draq5 (Cell Signaling Technology Inc)
Cell Signaling Technology Inc is a verified supplier 
Cell Signaling Technology Inc manufactures this product 
86
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Cell Signaling Technology Inc
draq5
Draq5, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/draq5/pm41933263-63-8-10
Average 86 stars, based on 1 article reviews
Draq5, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/draq5/pm41933263-63-8-10
Average 86 stars, based on 1 article reviews
draq5 - by Bioz Stars,
2026-09
86/100 stars
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Labeling:Article Title: IL-17 stimulates erythropoiesis in vivo by amplifying the response of erythroid progenitors to erythropoietin Article Snippet: All cells were post-labeled with DAPI (Thermofisher Scientific, Item # D1306) to exclude dead cells. .. Reticulocytes were measured by flow-cytometric analysis of blood labeled with the Staining:Article Title: Development of low-cost electrical stimulation device to promote hiPSC-CM differentiation and functionality Article Snippet: Cells were then washed with blocking buffer and incubated with anti-rabbit Alexa-488 secondary antibody in blocking buffer (1:400, Sigma-Aldrich, SAB4600044). .. Cells were co-stained with nuclear Incubation:Article Title: ISG15 dysregulates endoplasmic reticulum-mitochondrial contacts and calcium homeostasis in Ataxia telangiectasia. Article Snippet: Dysregulation of endoplasmic reticulum and mitochondrial (ER:Mit) contacts and mitochondrial calcium (mitCa2+) homeostasis are found in several neurodegenerative disorders, including Ataxia Telangiectasia (A-T).. However, the cellular basis of these defects remains unclear.. Previously, we demonstrated that the aberrantly elevated Interferon-Stimulated Gene 15 (ISG15) pathway inhibits protein polyubiquitylation, its dependent protein turnover, and mitophagy pathways in A-T. |
![(A) Measurement of the relative incorporation of [U- 14 C]glucose into protein, non-polar, RNA, DNA and polar fractions following 24 hours of labelling, n=3. (B) Relative incorporation of [1- 14 C]acetate into the lipid fraction of NFs and CAFs, n=3. (C) Gene Set Enrichment Analysis (GSEA) plot derived from RNA-Seq analysis of CAFs versus NFs, showing enrichment of a “lipid metabolic process” signature (GO:0006629). (D) Heatmap of cholesterol biosynthesis gene expression in NFs and CAFs, as determined by RNA-Seq analysis, n=3. (E) Quantification of relative total cholesterol levels in NFs and CAFs, n=3. (F) UMAP visualisation of 19,601 patient-derived breast tumour stromal cells analysed by single cell RNA-Seq (scRNA-Seq) from the Human Breast Cancer Atlas . Stromal clusters corresponding to immunomodulatory CAFs (iCAFs), myCAFs, perivascular (PVL) and endothelial cell subsets are indicated by colour (top). Feature plots illustrating expression of SQLE (bottom left) and SC5D (bottom right) in breast tumour stromal cell clusters. Gene expression is represented by log-normalised expression values. The myCAF cluster is delineated by a dashed line. (G) Representative filipin staining (green) of free cholesterol in NFs and CAFs. Nuclei are stained with <t>Draq5</t> (blue). Dashed boxes indicate magnified regions shown to the right. White scale bar represents 10 µm, yellow scale bar represents 5 µm. (H) Quantification of relative cholesterol levels in the ER-enriched fraction isolated from NFs and CAFs, n=3.](https://bio-rxiv-images-cdn.bioz.com/dois_ending_with_37/10__64898_slash_2026__02__16__706237/10__64898_slash_2026__02__16__706237___F1.large.jpg)