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p21 boster  (Boster Bio)


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    Structured Review

    Boster Bio p21 boster
    Figure 4: Expression of p-ATM, p53, <t>p21</t> and PCNA protein in the jejunum at 14 days of age (Immunohistochemistry, scale bar: 50μm).
    P21 Boster, supplied by Boster Bio, used in various techniques. Bioz Stars score: 92/100, based on 28 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/p21 boster/product/Boster Bio
    Average 92 stars, based on 28 article reviews
    p21 boster - by Bioz Stars, 2026-03
    92/100 stars

    Images

    1) Product Images from "The molecular mechanism of G2M cell cycle arrest induced by AFB1 in the jejunum."

    Article Title: The molecular mechanism of G2M cell cycle arrest induced by AFB1 in the jejunum.

    Journal: Oncotarget

    doi: 10.18632/oncotarget.9594

    Figure 4: Expression of p-ATM, p53, p21 and PCNA protein in the jejunum at 14 days of age (Immunohistochemistry, scale bar: 50μm).
    Figure Legend Snippet: Figure 4: Expression of p-ATM, p53, p21 and PCNA protein in the jejunum at 14 days of age (Immunohistochemistry, scale bar: 50μm).

    Techniques Used: Expressing, Immunohistochemistry

    Figure 6: Integrated optical density (IOD) of p-ATM, p53, p21, PCNA, p-Chk2, p-cdc25C and CyclinB protein expression in the jejunum. Note: Data are presented with the mean ± standard deviation (n = 6).*p < 0.05, **p < 0.01 compared with the control group.
    Figure Legend Snippet: Figure 6: Integrated optical density (IOD) of p-ATM, p53, p21, PCNA, p-Chk2, p-cdc25C and CyclinB protein expression in the jejunum. Note: Data are presented with the mean ± standard deviation (n = 6).*p < 0.05, **p < 0.01 compared with the control group.

    Techniques Used: Expressing, Standard Deviation, Control

    Figure 7: The levels of the ATM, Chk2, cdc25, CyclinB3, cdc2, mdm2, p53, p21 and PCNA mRNA expression in the jejunal mucosa. Note: Data are presented with the mean ± standard deviation (n = 6).*p < 0.05, **p < 0.01 compared with the control group.
    Figure Legend Snippet: Figure 7: The levels of the ATM, Chk2, cdc25, CyclinB3, cdc2, mdm2, p53, p21 and PCNA mRNA expression in the jejunal mucosa. Note: Data are presented with the mean ± standard deviation (n = 6).*p < 0.05, **p < 0.01 compared with the control group.

    Techniques Used: Expressing, Standard Deviation, Control



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    Figure 4: Expression of p-ATM, p53, <t>p21</t> and PCNA protein in the jejunum at 14 days of age (Immunohistochemistry, scale bar: 50μm).
    P21 Boster, supplied by Boster Bio, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Image Search Results


    Figure 4: Expression of p-ATM, p53, p21 and PCNA protein in the jejunum at 14 days of age (Immunohistochemistry, scale bar: 50μm).

    Journal: Oncotarget

    Article Title: The molecular mechanism of G2M cell cycle arrest induced by AFB1 in the jejunum.

    doi: 10.18632/oncotarget.9594

    Figure Lengend Snippet: Figure 4: Expression of p-ATM, p53, p21 and PCNA protein in the jejunum at 14 days of age (Immunohistochemistry, scale bar: 50μm).

    Article Snippet: Table 1: Antibodies used in immunohistochemistry Name Company Cat# Source Dilution p-ATM Bioss, China bs-2272R Rabbit 1:100 p-Chk2 Bioss, China bs-3721R Rabbit 1:100 p53 Boster, China BM0101 Mouse 1:100 p21 Boster, China BA0272 Rabbit 1:100 p-cdc25C Bioss, China bs-3482R Rabbit 1:100 Cyclin B1 Boster, China BA0766 Rabbit 1:100 PCNA Bioss, China bs-0754R Rabbit 1:100 Oncotarget35603www.impactjournals.com/oncotarget After centrifugation (200×g, 5 min), the supernatant was separated, the cells were incubated for 30 min at room temperature in the dark with 5 μL 0.25% Triton X-100 and 5 μL Propidium Iodide (PI) (Cat. No.51-66211E).

    Techniques: Expressing, Immunohistochemistry

    Figure 6: Integrated optical density (IOD) of p-ATM, p53, p21, PCNA, p-Chk2, p-cdc25C and CyclinB protein expression in the jejunum. Note: Data are presented with the mean ± standard deviation (n = 6).*p < 0.05, **p < 0.01 compared with the control group.

