microscope control and analysis software nis elements (Nikon)
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microscope control and analysis software nis elements
Microscope Control And Analysis Software Nis Elements, supplied by Nikon, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/nis+elements+microscope+control+software/pmc11031382-704-15-11
Average 90 stars, based on 1 article reviews
Microscope Control And Analysis Software Nis Elements, supplied by Nikon, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/nis+elements+microscope+control+software/pmc11031382-704-15-11
Average 90 stars, based on 1 article reviews
microscope control and analysis software nis elements - by Bioz Stars,
2026-10
90/100 stars
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Software:Article Title: Autism-associated SCN2A deficiency disrupts cortico-striatal circuitry in human brain assembloids Article Snippet: .. Colocalization of excitatory presynaptic (SYN1) and postsynaptic (PSD95) puncta was assessed using built-in Article Title: Microglial MS4A4A Protects against Epileptic Seizures in Alzheimer's Disease Article Snippet: Rhod‐2 fluorescence was measured using PerkinElmer Opera phenix. .. The images and movies were generated using Article Title: Supporting Information for AMBRA1 levels predict resistance to MAPK inhibitors in melanoma Article Snippet: .. The images were analyzed using Nikon’s Article Title: Rotenone induced acute miRNA alterations in extracellular vesicles produce mitochondrial dysfunction and cell death. Article Snippet: .. Posttreatment, the cell viability was assessed in (i) primary midbrain neurons and (ii) primary cortical neurons by using aMTT cell viability assay and b cell death detection by DNA damage ELISA. (iii) a Serum EVs were treated to primary midbrain neurons as mentioned above, and the cells were fixed posttreatment, and immunocytochemistry was performed in the cells usingTH (red) for specific dopaminergic neurons and MAP2 (green) as a general neuronal marker, and the cells were observed using confocal microscopy (merged image is displayed) and b % quantification of TH neurons over MAP2 was calculated by counting the cells using Article Title: Human endothelial cells promote a human neural stem cell type B phenotype via Notch signaling Article Snippet: Antibodies were as follows: Anti-GFAP, rabbit IgG, DAKO Z033429-2,1:200 Anti-GFAP, goat IgG, Abcamab53554,1:200 Anti-GFAP, mouse IgG, Sigma-Aldrich G3893, 1:200 Anti-SOX2, goat IgG, R&D SystemsAF2018, 1:200 Anti-CD31, mouse IgG, DAKO M0823, 1:200 Anti-LeX, mouse IgM,BD Biosciences 550382, 1:50 Anti-S100A6, rabbit IgG, Abcam ab181975, 1:100 Anti-AQP4, rabbit IgG, Sigma-Aldrich AB3594, 1:100 Anti-MAP2, mouse IgG, Sigma-Aldrich M9942, 1:200 Anti-DCX, goat IgG, Santa Cruz Biotechnology, SC-8066, 1:200 Anti-Ki67, rabbit IgG, Leica Biosystems, NCL-Ki67p, 1:500 Anti-Cleaved Caspase 3, rabbit IgG, Cell Signaling Technologies, 9661S, 1:200 AlexaFluor 488 donkey anti-rabbit IgG, Jackson ImmunoResearch, 711-545-152, 1:200 AlexaFluor 594 donkey anti-mouse IgG, Jackson ImmunoResearch, 715-585-151, 1:200 AlexaFluor 633 donkey anti-goat IgG, Thermo Scientific A-21082, 1:200 AlexaFluor 647 donkey anti-mouse IgM, Jackson ImmunoResearch, 715-605-020, 1:200 AlexaFluor 594 donkey anti-goat IgG, Jackson ImmunoResearch, 705-545-147, 1:200 AlexaFluor 488 donkey anti-mouse IgG, Jackson ImmunoResearch, 715-585-151, 1:200 .. Cells were imaged using a Nikon Eclipse Ti-E fluorescence microscope at 20× magnification and acquired using NIS Elements AR 4.51 image capturing and Article Title: Rotenone induced acute miRNA alterations in extracellular vesicles produce mitochondrial dysfunction and cell death Article Snippet: .. Posttreatment, the cell viability was assessed in ( i ) primary midbrain neurons and ( ii ) primary cortical neurons by using a MTT cell viability assay and b cell death detection by DNA damage ELISA. ( iii ) a Serum EVs were treated to primary midbrain neurons as mentioned above, and the cells were fixed posttreatment, and immunocytochemistry was performed in the cells using TH (red) for specific dopaminergic neurons and MAP2 (green) as a general neuronal marker, and the cells were observed using confocal microscopy (merged image is displayed) and b % quantification of TH neurons over MAP2 was calculated by counting the cells using Article Title: Rheological Characterization and 3D Fabrication of Artificial Bacterial Biofilms. Article Snippet: Biofilms are significantly involved in the progression of many diseases, such as cancer and upper respiratory infections, due to their ability