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gc tissue microarray (tma) cohort  (Human Protein Atlas)

 
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    Human Protein Atlas gc tissue microarray (tma) cohort
    Gc Tissue Microarray (Tma) Cohort, supplied by Human Protein Atlas, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/gc+tissue+microarray/gc+tissue+microarray++tma++cohort/pm39009956-94-6-12
    Average 90 stars, based on 1 article reviews
    gc tissue microarray (tma) cohort - by Bioz Stars, 2026-09
    90/100 stars

    Images

    Related Articles

    Immunohistochemistry:

    Article Title: The regulatory mechanism of m6A modification in gastric cancer.
    Article Snippet: Validated by IHC staining analysis from GC tissue microarray (TMA) cohort and Human Protein Atlas (HPA) database, YTHDF1/2/3 and hnRNPA2B1 of m6A “readers” were overexpressed at protein and gene expression level in GC, while HNRNPA2B1 and YTHDC1 had no difference in the expression level in GC [32].

    Staining:

    Article Title: The regulatory mechanism of m6A modification in gastric cancer.
    Article Snippet: Validated by IHC staining analysis from GC tissue microarray (TMA) cohort and Human Protein Atlas (HPA) database, YTHDF1/2/3 and hnRNPA2B1 of m6A “readers” were overexpressed at protein and gene expression level in GC, while HNRNPA2B1 and YTHDC1 had no difference in the expression level in GC [32].

    Microarray:

    Article Title: The regulatory mechanism of m6A modification in gastric cancer.
    Article Snippet: Validated by IHC staining analysis from GC tissue microarray (TMA) cohort and Human Protein Atlas (HPA) database, YTHDF1/2/3 and hnRNPA2B1 of m6A “readers” were overexpressed at protein and gene expression level in GC, while HNRNPA2B1 and YTHDC1 had no difference in the expression level in GC [32].

    Gene Expression:

    Article Title: The regulatory mechanism of m6A modification in gastric cancer.
    Article Snippet: Validated by IHC staining analysis from GC tissue microarray (TMA) cohort and Human Protein Atlas (HPA) database, YTHDF1/2/3 and hnRNPA2B1 of m6A “readers” were overexpressed at protein and gene expression level in GC, while HNRNPA2B1 and YTHDC1 had no difference in the expression level in GC [32].

    Expressing:

    Article Title: The regulatory mechanism of m6A modification in gastric cancer.
    Article Snippet: Validated by IHC staining analysis from GC tissue microarray (TMA) cohort and Human Protein Atlas (HPA) database, YTHDF1/2/3 and hnRNPA2B1 of m6A “readers” were overexpressed at protein and gene expression level in GC, while HNRNPA2B1 and YTHDC1 had no difference in the expression level in GC [32].



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    PIK3CB and SP1 were overexpressed in GC samples. (A) The top 10 genes co-expressed with PIK3CB in TCGA-STAD datasets were shown. PCC: Pearson correlation coefficient. (B) The expression relevance between PIK3CB and SP1 was predicted by GEPIA online tools. (C) PIK3CB and SP1 were upregulated in GC tissue samples form TCGA database using GEPIA. (D) Kaplan-Meier curves for patients with different expression levels of PIK3CB or SP1 via Kaplan-Meier plotter database. (E) qRT-PCR analysis of PIK3CB and SP1 expression in 40 paired GC tissue and adjacent normal tissues. C/N score ≥1.5 was viewed as upregulated. (F) The assessment of correlation between PIK3CB and SP1 expression in 40 paired GC tissues using linear regression. (G) PIK3CB and SP1 expression level were evaluated by immunohistochemistry staining on a GC tissue <t>microarray.</t> Representative staining images and expression correlation of PI3KCB and SP1 in tumor tissue were shown. Scale bar: 50 μm. *P < 0.01.
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    Image Search Results


    PIK3CB and SP1 were overexpressed in GC samples. (A) The top 10 genes co-expressed with PIK3CB in TCGA-STAD datasets were shown. PCC: Pearson correlation coefficient. (B) The expression relevance between PIK3CB and SP1 was predicted by GEPIA online tools. (C) PIK3CB and SP1 were upregulated in GC tissue samples form TCGA database using GEPIA. (D) Kaplan-Meier curves for patients with different expression levels of PIK3CB or SP1 via Kaplan-Meier plotter database. (E) qRT-PCR analysis of PIK3CB and SP1 expression in 40 paired GC tissue and adjacent normal tissues. C/N score ≥1.5 was viewed as upregulated. (F) The assessment of correlation between PIK3CB and SP1 expression in 40 paired GC tissues using linear regression. (G) PIK3CB and SP1 expression level were evaluated by immunohistochemistry staining on a GC tissue microarray. Representative staining images and expression correlation of PI3KCB and SP1 in tumor tissue were shown. Scale bar: 50 μm. *P < 0.01.

    Journal: Journal of Cancer

    Article Title: SP1 Mediated PIK3CB Upregulation Promotes Gastric Carcinogenesis

    doi: 10.7150/jca.83812

    Figure Lengend Snippet: PIK3CB and SP1 were overexpressed in GC samples. (A) The top 10 genes co-expressed with PIK3CB in TCGA-STAD datasets were shown. PCC: Pearson correlation coefficient. (B) The expression relevance between PIK3CB and SP1 was predicted by GEPIA online tools. (C) PIK3CB and SP1 were upregulated in GC tissue samples form TCGA database using GEPIA. (D) Kaplan-Meier curves for patients with different expression levels of PIK3CB or SP1 via Kaplan-Meier plotter database. (E) qRT-PCR analysis of PIK3CB and SP1 expression in 40 paired GC tissue and adjacent normal tissues. C/N score ≥1.5 was viewed as upregulated. (F) The assessment of correlation between PIK3CB and SP1 expression in 40 paired GC tissues using linear regression. (G) PIK3CB and SP1 expression level were evaluated by immunohistochemistry staining on a GC tissue microarray. Representative staining images and expression correlation of PI3KCB and SP1 in tumor tissue were shown. Scale bar: 50 μm. *P < 0.01.

    Article Snippet: The human GC tissue microarray containing 48 gastric cancer tissues and adjacent normal tissues were purchased from Zhuoli Biotech (Shanghai, China).

    Techniques: Expressing, Quantitative RT-PCR, Immunohistochemistry, Staining, Microarray