p38mapk (Cell Signaling Technology Inc)
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Cell Signaling Technology Inc
p38mapk
P38mapk, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 99/100, based on 30335 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/p38mapk/p38+MAPK+Antibody/pm42863182-43-5-15
Average 99 stars, based on 30335 article reviews
P38mapk, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 99/100, based on 30335 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/p38mapk/p38+MAPK+Antibody/pm42863182-43-5-15
Average 99 stars, based on 30335 article reviews
p38mapk - by Bioz Stars,
2026-11
99/100 stars
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other:Article Title: Araliadiol Protects Human Keratinocytes From Oxidative Stress, DNA Damage, and Apoptosis via Activation of Antioxidant Signaling Article Snippet: Primary antibodies specific for Nrf2 (sc-365949), HO-1 (sc-390991), and β-actin (sc-47778) were purchased from Santa Cruz Biotechnology, Inc. (Dallas, TX, USA). Article Title: Apoptosis and motor deficits in SPG76 hereditary spastic paraplegia: Calpain 2 inhibition as therapeutic strategy. Article Snippet: Anti LC3B (2775), phospho-Akt (4058), Akt (9272), Drosophila phospho-S505-Akt (4060), phospho-ERK1/2 (4370), ERK1/2 (4695), phospho-p38MAPK (9216), p38MAPK (8690), phospho-GSK3β-Ser9 (5558), GSK3β (9315), phospho-mTor (2971), mTor (2983), calpain 1 (2556), calpain 2 (2539), cleaved caspase 7 (9491), cleaved caspase 3 (9661), cleaved caspase 9 (9505), SDS Page:Article Title: GnIH-Induced Mitochondrial Dysfunction Lead to Oxidative Stress and Apoptosis in Thyroid Follicular Cells, Causing Hypothyroidism. Article Snippet: Thyroid disorders profoundly disrupt metabolism, development, growth, pubertal timing, and fertility in domestic animals.. Gonadotropin-inhibitory hormone (GnIH), a key inhibitory neuropeptide regulating reproductive function, has been implicated in metabolic dysfunction-associated infertility as well as thyroid dysfunction–related pubertal abnormalities.. These observations suggest potential crosstalk between GnIH and thyroid hormones (THs), positioning GnIH as a possible integrative regulator linking the hypothalamic-pituitary-thyroid (HPT) and hypothalamic-pituitary-gonadal (HPG) axes. Membrane:Article Title: GnIH-Induced Mitochondrial Dysfunction Lead to Oxidative Stress and Apoptosis in Thyroid Follicular Cells, Causing Hypothyroidism. Article Snippet: Thyroid disorders profoundly disrupt metabolism, development, growth, pubertal timing, and fertility in domestic animals.. Gonadotropin-inhibitory hormone (GnIH), a key inhibitory neuropeptide regulating reproductive function, has been implicated in metabolic dysfunction-associated infertility as well as thyroid dysfunction–related pubertal abnormalities.. These observations suggest potential crosstalk between GnIH and thyroid hormones (THs), positioning GnIH as a possible integrative regulator linking the hypothalamic-pituitary-thyroid (HPT) and hypothalamic-pituitary-gonadal (HPG) axes. Incubation:Article Title: GnIH-Induced Mitochondrial Dysfunction Lead to Oxidative Stress and Apoptosis in Thyroid Follicular Cells, Causing Hypothyroidism. Article Snippet: Thyroid disorders profoundly disrupt metabolism, development, growth, pubertal timing, and fertility in domestic animals.. Gonadotropin-inhibitory hormone (GnIH), a key inhibitory neuropeptide regulating reproductive function, has been implicated in metabolic dysfunction-associated infertility as well as thyroid dysfunction–related pubertal abnormalities.. These observations suggest potential crosstalk between GnIH and thyroid hormones (THs), positioning GnIH as a possible integrative regulator linking the hypothalamic-pituitary-thyroid (HPT) and hypothalamic-pituitary-gonadal (HPG) axes. Article Title: Multifunctional Biomimetic Hematoma Microspheres for Sustained Local Hydrogen Sulfide (H 2 S) Delivery to Enhance Vascularized Bone Regeneration. Article Snippet: While conventional large bone defect healing is a complicated process withmultiple physiological limitations, our approach leverages hematoma’s innate regenerative capabilities-particularly its structure and biological