to adhere to soft tissues.. Factors influencing biofilm development have been extensively studied on planar substrates; however, there is limited understanding regarding biofilm growth and interactions within 3D matrices.. Developing biofilm models that closely mimic natural bacterial communities’ chemical and mechanical properties in soft tissues is essential for developing next-generation antibacterial compounds and therapeutics, as 3D biofilms are more complex and less susceptible to treatment than their 2D counterparts. Article Title: An OMV-Based Nanovaccine as Antigen Presentation Signal Enhancer for Cancer Immunotherapy. Article Snippet: Antigen-presenting cells (APCs) process tumor vaccines and present tumor antigens as the first signals to T cells to activate anti-tumor immunity, which process requires the assistance of co-stimulatory second signals on APCs.. The immune checkpoint programmed death ligand 1 (PD-L1) not only mediates the immune escape of tumor cells but also acts as a co-inhibitory second signal on APCs.. The serious dysfunction of second signals due to the high expression of PD-L1 on APCs in the tumor body results in the inefficiency of tumor vaccines. Mutagenesis:Article Title: Autism-associated SCN2A deficiency disrupts cortico-striatal circuitry in human brain assembloids Article Snippet: .. Colocalization of excitatory presynaptic (SYN1) and postsynaptic (PSD95) puncta was assessed using built-in Generated:Article Title: Microglial MS4A4A Protects against Epileptic Seizures in Alzheimer's Disease Article Snippet: Rhod‐2 fluorescence was measured using PerkinElmer Opera phenix. .. The images and movies were generated using Viability Assay:Article Title: Rotenone induced acute miRNA alterations in extracellular vesicles produce mitochondrial dysfunction and cell death. Article Snippet: .. Posttreatment, the cell viability was assessed in (i) primary midbrain neurons and (ii) primary cortical neurons by using aMTT cell viability assay and b cell death detection by DNA damage ELISA. (iii) a Serum EVs were treated to primary midbrain neurons as mentioned above, and the cells were fixed posttreatment, and immunocytochemistry was performed in the cells usingTH (red) for specific dopaminergic neurons and MAP2 (green) as a general neuronal marker, and the cells were observed using confocal microscopy (merged image is displayed) and b % quantification of TH neurons over MAP2 was calculated by counting the cells using Article Title: Rotenone induced acute miRNA alterations in extracellular vesicles produce mitochondrial dysfunction and cell death Article Snippet: .. Posttreatment, the cell viability was assessed in ( i ) primary midbrain neurons and ( ii ) primary cortical neurons by using a MTT cell viability assay and b cell death detection by DNA damage ELISA. ( iii ) a Serum EVs were treated to primary midbrain neurons as mentioned above, and the cells were fixed posttreatment, and immunocytochemistry was performed in the cells using TH (red) for specific dopaminergic neurons and MAP2 (green) as a general neuronal marker, and the cells were observed using confocal microscopy (merged image is displayed) and b % quantification of TH neurons over MAP2 was calculated by counting the cells using Enzyme-linked Immunosorbent Assay:Article Title: Rotenone induced acute miRNA alterations in extracellular vesicles produce mitochondrial dysfunction and cell death. Article Snippet: .. Posttreatment, the cell viability was assessed in (i) primary midbrain neurons and (ii) primary cortical neurons by using aMTT cell viability assay and b cell death detection by DNA damage ELISA. (iii) a Serum EVs were treated to primary midbrain neurons as mentioned above, and the cells were fixed posttreatment, and immunocytochemistry was performed in the cells usingTH (red) for specific dopaminergic neurons and MAP2 (green) as a general neuronal marker, and the cells were observed using confocal microscopy (merged image is displayed) and b % quantification of TH neurons over MAP2 was calculated by counting the cells using Article Title: Rotenone induced acute miRNA alterations in extracellular vesicles produce mitochondrial dysfunction and cell death Article Snippet: .. Posttreatment, the cell viability was assessed in ( i ) primary midbrain neurons and ( ii ) primary cortical neurons by using a MTT cell viability assay and b cell death detection by DNA damage ELISA. ( iii ) a Serum EVs were treated to primary midbrain neurons as mentioned above, and the cells were fixed posttreatment, and immunocytochemistry was performed in the cells using TH (red) for specific