation arterioles in the somatosensory cortex of control and stressed mice. Red vertical lines were traced along vascular walls for visualization of penetrating arterioles. C , D , Time course traces and maximal relative amplitudes of arteriolar diameter changes following electrical stimulation for control (black, n = 10) and stressed brain (red, n = 11). Clearly, stimulation induces dilation for the control, but constriction for the stressed brain slice. E , F , Time course traces and maximal relative amplitudes of vascular diameter changes after stimulation in the presence (green, n = 6) or absence of TTX (gray, n = 5) in the control mouse brain slice. In the control brain slice, stimulation induces vascular dilation (1.08 ± 0.01), which is abolished by TTX application (0.98 ± 0.01, U (6,5) = 0 , p = 0.004, U test). G–J , Time course traces and maximal relative amplitudes of vessel diameter change induced by 2 min application (horizontal black line) of 30 μ m NMDA ( G , H ; control, n = 9; stressed, n = 7) and 10 μ m AMPA ( I , J ; control, n = 5; stressed, n = 5) for control (black) and stressed brain slice (red). Both NMDA and AMPA mimic the action of glutamate. Neither compounds produce any significant difference in vascular diameter for control versus stressed slice. K–M , Time course traces ( K , M ) and maximal relative amplitudes ( L , N ) of vessel diameter change induced by 2 min treatment of GABA receptor agonists (100 μ m muscimol + 50 μ m baclofen) in the absence ( K , M ) and presence of TTX ( L , N ) for control (black, n = 13 without and 8 with TTX) and stressed brain slices (red, n = 11 without and 11 with TTX). Data are mean ± SEM. ** p < 0.01, *** p < 0.001, n.s., not significant.

    Article Snippet: NMDA (M3262), AMPA hydrobromide (A9111), muscimol (M1523), baclofen (B5399), NBQX disodium salt hydrate (N183), SOM (S1763), and BIBP 3226 (B174) were purchased from Sigma-Aldrich.

    Techniques: Slice Preparation