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ZHUOLI IMAGING TECHNOLOGY CO LTD melanoma tissue microarray
Melanoma Tissue Microarray, supplied by ZHUOLI IMAGING TECHNOLOGY CO LTD, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/melanoma+tissue+microarray/tissue+microarray/pm40396326-51-7-1
Average 90 stars, based on 1 article reviews
melanoma tissue microarray - by Bioz Stars, 2026-10
90/100 stars

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Microarray:

Article Title: MicroRNA-6084 orchestrates angiogenesis and liver metastasis in colorectal cancer via extracellular vesicles.
Article Snippet: .. 665 666 Tissue microarray 667 The tissue microarray was purchased from Zhuoli Biotechnology Co. Ltd (Shanghai, China). ..

Article Title: Hypoxia-induced circPLOD2a/b promotes the aggressiveness of glioblastoma by suppressing XIRP1 through binding to HuR
Article Snippet: .. A tissue microarray (TMA) containing grade I ( n = 3), II ( n = 50), III ( n = 24) and IV ( n = 94) glioma tissues was purchased from Zhuoli Biotechnology (ZL-BraGsur1801, Zhuoli Biotechnology Co., Shanghai, China). ..

Article Title: Integration of histopathological images and immunological analysis to predict M2 macrophage infiltration and prognosis in patients with serous ovarian cancer
Article Snippet: .. In addition, we used an SOC tissue microarray and obtained associated clinical features from Shanghai Zhuoli Biotech Company (Shanghai, China). ..

Article Title: Hypoxia-induced circPLOD2a/b promotes the aggressiveness of glioblastoma by suppressing XIRP1 through binding to HuR.
Article Snippet: .. A tissue microarray (TMA) containing grade I (n = 3), II (n = 50), III (n = 24) and IV (n = 94) glioma tissues was purchased from Zhuoli Biotechnology (ZL-BraGsur1801, Zhuoli BiotechnologyCo., Shanghai, China). ..

Article Title: USP41 plays carcinogenic roles in human cutaneous melanoma through PI3K/Akt signaling pathway.
Article Snippet: strong invasiveness, many patients experience tumor metastasis during the treatment process, which makes treatment difficult [4, 5].. Although emerging therapeutic approaches, such as targeted therapy, i.e. BRAF and MEK inhibitors, and immunotherapy, i.e. PD-1/PD-L1 monoclonal antibodies, have been increasingly used to treat metastatic melanoma, there are still many limitations, such as tumor resistance, low mutation rate of common targeted genes in some patients, and the treatment of advanced melanoma is still very difficult [6].. Therefore, it is particularly important to further study the pathogenesis of melanoma and explore new therapeutic targets.

Article Title: Inhibition of Sox4 Increases the Sensitivity of Drug-resistant Melanoma Cells to Cisplatin through the P38 Signaling Pathway.
Article Snippet: Background: SRY-Box Transcription Factor 4 (Sox4) has been found to be overexpressed in a number of malignancies and is linked to medication resistance.. The underlying mechanism of Sox4 in cisplatin-resistant melanomas, however, remains unknown.. Methods: Immunohistochemistry (IHC) was used to examine the expression of Sox4 in melanoma, pigmented nevi, and normal skin tissue.

Biomarker Discovery:

Article Title: CD38 contributes to tumor progression and tumor microenvironment reshaping in epithelial ovarian cancer
Article Snippet: .. Tissue microarrays comprising 18 normal ovary tissue samples and 99 primary ovarian cancer specimens were obtained from ZhuoLi Biotech (Shanghai, China) for the validation of CD38 expression patterns. .. Antibody against CD38 (#5100, 1:200, CST, USA).

Expressing:

Article Title: CD38 contributes to tumor progression and tumor microenvironment reshaping in epithelial ovarian cancer
Article Snippet: .. Tissue microarrays comprising 18 normal ovary tissue samples and 99 primary ovarian cancer specimens were obtained from ZhuoLi Biotech (Shanghai, China) for the validation of CD38 expression patterns. .. Antibody against CD38 (#5100, 1:200, CST, USA).

Article Title: USP41 plays carcinogenic roles in human cutaneous melanoma through PI3K/Akt signaling pathway.
Article Snippet: strong invasiveness, many patients experience tumor metastasis during the treatment process, which makes treatment difficult [4, 5].. Although emerging therapeutic approaches, such as targeted therapy, i.e. BRAF and MEK inhibitors, and immunotherapy, i.e. PD-1/PD-L1 monoclonal antibodies, have been increasingly used to treat metastatic melanoma, there are still many limitations, such as tumor resistance, low mutation rate of common targeted genes in some patients, and the treatment of advanced melanoma is still very difficult [6].. Therefore, it is particularly important to further study the pathogenesis of melanoma and explore new therapeutic targets.

other:

Article Title: Development and validation of a prognostic model for predicting survival and immunotherapy benefits in melanoma based on metabolism-relevant genes.
Article Snippet: Written informed consent was obtained from all patients for the acquisition of tissue material, the Ethics Committee of ShangHai Zhuoli Biotech (China) Co., Ltd. approved the development and application of tissue microarrays (ZLL-15-01).



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Image Search Results


Upregulation of MHC class II antigen in human melanoma xenografts treated systemically with anle138b, and expression of α-synuclein, MHC class II, and MHC class I proteins in melanoma TMA cores. (a) A tissue section from a nontreated WM983-B tumor xenograft probed with an antibody to human MHC class II (HLA-DPB1) protein (pseudocolored green). (b) A tissue section from an anle138b-treated WM983-B tumor xenograft probed with an antibody to human MHC class II (HLA-DPB1) protein (pseudocolored green). (c) Fluorescence intensity level of expression of proteins – α-synuclein (pseudocolored red), MHC II (pseudocolored yellow), MHC I (pseudocolored green) – in areas, encircled by a white line, in tissue sections from three of the Stage II and from one of the Stage IV melanoma TMA cores in which α-synuclein or vice versa, MHC II, or MHC I protein was expressed more strongly. TMA, tissue microarray.

Journal: Melanoma Research

Article Title: Interfering with aggregated α-synuclein in advanced melanoma leads to a major upregulation of MHC class II proteins

doi: 10.1097/CMR.0000000000000982

Figure Lengend Snippet: Upregulation of MHC class II antigen in human melanoma xenografts treated systemically with anle138b, and expression of α-synuclein, MHC class II, and MHC class I proteins in melanoma TMA cores. (a) A tissue section from a nontreated WM983-B tumor xenograft probed with an antibody to human MHC class II (HLA-DPB1) protein (pseudocolored green). (b) A tissue section from an anle138b-treated WM983-B tumor xenograft probed with an antibody to human MHC class II (HLA-DPB1) protein (pseudocolored green). (c) Fluorescence intensity level of expression of proteins – α-synuclein (pseudocolored red), MHC II (pseudocolored yellow), MHC I (pseudocolored green) – in areas, encircled by a white line, in tissue sections from three of the Stage II and from one of the Stage IV melanoma TMA cores in which α-synuclein or vice versa, MHC II, or MHC I protein was expressed more strongly. TMA, tissue microarray.

Article Snippet: Four melanoma tissue microarray (TMA) slides ( https://www.biomax.us/tissue-arrays/Melanoma/ME811 ), each containing two tissue sections from the same 39 cases of melanoma, and tissue sections from three cases of normal skin tissue were heated at 60 °C, deparaffinized and rehydrated.

Techniques: Expressing, Fluorescence, Microarray