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cd5  (Miltenyi Biotec)


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    Miltenyi Biotec cd5
    Cd5, supplied by Miltenyi Biotec, used in various techniques. Bioz Stars score: 93/100, based on 14 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/cd5/CD5+Antibody%2C+anti-human%2C+REAfinity/pm42455847-67-85-87
    Average 93 stars, based on 14 article reviews
    cd5 - by Bioz Stars, 2026-10
    93/100 stars

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    Related Articles

    other:

    Article Title: Spatial proteogenomics reveals distinct and evolutionarily conserved hepatic macrophage niches
    Article Snippet: Anti-Human CD5 (REA782) PE , Miltenyi Biotec , 130-110-990; RRID: AB_2658593.

    Staining:

    Article Title: A novel Vδ1 engager targeting CD19 enhances human Vδ1 γδ T cell responses against CLL and CD19+ hematological malignancies.
    Article Snippet: γδ T cells are associated with favorable outcomes in many cancers likely through a mechanism of stress-directed cytotoxicity and antitumor cytokine production.. Vδ1 γδ T cells are especially promising for immunotherapy due to their broad stress recognition and resistance to activation-induced cell death.. Here, we generate a CD19 engager incorporating a novel Vδ1 binding moiety as proof-of-concept for treating CD19 cancers, including Chronic Lymphocytic Leukemia (CLL).

    Flow Cytometry:

    Article Title: A novel Vδ1 engager targeting CD19 enhances human Vδ1 γδ T cell responses against CLL and CD19+ hematological malignancies.
    Article Snippet: γδ T cells are associated with favorable outcomes in many cancers likely through a mechanism of stress-directed cytotoxicity and antitumor cytokine production.. Vδ1 γδ T cells are especially promising for immunotherapy due to their broad stress recognition and resistance to activation-induced cell death.. Here, we generate a CD19 engager incorporating a novel Vδ1 binding moiety as proof-of-concept for treating CD19 cancers, including Chronic Lymphocytic Leukemia (CLL).

    Article Title: Method for transduction of t cells in the presence of malignant cells
    Article Snippet: .. Malignant B cells were characterized by evaluation of surface markers using flow cytometry: CD45-Vioblue (Miltenyi Biotec, cat. No. 130-110-637), IgKappa-Viogreen (Miltenyi Biotec, cat. No. 130-122-224), IgLambda-APC-Vio (Miltenyi Biotec, cat. No. 130-105-106), CD5-PE (Miltenyi Biotec, cat. No. 130-110-991), CD10-APC (Miltenyi Biotec, cat. No. 130-114-503), CD20-PE-Vio (Miltenyi Biotec, cat. No. 130-111-340), CD34-PE (Miltenyi Biotec, cat. No. 130-120-515) and CD19-FITC (Miltenyi Biotec, cat. No. 130-113-645). ..



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    Image Search Results


    DNA methylation regulation of LGALS9 and its interaction with inflammatory cytokines in SD (A) LGALS9 locus plots reveal consistent genetic effects across SD GWAS, cis -mQTL, and cis -eQTL datasets ( p < 1 × 10−5). (B) Three-step SMR analysis demonstrated causal relationships between DNA methylation-mediated LGALS9 gene expressions and the onset of SD (P SMR multi < 0.05; HEIDI test p > 0.01, and Cochran’s Q p > 0.05). (C) Locus comparisons of LGALS9 gene expression and inflammatory cytokines (CD5, IL-10, and CST5) were performed via colocalization (PPH4 > 0.5). (D) ELISA validation of serum levels of LGALS9, CD5, IL-10, and CST5 in 16 patients with SD and 16 controls. Statistical analysis: SMR criteria as in . Data sources: eQTLGen and FinnGen R10. Colocalization: PPH4 > 0.5. ELISA: p = 0.0486 for LGALS9; ∗ p < 0.05, ∗∗ p < 0.01 versus controls. Boxplots show the median and IQR.

    Journal: iScience

    Article Title: Interferon signaling gene expression, and DNA methylation interactions in Sjögren’s disease via mendelian randomization

    doi: 10.1016/j.isci.2026.114950

    Figure Lengend Snippet: DNA methylation regulation of LGALS9 and its interaction with inflammatory cytokines in SD (A) LGALS9 locus plots reveal consistent genetic effects across SD GWAS, cis -mQTL, and cis -eQTL datasets ( p < 1 × 10−5). (B) Three-step SMR analysis demonstrated causal relationships between DNA methylation-mediated LGALS9 gene expressions and the onset of SD (P SMR multi < 0.05; HEIDI test p > 0.01, and Cochran’s Q p > 0.05). (C) Locus comparisons of LGALS9 gene expression and inflammatory cytokines (CD5, IL-10, and CST5) were performed via colocalization (PPH4 > 0.5). (D) ELISA validation of serum levels of LGALS9, CD5, IL-10, and CST5 in 16 patients with SD and 16 controls. Statistical analysis: SMR criteria as in . Data sources: eQTLGen and FinnGen R10. Colocalization: PPH4 > 0.5. ELISA: p = 0.0486 for LGALS9; ∗ p < 0.05, ∗∗ p < 0.01 versus controls. Boxplots show the median and IQR.

    Article Snippet: Galectin-9 regulates B cell activation by increasing colocalization with the inhibitory co-receptor CD5, thereby modulating TLR4 signal transduction.

    Techniques: DNA Methylation Assay, Gene Expression, Enzyme-linked Immunosorbent Assay, Biomarker Discovery