    Journal: Oncotarget

    Article Title: The molecular mechanism of G2M cell cycle arrest induced by AFB1 in the jejunum.

    doi: 10.18632/oncotarget.9594

    Figure Lengend Snippet: Figure 6: Integrated optical density (IOD) of p-ATM, p53, p21, PCNA, p-Chk2, p-cdc25C and CyclinB protein expression in the jejunum. Note: Data are presented with the mean ± standard deviation (n = 6).*p < 0.05, **p < 0.01 compared with the control group.

    Article Snippet: Table 1: Antibodies used in immunohistochemistry Name Company Cat# Source Dilution p-ATM Bioss, China bs-2272R Rabbit 1:100 p-Chk2 Bioss, China bs-3721R Rabbit 1:100 p53 Boster, China BM0101 Mouse 1:100 p21 Boster, China BA0272 Rabbit 1:100 p-cdc25C Bioss, China bs-3482R Rabbit 1:100 Cyclin B1 Boster, China BA0766 Rabbit 1:100 PCNA Bioss, China bs-0754R Rabbit 1:100 Oncotarget35603www.impactjournals.com/oncotarget After centrifugation (200×g, 5 min), the supernatant was separated, the cells were incubated for 30 min at room temperature in the dark with 5 μL 0.25% Triton X-100 and 5 μL Propidium Iodide (PI) (Cat. No.51-66211E).

    Techniques: Expressing, Standard Deviation, Control

    Figure 7: The levels of the ATM, Chk2, cdc25, CyclinB3, cdc2, mdm2, p53, p21 and PCNA mRNA expression in the jejunal mucosa. Note: Data are presented with the mean ± standard deviation (n = 6).*p < 0.05, **p < 0.01 compared with the control group.

    Journal: Oncotarget

    Article Title: The molecular mechanism of G2M cell cycle arrest induced by AFB1 in the jejunum.

    doi: 10.18632/oncotarget.9594

    Figure Lengend Snippet: Figure 7: The levels of the ATM, Chk2, cdc25, CyclinB3, cdc2, mdm2, p53, p21 and PCNA mRNA expression in the jejunal mucosa. Note: Data are presented with the mean ± standard deviation (n = 6).*p < 0.05, **p < 0.01 compared with the control group.

    Article Snippet: Table 1: Antibodies used in immunohistochemistry Name Company Cat# Source Dilution p-ATM Bioss, China bs-2272R Rabbit 1:100 p-Chk2 Bioss, China bs-3721R Rabbit 1:100 p53 Boster, China BM0101 Mouse 1:100 p21 Boster, China BA0272 Rabbit 1:100 p-cdc25C Bioss, China bs-3482R Rabbit 1:100 Cyclin B1 Boster, China BA0766 Rabbit 1:100 PCNA Bioss, China bs-0754R Rabbit 1:100 Oncotarget35603www.impactjournals.com/oncotarget After centrifugation (200×g, 5 min), the supernatant was separated, the cells were incubated for 30 min at room temperature in the dark with 5 μL 0.25% Triton X-100 and 5 μL Propidium Iodide (PI) (Cat. No.51-66211E).

    Techniques: Expressing, Standard Deviation, Control

    Antibodies Used in the Study.

    Journal: Journal of Histochemistry and Cytochemistry

    Article Title: Euptox A Induces G1 Arrest and Autophagy via p38 MAPK- and PI3K/Akt/mTOR-Mediated Pathways in Mouse Splenocytes

    doi: 10.1369/0022155417722118

    Figure Lengend Snippet: Antibodies Used in the Study.

    Article Snippet: No Source CDK2 Rabbit, polyclonal PB0562 Boster phospho(p)-CDK2 Rabbit, polyclonal BM4590 Boster cyclin D1 Rabbit, polyclonal BA0770 Boster p-cyclin D1 Rabbit, polyclonal BM4272 Boster p53 Rabbit, polyclonal {"type":"entrez-nucleotide","attrs":{"text":"A00001","term_id":"58418","term_text":"A00001"}} A00001 Boster p21 Waf1/Cip1 Rabbit, polyclonal BM4382 Boster Bcl-2 Rabbit, polyclonal D17C4 Cell Signaling Technology Bcl-xl Rabbit, polyclonal 54H6 Cell Signaling Technology p-PI3K Rabbit, polyclonal 4228S Cell Signaling Technology PI3K Rabbit, polyclonal 4249S Cell Signaling Technology p-p38 MAPK Rabbit, polyclonal 8690S Cell Signaling Technology p38 MAPK Rabbit, polyclonal 7257s Cell Signaling Technology p-Akt (Ser473) Rabbit, polyclonal 4060S Cell Signaling Technology Akt Rabbit, polyclonal 4691S Cell Signaling Technology p-mTOR Rabbit, polyclonal 5536S Cell Signaling Technology mTOR Rabbit, polyclonal 2983S Cell Signaling Technology Beclin 1 Rabbit, polyclonal 3495S Cell Signaling Technology LC3-I/II Rabbit, polyclonal 12741S Cell Signaling Technology p62 Rabbit, polyclonal 8025S Cell Signaling Technology PTEN Rabbit, polyclonal 9188S Cell Signaling Technology p-AMPK Rabbit, polyclonal 2537S Cell Signaling Technology AMPK Rabbit, polyclonal 4150S Cell Signaling Technology 4EBP1 Rabbit, polyclonal 9644S Cell Signaling Technology p-4EBP Rabbit, monoclonal 2855S Cell Signaling Technology p-P70S6K Rabbit, monoclonal 9204S Cell Signaling Technology Open in a separate window Antibodies Used in the Study.

