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clindamycin  (MedChemExpress)


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    MedChemExpress clindamycin
    Clindamycin, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 91/100, based on 2 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/clindamycin/product/MedChemExpress
    Average 91 stars, based on 2 article reviews
    clindamycin - by Bioz Stars, 2026-02
    91/100 stars

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    Attenuator LRR is of good regulatory performance. A. Regulation mechanism of the attenuator LRR. LRR forms a terminator structure under no antibiotic condition, which inhibits the transcription of lacZ mRNA and generates short transcripts. In the presence of antibiotics, ribosome stalling induced by antibiotics results in the formation of an anti‐terminator structure of LRR. Consequently, the expression of lacZ is increased obviously. B. Antibiotics induce LacZ expression. NA, no antibiotics; Tet, tetracycline (1 μg ml −1 , 2.25 μM); Vgm, virgimycin M (1 μg ml −1 , 1.903 μM); Rap, rapamycin (8 μg ml −1 , 8.751 μM); Lin, <t>lincomycin</t> (4 μg ml −1 , 9.839 μM); Pnm, pristinamycin IA (4 μg ml −1 , 4.614 μM); Cam, chloramphenicol (1 μg ml −1 , 3.095 μM); Lnz, linezolid (0.2 μg ml −1 , 0.593 μM); Tel, telithromycin (2.5 μg ml −1 , 3.079 μM); Kan, kanamycin (2 μg ml −1 , 3.433 μM); Rtm, retapamulin (0.5 μg ml −1 , 0.965 μM); Van, vancomycin (1.5 μg ml −1 , 1.01 μM). The numbers above the columns are the fold change induced by antibiotics. The fold change was calculated by dividing the Miller units under antibiotic conditions by the Miller units under no antibiotic condition.
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    Attenuator LRR is of good regulatory performance. A. Regulation mechanism of the attenuator LRR. LRR forms a terminator structure under no antibiotic condition, which inhibits the transcription of lacZ mRNA and generates short transcripts. In the presence of antibiotics, ribosome stalling induced by antibiotics results in the formation of an anti‐terminator structure of LRR. Consequently, the expression of lacZ is increased obviously. B. Antibiotics induce LacZ expression. NA, no antibiotics; Tet, tetracycline (1 μg ml −1 , 2.25 μM); Vgm, virgimycin M (1 μg ml −1 , 1.903 μM); Rap, rapamycin (8 μg ml −1 , 8.751 μM); Lin, lincomycin (4 μg ml −1 , 9.839 μM); Pnm, pristinamycin IA (4 μg ml −1 , 4.614 μM); Cam, chloramphenicol (1 μg ml −1 , 3.095 μM); Lnz, linezolid (0.2 μg ml −1 , 0.593 μM); Tel, telithromycin (2.5 μg ml −1 , 3.079 μM); Kan, kanamycin (2 μg ml −1 , 3.433 μM); Rtm, retapamulin (0.5 μg ml −1 , 0.965 μM); Van, vancomycin (1.5 μg ml −1 , 1.01 μM). The numbers above the columns are the fold change induced by antibiotics. The fold change was calculated by dividing the Miller units under antibiotic conditions by the Miller units under no antibiotic condition.

    Journal: Microbial Biotechnology

    Article Title: Attenuator LRR – a regulatory tool for modulating gene expression in Gram‐positive bacteria

    doi: 10.1111/1751-7915.13797

    Figure Lengend Snippet: Attenuator LRR is of good regulatory performance. A. Regulation mechanism of the attenuator LRR. LRR forms a terminator structure under no antibiotic condition, which inhibits the transcription of lacZ mRNA and generates short transcripts. In the presence of antibiotics, ribosome stalling induced by antibiotics results in the formation of an anti‐terminator structure of LRR. Consequently, the expression of lacZ is increased obviously. B. Antibiotics induce LacZ expression. NA, no antibiotics; Tet, tetracycline (1 μg ml −1 , 2.25 μM); Vgm, virgimycin M (1 μg ml −1 , 1.903 μM); Rap, rapamycin (8 μg ml −1 , 8.751 μM); Lin, lincomycin (4 μg ml −1 , 9.839 μM); Pnm, pristinamycin IA (4 μg ml −1 , 4.614 μM); Cam, chloramphenicol (1 μg ml −1 , 3.095 μM); Lnz, linezolid (0.2 μg ml −1 , 0.593 μM); Tel, telithromycin (2.5 μg ml −1 , 3.079 μM); Kan, kanamycin (2 μg ml −1 , 3.433 μM); Rtm, retapamulin (0.5 μg ml −1 , 0.965 μM); Van, vancomycin (1.5 μg ml −1 , 1.01 μM). The numbers above the columns are the fold change induced by antibiotics. The fold change was calculated by dividing the Miller units under antibiotic conditions by the Miller units under no antibiotic condition.

    Article Snippet: Antibiotics Rtm, tetracycline (Tet), Vgm, rapamycin (Rap), lincomycin (Lin), Pnm, Cam, Lnz, telithromycin (Tel), kanamycin (Kan) and vancomycin (Van) (MedChem Express, Monmouth Junction, NJ, USA) were added as required and cultivation was continued for different times.

    Techniques: Expressing