dopaminergic neurons and MAP2 (green) as a general neuronal marker, and the cells were observed using confocal microscopy (merged image is displayed) and b % quantification of TH neurons over MAP2 was calculated by counting the cells using Immunocytochemistry:Article Title: Rotenone induced acute miRNA alterations in extracellular vesicles produce mitochondrial dysfunction and cell death. Article Snippet: .. Posttreatment, the cell viability was assessed in (i) primary midbrain neurons and (ii) primary cortical neurons by using aMTT cell viability assay and b cell death detection by DNA damage ELISA. (iii) a Serum EVs were treated to primary midbrain neurons as mentioned above, and the cells were fixed posttreatment, and immunocytochemistry was performed in the cells usingTH (red) for specific dopaminergic neurons and MAP2 (green) as a general neuronal marker, and the cells were observed using confocal microscopy (merged image is displayed) and b % quantification of TH neurons over MAP2 was calculated by counting the cells using Article Title: Rotenone induced acute miRNA alterations in extracellular vesicles produce mitochondrial dysfunction and cell death Article Snippet: .. Posttreatment, the cell viability was assessed in ( i ) primary midbrain neurons and ( ii ) primary cortical neurons by using a MTT cell viability assay and b cell death detection by DNA damage ELISA. ( iii ) a Serum EVs were treated to primary midbrain neurons as mentioned above, and the cells were fixed posttreatment, and immunocytochemistry was performed in the cells using TH (red) for specific dopaminergic neurons and MAP2 (green) as a general neuronal marker, and the cells were observed using confocal microscopy (merged image is displayed) and b % quantification of TH neurons over MAP2 was calculated by counting the cells using Marker:Article Title: Rotenone induced acute miRNA alterations in extracellular vesicles produce mitochondrial dysfunction and cell death. Article Snippet: .. Posttreatment, the cell viability was assessed in (i) primary midbrain neurons and (ii) primary cortical neurons by using aMTT cell viability assay and b cell death detection by DNA damage ELISA. (iii) a Serum EVs were treated to primary midbrain neurons as mentioned above, and the cells were fixed posttreatment, and immunocytochemistry was performed in the cells usingTH (red) for specific dopaminergic neurons and MAP2 (green) as a general neuronal marker, and the cells were observed using confocal microscopy (merged image is displayed) and b % quantification of TH neurons over MAP2 was calculated by counting the cells using Article Title: Rotenone induced acute miRNA alterations in extracellular vesicles produce mitochondrial dysfunction and cell death Article Snippet: .. Posttreatment, the cell viability was assessed in ( i ) primary midbrain neurons and ( ii ) primary cortical neurons by using a MTT cell viability assay and b cell death detection by DNA damage ELISA. ( iii ) a Serum EVs were treated to primary midbrain neurons as mentioned above, and the cells were fixed posttreatment, and immunocytochemistry was performed in the cells using TH (red) for specific dopaminergic neurons and MAP2 (green) as a general neuronal marker, and the cells were observed using confocal microscopy (merged image is displayed) and b % quantification of TH neurons over MAP2 was calculated by counting the cells using Confocal Microscopy:Article Title: Rotenone induced acute miRNA alterations in extracellular vesicles produce mitochondrial dysfunction and cell death. Article Snippet: .. Posttreatment, the cell viability was assessed in (i) primary midbrain neurons and (ii) primary cortical neurons by using aMTT cell viability assay and b cell death detection by DNA damage ELISA. (iii) a Serum EVs were treated to primary midbrain neurons as mentioned above, and the cells were fixed posttreatment, and immunocytochemistry was performed in the cells usingTH (red) for specific dopaminergic neurons and MAP2 (green) as a general neuronal marker, and the cells were observed using confocal microscopy (merged image is displayed) and b % quantification of TH neurons over MAP2 was calculated by counting the cells using Article Title: Rotenone induced acute miRNA alterations in extracellular vesicles produce mitochondrial dysfunction and cell death Article Snippet: .. Posttreatment, the cell viability was assessed in ( i ) primary midbrain neurons and ( ii ) primary cortical neurons by using a MTT cell viability assay and b cell death detection by DNA damage ELISA. ( iii ) a Serum EVs were treated to primary midbrain neurons as mentioned