primescript reverse transcriptase kit (Promega)
90
Structured Review
Promega
primescript reverse transcriptase kit
Primescript Reverse Transcriptase Kit, supplied by Promega, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primescript+reverse+transcriptase+reagent+kit/primescript+reverse+transcriptase+kit/pm32317186-64-9-13
Average 90 stars, based on 1 article reviews
Primescript Reverse Transcriptase Kit, supplied by Promega, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primescript+reverse+transcriptase+reagent+kit/primescript+reverse+transcriptase+kit/pm32317186-64-9-13
Average 90 stars, based on 1 article reviews
primescript reverse transcriptase kit - by Bioz Stars,
2026-09
90/100 stars
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Related Articles
Reverse Transcription:Article Title: EPIC-0628 abrogates HOTAIR/EZH2 interaction and enhances the temozolomide efficacy via promoting ATF3 expression and inhibiting DNA damage repair in glioblastoma. Article Snippet: The efficacy of temozolomide (TMZ) treatment in glioblastoma (GBM) is influenced by various mechanisms, mainly including the level of O6-methylguanine-DNA methyltransferase (MGMT) and the activity of DNA damage repair (DDR) pathways.. In our previous study, we had proved that long non-coding RNA HOTAIR regulated the GBM progression and mediated DDR by interacting with EZH2, the catalytic subunit of PRC2.. In this study, we developed a small-molecule inhibitor called EPIC-0628 that selectively disrupted the HOTAIREZH2 interaction and promoted ATF3 expression. Article Title: Knockdown of fibronectin extra domain B suppresses TGF-β1-mediated cell proliferation and collagen deposition in keloid fibroblasts via AKT/ERK signaling pathway. Article Snippet: Keloids represent a dermal fibrotic disease characterized by excess collagen deposition and invasion of normal skin beyond the wound boundary, similar to malignant tumor features.. Fibronectin extra domain B (EDB) is highly expressed in many tumors but has not been studied in keloids.. The present study aimed to investigate the expression and the influence of EDB on keloid and elucidate the putative signaling pathway. Quantitative RT-PCR:Article Title: Knockdown of fibronectin extra domain B suppresses TGF-β1-mediated cell proliferation and collagen deposition in keloid fibroblasts via AKT/ERK signaling pathway. Article Snippet: Keloids represent a dermal fibrotic disease characterized by excess collagen deposition and invasion of normal skin beyond the wound boundary, similar to malignant tumor features.. Fibronectin extra domain B (EDB) is highly expressed in many tumors but has not been studied in keloids.. The present study aimed to investigate the expression and the influence of EDB on keloid and elucidate the putative signaling pathway. SYBR Green Assay:Article Title: Knockdown of fibronectin extra domain B suppresses TGF-β1-mediated cell proliferation and collagen deposition in keloid fibroblasts via AKT/ERK signaling pathway. Article Snippet: Keloids represent a dermal fibrotic disease characterized by excess collagen deposition and invasion of normal skin beyond the wound boundary, similar to malignant tumor features.. Fibronectin extra domain B (EDB) is highly expressed in many tumors but has not been studied in keloids.. The present study aimed to investigate the expression and the influence of EDB on keloid and elucidate the putative signaling pathway. |