functions, inculding multi-functional regulation response, activating cell autophagy, resisting persistent inflammation and enhancing angiogenesis/osteogenesis eventually, which supports a new strategy to bone repair solutions.. This biomimetic strategy ismore compatible with the process of bone regeneration, thereby shifting the implant paradigm from passive structural support to active biological regulation.. Inspired by the balloon structure, we developed microfluidic biomimetic hematoma microspheres featuring: pH-responsive GelMA outer layer and NaHS-loaded liposomal core. Control:Article Title: GnIH-Induced Mitochondrial Dysfunction Lead to Oxidative Stress and Apoptosis in Thyroid Follicular Cells, Causing Hypothyroidism. Article Snippet: Thyroid disorders profoundly disrupt metabolism, development, growth, pubertal timing, and fertility in domestic animals.. Gonadotropin-inhibitory hormone (GnIH), a key inhibitory neuropeptide regulating reproductive function, has been implicated in metabolic dysfunction-associated infertility as well as thyroid dysfunction–related pubertal abnormalities.. These observations suggest potential crosstalk between GnIH and thyroid hormones (THs), positioning GnIH as a possible integrative regulator linking the hypothalamic-pituitary-thyroid (HPT) and hypothalamic-pituitary-gonadal (HPG) axes. Blocking Assay:Article Title: Multifunctional Biomimetic Hematoma Microspheres for Sustained Local Hydrogen Sulfide (H 2 S) Delivery to Enhance Vascularized Bone Regeneration. Article Snippet: While conventional large bone defect healing is a complicated process withmultiple physiological limitations, our approach leverages hematoma’s innate regenerative capabilities-particularly its structure and biological functions, inculding multi-functional regulation response, activating cell autophagy, resisting persistent inflammation and enhancing angiogenesis/osteogenesis eventually, which supports a new strategy to bone repair solutions.. This biomimetic strategy ismore compatible with the process of bone regeneration, thereby shifting the implant paradigm from passive structural support to active biological regulation.. Inspired by the balloon structure, we developed microfluidic biomimetic hematoma microspheres featuring: pH-responsive GelMA outer layer and NaHS-loaded liposomal core. Stripping Membranes:Article Title: Multifunctional Biomimetic Hematoma Microspheres for Sustained Local Hydrogen Sulfide (H 2 S) Delivery to Enhance Vascularized Bone Regeneration. Article Snippet: While conventional large bone defect healing is a complicated process withmultiple physiological limitations, our approach leverages hematoma’s innate regenerative capabilities-particularly its structure and biological functions, inculding multi-functional regulation response, activating cell autophagy, resisting persistent inflammation and enhancing angiogenesis/osteogenesis eventually, which supports a new strategy to bone repair solutions.. This biomimetic strategy ismore compatible with the process of bone regeneration, thereby shifting the implant paradigm from passive structural support to active biological regulation.. Inspired by the balloon structure, we developed microfluidic biomimetic hematoma microspheres featuring: pH-responsive GelMA outer layer and NaHS-loaded liposomal core. Western Blot:Article Title: Curcumin loaded solid lipid nanoparticles ameliorates poly (I:C)-induced exacerbation of allergic asthma. Article Snippet: Virus-mediated asthma exacerbations represent a major clinical burden, characterized by neutrophilic inflammation, airway hyperresponsiveness, and oxidative stress leading to the worsening of asthma symptoms.. Recently, we have reported that poly (I:C)-mediated asthma exacerbation is unresponsive to dexamethasone (corticosteroid class of drugs) underscoring the need for alternative therapeutic strategy.. Given the wellestablished anti-inflammatory and antioxidant potential of curcumin, the present study aimed at investigating the protective potential of curcumin loaded solid lipid nanoparticles (SLNs) against poly (I:C) mediated exacerbation of allergic asthma in mice and SLN preparation and characterization. |
