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Low-density lipoprotein-cholesterol (LDL-C) levels
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Activation of the AMPK-SIRT1 pathway contributes to protective effects of Salvianolic acid A against lipotoxicity in hepatocytes and NAFLD in mice 
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Sal A treatment alleviated HFCD-induced increase in body weight and plasma lipids content in C57bl/6 mice. (A) Chemical structures of the Sal A. (B) Morphological photographs of the mice. (C) Dynamic alteration of body weight. (D) Body weight gain. (E) Daily food intake. (F) Plasma triglycerides (TG) level. (G) Plasma glycerol level. (H) Plasma free fatty acids (FFA) level. (I) Plasma total <t>cholesterol</t> level. (J) Plasma high-density <t>lipoprotein-cholesterol</t> (HDL-C) level. (K) plasma low-density lipoprotein-cholesterol <t>(LDL-C)</t> level. All values are denoted as means ± SD. The values with different superscripts are significantly different at p < 0.05. # Comparisons with normal diet (ND) group; * Comparisons with high-fat diet (HFCD) group. All groups contain 12 animals ( n = 12).
Activation of the AMPK-SIRT1 pathway contributes to protective effects of Salvianolic acid A against lipotoxicity in hepatocytes and NAFLD in mice Frontiers in Pharmacology, 2020 Nov 30
"Sal A treatment alleviated HFCD-induced increase in body weight and plasma lipids content in C57bl/6 mice. (A) Chemical structures of the Sal A. (B) Morphological photographs of the mice. (C) Dynamic alteration of body weight. (D) Body weight gain. (E) Daily food intake. (F) Plasma triglycerides (TG) level. (G) Plasma glycerol level. (H) Plasma free fatty acids (FFA) level. (I) Plasma total <t>cholesterol</t> level. (J) Plasma high-density <t>lipoprotein-cholesterol</t> (HDL-C) level. (K) plasma low-density lipoprotein-cholesterol <t>(LDL-C)</t> level. All values are denoted as means ± SD. The values with different superscripts are significantly different at p < 0.05. # Comparisons with normal diet (ND) group; * Comparisons with high-fat diet (HFCD) group. All groups contain 12 animals ( n = 12). "
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