zero-d software package (scanalytics inc)
90
Structured Review
scanalytics inc
zero-d software package
Zero D Software Package, supplied by scanalytics inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/zero+d+software+package/zero+d+software/pmc02234589-333-23-24
Average 90 stars, based on 1 article reviews
Zero D Software Package, supplied by scanalytics inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/zero+d+software+package/zero+d+software/pmc02234589-333-23-24
Average 90 stars, based on 1 article reviews
zero-d software package - by Bioz Stars,
2026-10
90/100 stars
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Software:Article Title: 3-Methylcholanthrene and Other Aryl Hydrocarbon Receptor Agonists Directly Activate Estrogen Receptor α Article Snippet: After autoradiography, band intensities were determined by a scanning laser densitometer (Sharp Electronics Corporation, Mahwah, NJ) using Article Title: Vascular Endothelial Growth Factor Receptor-2 Expression Is Down-Regulated by 17?-Estradiol in MCF-7 Breast Cancer Cells by Estrogen Receptor ?/Sp Proteins Article Snippet: Band intensity values were obtained by scanning the film on a Sharp JX-330 scanner (Sharp Electronics, Mahwah, NJ) and by densitometry using the Article Title: Vascular endothelial growth factor receptor-2 expression is induced by 17beta-estradiol in ZR-75 breast cancer cells by estrogen receptor alpha/Sp proteins. Article Snippet: Band intensity values were obtained by scanning the film on a Sharp JX-330 scanner (Sharp Electronics, Mahwah, NJ) and by densitometry using the Article Title: GeNOSA: inferring and experimentally supporting quantitative gene regulatory networks in prokaryotes. Article Snippet: The quantitative results obtained using Article Title: Regulation of vascular endothelial growth factor receptor-2 expression in pancreatic cancer cells by Sp proteins. Article Snippet: Vascular endothelial growth factor receptor-2 (VEGFR2/KDR) is an important mediator of angiogenesis, and VEGFR2 mRNA is expressed in several pancreatic cancer cell lines.. Deletion analysis of the VEGFR2 promoter in Panc-1, AsPC-1, and MiaPaCa-2 pancreatic cancer cells shows that the proximal region of the promoter is primarily responsible for VEGFR2 expression, and two GC-rich sites at 58 and 44 are critical elements in all three cell lines.. Panc-1, AsPC-1, and MiaPaCa-2 cells also express Sp1, Sp3, and Sp4 proteins which bind to the GC-rich region of the VEGFR2 promoter in electrophoretic mobility shift and chromatin immunoprecipitation assays. Article Title: Cyclooxygenase-2 inhibitors decrease vascular endothelial growth factor expression in colon cancer cells by enhanced degradation of Sp1 and Sp4 proteins. Article Snippet: Cyclooxygenase 2 (COX-2) inhibitors, such as celecoxib (Cel), nimesulfide (NM), and NS-398 [NS; N-[2-(cyclohexyloxy)-4-nitrophenyl]methanesulfonamide] and other nonsteroidal anti-inflammatory drugs inhibit colon cancer growth and angiogenesis; however, the mechanism of this response is not well defined.. Treatment of SW-480 colon cancer cells with Cel, NS, or NM decreased vascular endothelial growth factor (VEGF) mRNA and immunoreactive protein expression.. This was also accompanied by decreased transactivation in cells transfected with constructs containing VEGF gene promoter inserts. Article Title: 2-Cyano-3,12-dioxoolean-1,9-dien-28-oic acid and related compounds inhibit growth of colon cancer cells through peroxisome proliferator-activated receptor gamma-dependent and -independent pathways. Article Snippet: 2-Cyano-3,12-dioxoolean-1,9-dien-28-oic acid (CDDO) and the corresponding methyl (CDDO-Me) and imidazole (CDDOIm) esters induce peroxisome proliferator-activated receptor (PPAR )-dependent transactivation in SW-480 colon cancer cells, and these responses were inhibited by small inhibitory RNA for PPAR .. Moreover, in a mammalian two-hybrid assay using the PPAR 2-VP16 fusion plasmid and GAL4-coactivator/ corepressor chimeras and a construct (pGAL4) containing five tandem GAL4 response elements, CDDO, CDDO-Me, and CDDO-IM induce transactivation and PPAR interaction with multiple coactivators.. A major difference among the three PPAR agonists was the higher activity of CDDO-Im to induce