    Techniques:

    Effect of Euptox A on the expression levels of key regulators of cell cycle in the splenocytes. Splenocytes were treated with Euptox A at concentrations of 200, 400, and 800 mg/kg/day for 30 days and then protein samples of cells were subjected to a western blotting assay. (A) Representative blots for CDK4, CDK2, cyclin D1, p53, p27 Kip1, p21 Waf1/Cip1, Chk1, PCNA, and E2F1, and (B) bar graphs showing the relative expression level of CDK4, CDK2, cyclin D1, p53, p27 Kip1, p21 Waf1/Cip1, Chk1, PCNA, and E2F1 in the splenocytes. All data are presented with the means ± SD and mean values of three independent experiments. *p<0.05, compared with the control group; **p<0.01, compared with the control group.

    Journal: Journal of Histochemistry and Cytochemistry

    Article Title: Euptox A Induces G1 Arrest and Autophagy via p38 MAPK- and PI3K/Akt/mTOR-Mediated Pathways in Mouse Splenocytes

    doi: 10.1369/0022155417722118

    Figure Lengend Snippet: Effect of Euptox A on the expression levels of key regulators of cell cycle in the splenocytes. Splenocytes were treated with Euptox A at concentrations of 200, 400, and 800 mg/kg/day for 30 days and then protein samples of cells were subjected to a western blotting assay. (A) Representative blots for CDK4, CDK2, cyclin D1, p53, p27 Kip1, p21 Waf1/Cip1, Chk1, PCNA, and E2F1, and (B) bar graphs showing the relative expression level of CDK4, CDK2, cyclin D1, p53, p27 Kip1, p21 Waf1/Cip1, Chk1, PCNA, and E2F1 in the splenocytes. All data are presented with the means ± SD and mean values of three independent experiments. *p<0.05, compared with the control group; **p<0.01, compared with the control group.

    Article Snippet: No Source CDK2 Rabbit, polyclonal PB0562 Boster phospho(p)-CDK2 Rabbit, polyclonal BM4590 Boster cyclin D1 Rabbit, polyclonal BA0770 Boster p-cyclin D1 Rabbit, polyclonal BM4272 Boster p53 Rabbit, polyclonal {"type":"entrez-nucleotide","attrs":{"text":"A00001","term_id":"58418","term_text":"A00001"}} A00001 Boster p21 Waf1/Cip1 Rabbit, polyclonal BM4382 Boster Bcl-2 Rabbit, polyclonal D17C4 Cell Signaling Technology Bcl-xl Rabbit, polyclonal 54H6 Cell Signaling Technology p-PI3K Rabbit, polyclonal 4228S Cell Signaling Technology PI3K Rabbit, polyclonal 4249S Cell Signaling Technology p-p38 MAPK Rabbit, polyclonal 8690S Cell Signaling Technology p38 MAPK Rabbit, polyclonal 7257s Cell Signaling Technology p-Akt (Ser473) Rabbit, polyclonal 4060S Cell Signaling Technology Akt Rabbit, polyclonal 4691S Cell Signaling Technology p-mTOR Rabbit, polyclonal 5536S Cell Signaling Technology mTOR Rabbit, polyclonal 2983S Cell Signaling Technology Beclin 1 Rabbit, polyclonal 3495S Cell Signaling Technology LC3-I/II Rabbit, polyclonal 12741S Cell Signaling Technology p62 Rabbit, polyclonal 8025S Cell Signaling Technology PTEN Rabbit, polyclonal 9188S Cell Signaling Technology p-AMPK Rabbit, polyclonal 2537S Cell Signaling Technology AMPK Rabbit, polyclonal 4150S Cell Signaling Technology 4EBP1 Rabbit, polyclonal 9644S Cell Signaling Technology p-4EBP Rabbit, monoclonal 2855S Cell Signaling Technology p-P70S6K Rabbit, monoclonal 9204S Cell Signaling Technology Open in a separate window Antibodies Used in the Study.

    Techniques: Expressing, Western Blot