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normal esophageal epithelial cells heec  (Procell Inc)

 
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    Procell Inc normal esophageal epithelial cells heec
    MMP12 was identified as a key gene and its expression was upregulated in EC cells. (A) Expression analysis of INHBA, MMP3, MMP12, and SPP1 in EC tissues and normal <t>esophageal</t> tissues through the GEPIA database. (B) Expression analysis of INHBA, MMP3, MMP12, and SPP1 in <t>HEEC</t> and KYSE150 cells. (C) MMP12 expression analysis in pan-cancer tissues and normal tissues through the GEPIA database. (D) qRT-PCR was performed to detect MMP12 protein expression in HEEC and KYSE150 cells. (E) The efficiency of MMP12 knockdown was determined by Western blotting in KYSE150 cells. ns: not significant, ∗ P < 0.05, ∗∗ P < 0.01 and ∗∗∗ P < 0.001.
    Normal Esophageal Epithelial Cells Heec, supplied by Procell Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/normal esophageal epithelial cells heec/product/Procell Inc
    Average 86 stars, based on 1 article reviews
    normal esophageal epithelial cells heec - by Bioz Stars, 2026-05
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    1) Product Images from "WTAP stabilizes MMP12 expression to promote the malignant phenotypes of esophageal cancer cells"

    Article Title: WTAP stabilizes MMP12 expression to promote the malignant phenotypes of esophageal cancer cells

    Journal: Regenerative Therapy

    doi: 10.1016/j.reth.2026.101101

    MMP12 was identified as a key gene and its expression was upregulated in EC cells. (A) Expression analysis of INHBA, MMP3, MMP12, and SPP1 in EC tissues and normal esophageal tissues through the GEPIA database. (B) Expression analysis of INHBA, MMP3, MMP12, and SPP1 in HEEC and KYSE150 cells. (C) MMP12 expression analysis in pan-cancer tissues and normal tissues through the GEPIA database. (D) qRT-PCR was performed to detect MMP12 protein expression in HEEC and KYSE150 cells. (E) The efficiency of MMP12 knockdown was determined by Western blotting in KYSE150 cells. ns: not significant, ∗ P < 0.05, ∗∗ P < 0.01 and ∗∗∗ P < 0.001.
    Figure Legend Snippet: MMP12 was identified as a key gene and its expression was upregulated in EC cells. (A) Expression analysis of INHBA, MMP3, MMP12, and SPP1 in EC tissues and normal esophageal tissues through the GEPIA database. (B) Expression analysis of INHBA, MMP3, MMP12, and SPP1 in HEEC and KYSE150 cells. (C) MMP12 expression analysis in pan-cancer tissues and normal tissues through the GEPIA database. (D) qRT-PCR was performed to detect MMP12 protein expression in HEEC and KYSE150 cells. (E) The efficiency of MMP12 knockdown was determined by Western blotting in KYSE150 cells. ns: not significant, ∗ P < 0.05, ∗∗ P < 0.01 and ∗∗∗ P < 0.001.

    Techniques Used: Expressing, Quantitative RT-PCR, Knockdown, Western Blot



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    Image Search Results


    MMP12 was identified as a key gene and its expression was upregulated in EC cells. (A) Expression analysis of INHBA, MMP3, MMP12, and SPP1 in EC tissues and normal esophageal tissues through the GEPIA database. (B) Expression analysis of INHBA, MMP3, MMP12, and SPP1 in HEEC and KYSE150 cells. (C) MMP12 expression analysis in pan-cancer tissues and normal tissues through the GEPIA database. (D) qRT-PCR was performed to detect MMP12 protein expression in HEEC and KYSE150 cells. (E) The efficiency of MMP12 knockdown was determined by Western blotting in KYSE150 cells. ns: not significant, ∗ P < 0.05, ∗∗ P < 0.01 and ∗∗∗ P < 0.001.

