antibodies against stat1 (Proteintech)
Proteintech is a verified supplier 
96
Structured Review
Proteintech
antibodies against stat1
Antibodies Against Stat1, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 446 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+stat1/STAT1+Antibody/pm42929875-72-23-26
Average 96 stars, based on 446 article reviews
Antibodies Against Stat1, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 446 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+stat1/STAT1+Antibody/pm42929875-72-23-26
Average 96 stars, based on 446 article reviews
antibodies against stat1 - by Bioz Stars,
2026-11
96/100 stars
Images
Related Articles
Bioprocessing:Article Title: ST6Gal1 influences the esophageal squamous cell carcinoma resistance to IFN-γ by regulating the expression of IFN-γ receptor 1. Article Snippet: ST6Gal1 ( 2,6-sialyltransferase1), the primary glycosyltransferase catalyzing 2,6-sialylation of N-glycans, plays a pivotal role in the progression of various human cancers.. However, the role of ST6Gal1-mediated 2,6-sialylation in human esophageal squamous cell carcinoma (ESCC) remains poorly understood.. In this study, we established ST6Gal1 knockdown (Kyse450-KD) and overexpressed (Kyse150-OE) ESCC cell models to investigate its functional significance. Western Blot:Article Title: RUNX1 restrains STAT1-GITRL signaling to shape an immunosuppressive CRC microenvironment Article Snippet: .. The following antibodies were used for immunoblotting: anti-GITRL (NBP1-77240, Novus; 1:1000), anti-RUNX1 (25315-1-AP, Proteintech; 1:1000), Article Title: RUNX1 restrains STAT1-GITRL signaling to shape an immunosuppressive CRC microenvironment. Article Snippet: .. The following antibodies were used for immunoblotting: anti-GITRL (NBP1-77240, Novus; 1:1000), anti-RUNX1 (25315- 1-AP, Proteintech; 1:1000), Article Title: KAT5-mediated HSPA1A lactylation drives immunotherapy resistance by inhibiting STAT1 degradation in esophageal squamous cell carcinoma. Article Snippet: Immunotherapy has shown promising efficacy in esophageal squamous cell carcinoma (ESCC), yet its clinical benefits remain limited due to immune evasion.. Lactate accumulation in the tumor microenvironment has been found to facilitate immune evasion through protein lactylation.. However, the key lactylated substrates and mechanisms driving immune evasion remain undefined. Incubation:Article Title: Transcriptomic changes in the lacrimal glands of a Sjogren’s disease animal model highlight key molecular mediators and altered biological functions underpinning glandular inflammation and hypofunction Article Snippet: Then, tissue permeabilization was performed by incubation with 0.1% Triton X-100 in PBS at RT for 10 min. After three washes in PBS, non-specific binding sites were then blocked using 10% normal donkey serum (Jackson ImmunoResearch Laboratories, West Grove, PA) for 1 h at RT. .. Tissue sections were then incubated with Article Title: FLASH effect on the liver and spleen of mice-a transcriptional perspective. Article Snippet: Funding information National Key Research and Development Program of China, Grant/Award Number: 2023YFC2413200/2023YFC2413201; National Natural Science Foundation of China, Grant/Award Number: 12375334/12575369; Discipline Construction Support Project, Grant/Award Number: XKTJ-XK202410; Zhejiang Provincial Natural Science Foundation Project, Grant/Award Number: LMS26H220002; International Collaboration Fund for Creative Research Teams (ICFCRT), Grant/Award Number: W2541004 Abstract Background: FLASH radiotherapy (FLASH-RT),delivered at an ultra-high dose rate (typically ≥ 40 Gy/s), has been shown to reduce normal tissue toxicity compared with conventional dose-rate radiotherapy (CONV-RT).. However, the transcriptomic mechanisms underlying FLASH-mediated protection remain incompletely understood.. Purpose: This study aimed to compare the biological effects of FLASH and CONV irradiation on mouse liver and spleen tissues and to identify molecular pathways associated with the FLASH effect from a transcriptional perspective. Biomarker Discovery:Article Title: FLASH effect on the liver and spleen of mice-a transcriptional perspective. Article Snippet: Funding information National Key Research and Development Program of China, Grant/Award Number: 2023YFC2413200/2023YFC2413201; National Natural Science Foundation of China, Grant/Award Number: 12375334/12575369; Discipline Construction Support Project, Grant/Award Number: XKTJ-XK202410; Zhejiang Provincial Natural Science Foundation Project, Grant/Award Number: LMS26H220002; International Collaboration Fund for Creative Research Teams (ICFCRT), Grant/Award Number: W2541004 Abstract Background: FLASH radiotherapy (FLASH-RT),delivered at an ultra-high dose rate (typically ≥ 40 Gy/s), has been shown to reduce normal tissue toxicity compared with conventional dose-rate radiotherapy (CONV-RT).. However, the transcriptomic mechanisms underlying FLASH-mediated protection remain incompletely understood.. Purpose: This study aimed to compare the biological effects of FLASH and CONV irradiation on mouse liver and spleen tissues and to identify molecular pathways associated with the FLASH effect from a transcriptional perspective. Cell-Signaling:Article Title: FLASH effect on the liver and spleen of mice-a transcriptional perspective. Article Snippet: Funding information National Key Research and Development Program of China, Grant/Award Number: 2023YFC2413200/2023YFC2413201; National Natural Science Foundation of China, Grant/Award Number: 12375334/12575369; Discipline Construction Support Project, Grant/Award Number: XKTJ-XK202410; Zhejiang Provincial Natural Science Foundation Project, Grant/Award Number: LMS26H220002; International Collaboration Fund for Creative Research Teams (ICFCRT), Grant/Award Number: W2541004 Abstract Background: FLASH radiotherapy (FLASH-RT),delivered at an ultra-high dose rate (typically ≥ 40 Gy/s), has been shown to reduce normal tissue toxicity compared with conventional dose-rate radiotherapy (CONV-RT).. However, the transcriptomic mechanisms underlying FLASH-mediated protection remain incompletely understood.. Purpose: This study aimed to compare the biological effects of FLASH and CONV irradiation on mouse liver and spleen tissues and to identify molecular pathways associated with the FLASH effect from a transcriptional perspective. Immunoprecipitation:Article Title: KAT5-mediated HSPA1A lactylation drives immunotherapy resistance by inhibiting STAT1 degradation in esophageal squamous cell carcinoma. Article Snippet: Immunotherapy has shown promising efficacy in esophageal squamous cell carcinoma (ESCC), yet its clinical benefits remain limited due to immune evasion.. Lactate accumulation in the tumor microenvironment has been found to facilitate immune evasion through protein lactylation.. However, the key lactylated substrates and mechanisms driving immune evasion remain undefined. Control:Article Title: KAT5-mediated HSPA1A lactylation drives immunotherapy resistance by inhibiting STAT1 degradation in esophageal squamous cell carcinoma. Article Snippet: Immunotherapy has shown promising efficacy in esophageal squamous cell carcinoma (ESCC), yet its clinical benefits remain limited due to immune evasion.. Lactate accumulation in the tumor microenvironment has been found to facilitate immune evasion through protein lactylation.. However, the key lactylated substrates and mechanisms driving immune evasion remain undefined. |