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dna mass spectrometry  (agena bioscience)


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    Structured Review

    agena bioscience dna mass spectrometry
    Dna Mass Spectrometry, supplied by agena bioscience, used in various techniques. Bioz Stars score: 97/100, based on 19726 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/dna+massarray/MassARRAY+Analyzer+4/pm39551624-49-4-7
    Average 97 stars, based on 19726 article reviews
    dna mass spectrometry - by Bioz Stars, 2026-11
    97/100 stars

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    Related Articles

    Oligomer Restriction:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    Extraction:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    Control:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    QC/EQA/Proficiency:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    Software:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    Genotyping:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    DNA Extraction:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    Blood/Plasma/Serum Samples:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    Clinical Proteomics:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    Analysis:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    DNA Methylation Assay:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    Molecular Weight:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    Methylation:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    Polymerase Chain Reaction:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    Functional Assay:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    Chromatin Immunoprecipitation:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    RNA sequencing:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    Expressing:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from

    In Vitro:

    Article Title: Plasma Lipidome Is Dysregulated in Amyotrophic Lateral Sclerosis and Associated With Life‐Course Adiposity Polygenic Risk
    Article Snippet: cted in 182 plasma samples. The LC‐MS data (an 890 × 182 data matrix) were then exported to Microsoft Excel for further analysis. Following standard DNA extraction, SNP genotyping was conducted on the MassARRAY MALDI‐TOF System with genotype assignment via TYPER software. Quality control was performed using the CRLMM package in R, excluding SNPs with a call rate < 95%, MAF < 0.01 or deviation from



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    Sequenom massarray quantitative dna methylation analysis
    ZNF582-AS1 expression was regulated by <t>DNA</t> <t>methylation</t> in ccRCC. a Detection of CpG islands in ZNF582-AS1 promoter and design of MSP primers. The horizontal axis of the curved lines represents the input sequence of ZNF582-AS1, and the vertical axis of the curved lines represents GC percentage. TSS: Transcription Start Sites. b MSP analysis of ZNF582-AS1 promoter DNA methylation status in ccRCC cell lines. c MSP analysis of ZNF582-AS1 promoter DNA methylation status in ccRCC tissues. d Detection of 38 CpG sites in ZNF582-AS1 promoter. e Quantitative detection of DNA methylation level of 38 CpG sites in ZNF582-AS1 promoter using Sequenom <t>MassARRAY</t> quantitative DNA methylation analysis. f and g Comparison of the DNA methylation levels of 38 CpG sites in ccRCC and adjacent normal renal tissues. h Treatment with 5-aza-dC and TSA demethylated ZNF582-AS1 promoter and increased ZNF582-AS1 expression in OSRC2 and Caki-1 cells. T refers to Tumor tissue of ccRCC, N refers to Adjacent normal kidney tissue. M = Methylated, U = Unmethylated
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    Image Search Results


    ZNF582-AS1 expression was regulated by DNA methylation in ccRCC. a Detection of CpG islands in ZNF582-AS1 promoter and design of MSP primers. The horizontal axis of the curved lines represents the input sequence of ZNF582-AS1, and the vertical axis of the curved lines represents GC percentage. TSS: Transcription Start Sites. b MSP analysis of ZNF582-AS1 promoter DNA methylation status in ccRCC cell lines. c MSP analysis of ZNF582-AS1 promoter DNA methylation status in ccRCC tissues. d Detection of 38 CpG sites in ZNF582-AS1 promoter. e Quantitative detection of DNA methylation level of 38 CpG sites in ZNF582-AS1 promoter using Sequenom MassARRAY quantitative DNA methylation analysis. f and g Comparison of the DNA methylation levels of 38 CpG sites in ccRCC and adjacent normal renal tissues. h Treatment with 5-aza-dC and TSA demethylated ZNF582-AS1 promoter and increased ZNF582-AS1 expression in OSRC2 and Caki-1 cells. T refers to Tumor tissue of ccRCC, N refers to Adjacent normal kidney tissue. M = Methylated, U = Unmethylated

    Journal: Journal of Experimental & Clinical Cancer Research : CR

    Article Title: Downregulation of lncRNA ZNF582-AS1 due to DNA hypermethylation promotes clear cell renal cell carcinoma growth and metastasis by regulating the N(6)-methyladenosine modification of MT-RNR1

    doi: 10.1186/s13046-021-01889-8

    Figure Lengend Snippet: ZNF582-AS1 expression was regulated by DNA methylation in ccRCC. a Detection of CpG islands in ZNF582-AS1 promoter and design of MSP primers. The horizontal axis of the curved lines represents the input sequence of ZNF582-AS1, and the vertical axis of the curved lines represents GC percentage. TSS: Transcription Start Sites. b MSP analysis of ZNF582-AS1 promoter DNA methylation status in ccRCC cell lines. c MSP analysis of ZNF582-AS1 promoter DNA methylation status in ccRCC tissues. d Detection of 38 CpG sites in ZNF582-AS1 promoter. e Quantitative detection of DNA methylation level of 38 CpG sites in ZNF582-AS1 promoter using Sequenom MassARRAY quantitative DNA methylation analysis. f and g Comparison of the DNA methylation levels of 38 CpG sites in ccRCC and adjacent normal renal tissues. h Treatment with 5-aza-dC and TSA demethylated ZNF582-AS1 promoter and increased ZNF582-AS1 expression in OSRC2 and Caki-1 cells. T refers to Tumor tissue of ccRCC, N refers to Adjacent normal kidney tissue. M = Methylated, U = Unmethylated

    Article Snippet: TSS: Transcription Start Sites. b MSP analysis of ZNF582-AS1 promoter DNA methylation status in ccRCC cell lines. c MSP analysis of ZNF582-AS1 promoter DNA methylation status in ccRCC tissues. d Detection of 38 CpG sites in ZNF582-AS1 promoter. e Quantitative detection of DNA methylation level of 38 CpG sites in ZNF582-AS1 promoter using Sequenom MassARRAY quantitative DNA methylation analysis. f and g Comparison of the DNA methylation levels of 38 CpG sites in ccRCC and adjacent normal renal tissues. h Treatment with 5-aza-dC and TSA demethylated ZNF582-AS1 promoter and increased ZNF582-AS1 expression in OSRC2 and Caki-1 cells.

    Techniques: Expressing, DNA Methylation Assay, Sequencing, Comparison, Methylation