    Journal: Regenerative Therapy

    Article Title: WTAP stabilizes MMP12 expression to promote the malignant phenotypes of esophageal cancer cells

    doi: 10.1016/j.reth.2026.101101

    Figure Lengend Snippet: MMP12 was identified as a key gene and its expression was upregulated in EC cells. (A) Expression analysis of INHBA, MMP3, MMP12, and SPP1 in EC tissues and normal esophageal tissues through the GEPIA database. (B) Expression analysis of INHBA, MMP3, MMP12, and SPP1 in HEEC and KYSE150 cells. (C) MMP12 expression analysis in pan-cancer tissues and normal tissues through the GEPIA database. (D) qRT-PCR was performed to detect MMP12 protein expression in HEEC and KYSE150 cells. (E) The efficiency of MMP12 knockdown was determined by Western blotting in KYSE150 cells. ns: not significant, ∗ P < 0.05, ∗∗ P < 0.01 and ∗∗∗ P < 0.001.

    Article Snippet: EC cell (KYSE-150 and TE-10), normal esophageal epithelial cells (HEEC) and THP-1 cells were provided by Procell (Wuhan, China).

    Techniques: Expressing, Quantitative RT-PCR, Knockdown, Western Blot

    Expression of GNL3L in esophageal cancer (EC) and the cell line model of GNL3L knockdown. (a) GNL3L expression was analyzed in EC tumor tissues ( n = 182) and normal esophageal tissues ( n = 286) using the GEPIA database. (b) Kaplan–Meier analysis of survival rates between patients with EC and low expression of GNL3L ( n = 138) vs. high expression of GNL3L ( n = 46). In addition to the expression of GNL3L , the survival rate was also related to tumor grade. The patient cohort was obtained from the gene information of TCGA Esophageal cancer datasets downloaded from the UALCAN website. (c) Immunohistochemistry was performed to detect differential expression of GNL3L protein in an esophageal squamous cell carcinoma (ESCC) specimen. (d) qRT‐PCR was used to detect GNL3L expression in ESCC cell lines (TE‐1, KYSE‐410, KYSE‐30, and EC9706) and human normal esophageal epithelial cell line (HEEC). (e) The expression of GNL3L protein in ESCC cell lines and HEEC was assessed using western blotting (WB). (f) Efficiency of sh‐GNL3L for GNL3L in TE‐1 cells by qRT‐PCR. (g) The protein expression of GNL3L in TE‐1 cells. (NC: Negative control). (* p < 0.05, ** p < 0.01).

    Journal: Cancer Medicine

    Article Title: The GNL3L ‐ MDM2 Interaction Drives Esophageal Squamous Cell Carcinoma Progression

    doi: 10.1002/cam4.71146

    Figure Lengend Snippet: Expression of GNL3L in esophageal cancer (EC) and the cell line model of GNL3L knockdown. (a) GNL3L expression was analyzed in EC tumor tissues ( n = 182) and normal esophageal tissues ( n = 286) using the GEPIA database. (b) Kaplan–Meier analysis of survival rates between patients with EC and low expression of GNL3L ( n = 138) vs. high expression of GNL3L ( n = 46). In addition to the expression of GNL3L , the survival rate was also related to tumor grade. The patient cohort was obtained from the gene information of TCGA Esophageal cancer datasets downloaded from the UALCAN website. (c) Immunohistochemistry was performed to detect differential expression of GNL3L protein in an esophageal squamous cell carcinoma (ESCC) specimen. (d) qRT‐PCR was used to detect GNL3L expression in ESCC cell lines (TE‐1, KYSE‐410, KYSE‐30, and EC9706) and human normal esophageal epithelial cell line (HEEC). (e) The expression of GNL3L protein in ESCC cell lines and HEEC was assessed using western blotting (WB). (f) Efficiency of sh‐GNL3L for GNL3L in TE‐1 cells by qRT‐PCR. (g) The protein expression of GNL3L in TE‐1 cells. (NC: Negative control). (* p < 0.05, ** p < 0.01).

    Article Snippet: ESCC (TE‐1, KYSE‐410, KYSE‐30, EC9706, and ECA‐109) and human normal esophageal epithelial (HEEC) cell lines were obtained from Procell Life Science &Technology Co. Ltd. (Wuhan, China).

    Techniques: Expressing, Knockdown, Immunohistochemistry, Quantitative Proteomics, Quantitative RT-PCR, Western Blot, Negative Control