above, and the cells were fixed posttreatment, and immunocytochemistry was performed in the cells using TH (red) for specific dopaminergic neurons and MAP2 (green) as a general neuronal marker, and the cells were observed using confocal microscopy (merged image is displayed) and b % quantification of TH neurons over MAP2 was calculated by counting the cells using Fluorescence:Article Title: Human endothelial cells promote a human neural stem cell type B phenotype via Notch signaling Article Snippet: Antibodies were as follows: Anti-GFAP, rabbit IgG, DAKO Z033429-2,1:200 Anti-GFAP, goat IgG, Abcamab53554,1:200 Anti-GFAP, mouse IgG, Sigma-Aldrich G3893, 1:200 Anti-SOX2, goat IgG, R&D SystemsAF2018, 1:200 Anti-CD31, mouse IgG, DAKO M0823, 1:200 Anti-LeX, mouse IgM,BD Biosciences 550382, 1:50 Anti-S100A6, rabbit IgG, Abcam ab181975, 1:100 Anti-AQP4, rabbit IgG, Sigma-Aldrich AB3594, 1:100 Anti-MAP2, mouse IgG, Sigma-Aldrich M9942, 1:200 Anti-DCX, goat IgG, Santa Cruz Biotechnology, SC-8066, 1:200 Anti-Ki67, rabbit IgG, Leica Biosystems, NCL-Ki67p, 1:500 Anti-Cleaved Caspase 3, rabbit IgG, Cell Signaling Technologies, 9661S, 1:200 AlexaFluor 488 donkey anti-rabbit IgG, Jackson ImmunoResearch, 711-545-152, 1:200 AlexaFluor 594 donkey anti-mouse IgG, Jackson ImmunoResearch, 715-585-151, 1:200 AlexaFluor 633 donkey anti-goat IgG, Thermo Scientific A-21082, 1:200 AlexaFluor 647 donkey anti-mouse IgM, Jackson ImmunoResearch, 715-605-020, 1:200 AlexaFluor 594 donkey anti-goat IgG, Jackson ImmunoResearch, 705-545-147, 1:200 AlexaFluor 488 donkey anti-mouse IgG, Jackson ImmunoResearch, 715-585-151, 1:200 .. Cells were imaged using a Nikon Eclipse Ti-E fluorescence microscope at 20× magnification and acquired using NIS Elements AR 4.51 image capturing and Article Title: An OMV-Based Nanovaccine as Antigen Presentation Signal Enhancer for Cancer Immunotherapy. Article Snippet: Antigen-presenting cells (APCs) process tumor vaccines and present tumor antigens as the first signals to T cells to activate anti-tumor immunity, which process requires the assistance of co-stimulatory second signals on APCs.. The immune checkpoint programmed death ligand 1 (PD-L1) not only mediates the immune escape of tumor cells but also acts as a co-inhibitory second signal on APCs.. The serious dysfunction of second signals due to the high expression of PD-L1 on APCs in the tumor body results in the inefficiency of tumor vaccines. Microscopy:Article Title: Human endothelial cells promote a human neural stem cell type B phenotype via Notch signaling Article Snippet: Antibodies were as follows: Anti-GFAP, rabbit IgG, DAKO Z033429-2,1:200 Anti-GFAP, goat IgG, Abcamab53554,1:200 Anti-GFAP, mouse IgG, Sigma-Aldrich G3893, 1:200 Anti-SOX2, goat IgG, R&D SystemsAF2018, 1:200 Anti-CD31, mouse IgG, DAKO M0823, 1:200 Anti-LeX, mouse IgM,BD Biosciences 550382, 1:50 Anti-S100A6, rabbit IgG, Abcam ab181975, 1:100 Anti-AQP4, rabbit IgG, Sigma-Aldrich AB3594, 1:100 Anti-MAP2, mouse IgG, Sigma-Aldrich M9942, 1:200 Anti-DCX, goat IgG, Santa Cruz Biotechnology, SC-8066, 1:200 Anti-Ki67, rabbit IgG, Leica Biosystems, NCL-Ki67p, 1:500 Anti-Cleaved Caspase 3, rabbit IgG, Cell Signaling Technologies, 9661S, 1:200 AlexaFluor 488 donkey anti-rabbit IgG, Jackson ImmunoResearch, 711-545-152, 1:200 AlexaFluor 594 donkey anti-mouse IgG, Jackson ImmunoResearch, 715-585-151, 1:200 AlexaFluor 633 donkey anti-goat IgG, Thermo Scientific A-21082, 1:200 AlexaFluor 647 donkey anti-mouse IgM, Jackson ImmunoResearch, 715-605-020, 1:200 AlexaFluor 594 donkey anti-goat IgG, Jackson ImmunoResearch, 705-545-147, 1:200 AlexaFluor 488 donkey anti-mouse IgG, Jackson ImmunoResearch, 715-585-151, 1:200 .. Cells were imaged using a Nikon Eclipse Ti-E fluorescence microscope at 20× magnification and acquired using NIS Elements AR 4.51 image capturing and MTT Assay:Article Title: Rotenone induced acute miRNA alterations in extracellular vesicles produce mitochondrial dysfunction and cell death Article Snippet: .. Posttreatment, the cell viability was assessed in ( i ) primary midbrain neurons and ( ii ) primary cortical neurons by using a MTT cell viability assay and b cell death detection by DNA damage ELISA. ( iii ) a Serum EVs were treated to primary midbrain neurons as mentioned above, and the cells were fixed posttreatment, and immunocytochemistry was performed in the cells using TH (red) for specific dopaminergic neurons and MAP2 (green) as a general neuronal marker, and the cells were observed using confocal microscopy (merged image is displayed) and b % quantification of TH neurons over MAP2 was calculated by counting the cells using |