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biotinylated rat anti cd5  (Cedarlane)


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    Cedarlane biotinylated rat anti cd5
    Biotinylated Rat Anti Cd5, supplied by Cedarlane, used in various techniques. Bioz Stars score: 93/100, based on 2 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/cd5/Anti-Rat+CD5+Biotin+(Clone+OX-19)+(mouse+IgG1)/pmc07032004-75-15-18
    Average 93 stars, based on 2 article reviews
    biotinylated rat anti cd5 - by Bioz Stars, 2026-09
    93/100 stars

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    Related Articles

    Incubation:

    Article Title: Antibody mediated cell labeling of peripheral T cells with micron sized iron oxide particles (MPIOs) allows single cell detection by MRI
    Article Snippet: Peripheral blood was harvested from adult Sprague-Dawley rats. .. Fresh, whole blood (5 × 10 6 T cells/ml) was incubated with various amounts of biotinylated rat anti-CD5 (Cedarlane Laboratories, Ontario, Canada), a pan T-cell antibody, for one hour, both on ice and at room temperature, and with and without shaking. ..



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    DNA methylation regulation of LGALS9 and its interaction with inflammatory cytokines in SD (A) LGALS9 locus plots reveal consistent genetic effects across SD GWAS, cis -mQTL, and cis -eQTL datasets ( p < 1 × 10−5). (B) Three-step SMR analysis demonstrated causal relationships between DNA methylation-mediated LGALS9 gene expressions and the onset of SD (P SMR multi < 0.05; HEIDI test p > 0.01, and Cochran’s Q p > 0.05). (C) Locus comparisons of LGALS9 gene expression and inflammatory cytokines (CD5, IL-10, and CST5) were performed via colocalization (PPH4 > 0.5). (D) ELISA validation of serum levels of LGALS9, CD5, IL-10, and CST5 in 16 patients with SD and 16 controls. Statistical analysis: SMR criteria as in . Data sources: eQTLGen and FinnGen R10. Colocalization: PPH4 > 0.5. ELISA: p = 0.0486 for LGALS9; ∗ p < 0.05, ∗∗ p < 0.01 versus controls. Boxplots show the median and IQR.

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    Figure Lengend Snippet: DNA methylation regulation of LGALS9 and its interaction with inflammatory cytokines in SD (A) LGALS9 locus plots reveal consistent genetic effects across SD GWAS, cis -mQTL, and cis -eQTL datasets ( p < 1 × 10−5). (B) Three-step SMR analysis demonstrated causal relationships between DNA methylation-mediated LGALS9 gene expressions and the onset of SD (P SMR multi < 0.05; HEIDI test p > 0.01, and Cochran’s Q p > 0.05). (C) Locus comparisons of LGALS9 gene expression and inflammatory cytokines (CD5, IL-10, and CST5) were performed via colocalization (PPH4 > 0.5). (D) ELISA validation of serum levels of LGALS9, CD5, IL-10, and CST5 in 16 patients with SD and 16 controls. Statistical analysis: SMR criteria as in . Data sources: eQTLGen and FinnGen R10. Colocalization: PPH4 > 0.5. ELISA: p = 0.0486 for LGALS9; ∗ p < 0.05, ∗∗ p < 0.01 versus controls. Boxplots show the median and IQR.

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    Techniques: DNA Methylation Assay, Gene Expression, Enzyme-linked Immunosorbent Assay, Biomarker Discovery