PPAR interactions with the corepressor SMRT. Article Title: Tolfenamic acid enhances pancreatic cancer cell and tumor response to radiation therapy by inhibiting survivin protein expression Article Snippet: After autoradiography, band intensities were determined by a scanning laser densitometer (Sharp Electronics) using Migration:Article Title: 3-Methylcholanthrene and Other Aryl Hydrocarbon Receptor Agonists Directly Activate Estrogen Receptor α Article Snippet: After autoradiography, band intensities were determined by a scanning laser densitometer (Sharp Electronics Corporation, Mahwah, NJ) using Article Title: Vascular Endothelial Growth Factor Receptor-2 Expression Is Down-Regulated by 17?-Estradiol in MCF-7 Breast Cancer Cells by Estrogen Receptor ?/Sp Proteins Article Snippet: Band intensity values were obtained by scanning the film on a Sharp JX-330 scanner (Sharp Electronics, Mahwah, NJ) and by densitometry using the Article Title: Vascular endothelial growth factor receptor-2 expression is induced by 17beta-estradiol in ZR-75 breast cancer cells by estrogen receptor alpha/Sp proteins. Article Snippet: Band intensity values were obtained by scanning the film on a Sharp JX-330 scanner (Sharp Electronics, Mahwah, NJ) and by densitometry using the Article Title: GeNOSA: inferring and experimentally supporting quantitative gene regulatory networks in prokaryotes. Article Snippet: The quantitative results obtained using Article Title: Regulation of vascular endothelial growth factor receptor-2 expression in pancreatic cancer cells by Sp proteins. Article Snippet: Vascular endothelial growth factor receptor-2 (VEGFR2/KDR) is an important mediator of angiogenesis, and VEGFR2 mRNA is expressed in several pancreatic cancer cell lines.. Deletion analysis of the VEGFR2 promoter in Panc-1, AsPC-1, and MiaPaCa-2 pancreatic cancer cells shows that the proximal region of the promoter is primarily responsible for VEGFR2 expression, and two GC-rich sites at 58 and 44 are critical elements in all three cell lines.. Panc-1, AsPC-1, and MiaPaCa-2 cells also express Sp1, Sp3, and Sp4 proteins which bind to the GC-rich region of the VEGFR2 promoter in electrophoretic mobility shift and chromatin immunoprecipitation assays. Article Title: Cyclooxygenase-2 inhibitors decrease vascular endothelial growth factor expression in colon cancer cells by enhanced degradation of Sp1 and Sp4 proteins. Article Snippet: Cyclooxygenase 2 (COX-2) inhibitors, such as celecoxib (Cel), nimesulfide (NM), and NS-398 [NS; N-[2-(cyclohexyloxy)-4-nitrophenyl]methanesulfonamide] and other nonsteroidal anti-inflammatory drugs inhibit colon cancer growth and angiogenesis; however, the mechanism of this response is not well defined.. Treatment of SW-480 colon cancer cells with Cel, NS, or NM decreased vascular endothelial growth factor (VEGF) mRNA and immunoreactive protein expression.. This was also accompanied by decreased transactivation in cells transfected with constructs containing VEGF gene promoter inserts. Article Title: 2-Cyano-3,12-dioxoolean-1,9-dien-28-oic acid and related compounds inhibit growth of colon cancer cells through peroxisome proliferator-activated receptor gamma-dependent and -independent pathways. Article Snippet: 2-Cyano-3,12-dioxoolean-1,9-dien-28-oic acid (CDDO) and the corresponding methyl (CDDO-Me) and imidazole (CDDOIm) esters induce peroxisome proliferator-activated receptor (PPAR )-dependent transactivation in SW-480 colon cancer cells, and these responses were inhibited by small inhibitory RNA for PPAR .. Moreover, in a mammalian two-hybrid assay using the PPAR 2-VP16 fusion plasmid and GAL4-coactivator/ corepressor chimeras and a construct (pGAL4) containing five tandem GAL4 response elements, CDDO, CDDO-Me, and CDDO-IM induce transactivation and PPAR interaction with multiple coactivators.. A major difference among the three PPAR agonists was the higher activity of CDDO-Im to induce PPAR interactions with the corepressor SMRT. Article Title: Tolfenamic acid enhances pancreatic cancer cell and tumor response to radiation therapy by inhibiting survivin protein expression Article Snippet: After autoradiography, band intensities were determined by a scanning laser densitometer (Sharp Electronics) using |