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hypoxia chamber  (Baker Company)


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    Baker Company hypoxia chamber
    Hypoxia Chamber, supplied by Baker Company, used in various techniques. Bioz Stars score: 97/100, based on 1701 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/hypoxia+chamber/InvivO2/pm42190812-37-24-26
    Average 97 stars, based on 1701 article reviews
    hypoxia chamber - by Bioz Stars, 2026-09
    97/100 stars

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    Related Articles

    Incubation:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Control:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Clone Assay:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Microscopy:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Sequencing:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Transfection:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Confocal Microscopy:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Immunofluorescence:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Polymerase Chain Reaction:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Real-time Polymerase Chain Reaction:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Plasmid Preparation:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    cDNA Synthesis:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Injection:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Synthesized:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Cell Culture:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Concentration Assay:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Generated:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer

    Transformation Assay:

    Article Title: NLRP3 Regulation in Neonatal Hypoxic-Ischemic Encephalopathy-Focus on Microglial Activation.
    Article Snippet: 27177853 rity threshold of 90% was applied. The cells were subjected to oxygen–glucose deprivation (OGD) the following day. OGD was performed in a hypoxic chamber (0.1% O2, 5% CO2, 37 ◦C; InvivO2 400, Baker Ruskinn, Sanford, ME, USA). Cells were washed and incubated in glucose-free DMEM for 6 h (pilot experiments; cell death < 30%). The medium was then replaced with culture medium and the cells wer



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    Image Search Results


    ( A ) Oxygen levels (PO 2 (mmHg)) in cell culture media under control conditions and following exposure to hypoxia for 24 hr in the C-chamber hypoxia sub-chamber (Biospherix; unpaired t -test, p<0.0001); ( B ) Hierarchical clustering, using Ward’s criterion, of the first three principal components of the normalized gene expression profiles of all 17,777 genes passing quality control shows clear separation of 12 and 24 hr hypoxia-exposed samples versus control and from reoxygenated samples. Bar charts: mean ± s.e.m.

    Journal: eLife

    Article Title: Adrenomedullin restores the human cortical interneurons migration defects induced by hypoxia

    doi: 10.7554/eLife.108134

    Figure Lengend Snippet: ( A ) Oxygen levels (PO 2 (mmHg)) in cell culture media under control conditions and following exposure to hypoxia for 24 hr in the C-chamber hypoxia sub-chamber (Biospherix; unpaired t -test, p<0.0001); ( B ) Hierarchical clustering, using Ward’s criterion, of the first three principal components of the normalized gene expression profiles of all 17,777 genes passing quality control shows clear separation of 12 and 24 hr hypoxia-exposed samples versus control and from reoxygenated samples. Bar charts: mean ± s.e.m.

    Article Snippet: For these experiments, we induced hypoxia using the C-chamber hypoxia sub-chamber (Biospherix).

    Techniques: Cell Culture, Control, Gene Expression