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image analysis software zen 3 13  (Carl Zeiss)


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    Structured Review

    Carl Zeiss image analysis software zen 3 13
    Image Analysis Software Zen 3 13, supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 95/100, based on 188 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/analysis+software/ZEN+Module+Image+Analysis/pm42767265-63-12-17
    Average 95 stars, based on 188 article reviews
    image analysis software zen 3 13 - by Bioz Stars, 2026-10
    95/100 stars

    Images

    Related Articles

    Imaging:

    Article Title: Quantitative analysis of trabecular bone tissue cryosections via a fully automated neural network-based approach.
    Article Snippet: .. Quantitative analysis of histological imaging is commercially available through digital analysis software like ZEISS ZEN—Digital Imaging for light-microscopy (Carl Zeiss MicroImaging GmbH, Jena, Germany). ..

    Software:

    Article Title: Quantitative analysis of trabecular bone tissue cryosections via a fully automated neural network-based approach.
    Article Snippet: .. Quantitative analysis of histological imaging is commercially available through digital analysis software like ZEISS ZEN—Digital Imaging for light-microscopy (Carl Zeiss MicroImaging GmbH, Jena, Germany). ..

    Article Title: Suppressed basal mitophagy drives cellular aging phenotypes that can be reversed by a p62-targeting small molecule.
    Article Snippet: .. Fluorescence images were obtained using an LSM700 confocal microscope (Zeiss) and analysis software (Zen 2011, Zeiss), an inverted DM5500 microscope (Leica) or an inverted DMi8 microscope (Leica) with a Plan-Apochromat 63x/1.40 oil immersion lens, equipped with an ORCA-Flash4v2.0 camera (Hamamatsu). ..

    Article Title: Key mechanisms of angiogenesis in the infarct core: association of macrophage infiltration with venogenesis.
    Article Snippet: .. Zeiss analysis software was used to measure the length and diameter of the draining veins in the ROI. ..

    Article Title: Alzheimer’s disease associated isoforms of human CD33 distinctively modulate microglial cell responses in 5XFAD mice
    Article Snippet: .. Automated counting was done by Zeiss analysis software, and the total number of assemblies with a diameter larger than 20 μm were quantified. ..

    Article Title: Plastid LPAT1 is an integral inner envelope membrane protein with the acyltransferase domain located in the stroma.
    Article Snippet: Key message The N-terminal transmembrane domain of LPAT1 crosses the inner membrane placing the N terminus in the intermembrane space and the C-terminal enzymatic domain in the stroma.. Abstract Galactolipids monoand di-galactosyl diacylglycerol are the major and vital lipids of photosynthetic membranes.. They are synthesized by five enzymes hosted at different sub-chloroplast locations.

    Article Title: Retrospective analysis of non-invasive prenatal testing results in 8237 pregnant women in Luohe District
    Article Snippet: .. The chromosome karyotype scanning system and analysis software are manufactured by Carl Zeiss AG in Germany. ..

    Article Title: Chrysin alleviates DNA damage to improve disturbed immune homeostasis and pro-angiogenic environment in laser-induced choroidal neovascularization.
    Article Snippet: Choroidal neovascularization (CNV) is a devastating pathology of numerous ocular diseases, such as wet agerelated macular degeneration (wAMD), which causes irreversible vision loss.. Although anti-vascular endothelial growth factor (VEGF) therapy has been widely used, poor response or no response still exists in some cases, suggesting that there are other components involved in the angiogenic process.. Therefore, the underlying mechanism needs to be clarified and new target of anti-angiogenic therapy is urgently needed.

    Article Title: Dietary Zinc Restriction and Chronic Restraint Stress Affect Mice Physiology, Immune Organ Morphology, and Liver Function.
    Article Snippet: Stained slides were observed in brightfield using a light microscope (Axio Lab A1 430037-9010-000) with an Axiocam color 105 camera (all from Carl Zeiss, Tokyo, Japan). .. Collected microscopic images were examined using the following graphical analysis software: ZEN 2.3 blue edition software 3.7.97.01000 (Carl Zeiss) and ImageJ (version 1.53; US National Institutes of Health, Bethesda, MD, USA; available at http://rsb.info.nih.gov/ij/index.html; accessed 20 May 2023). ..

    Light Microscopy:

    Article Title: Quantitative analysis of trabecular bone tissue cryosections via a fully automated neural network-based approach.
    Article Snippet: .. Quantitative analysis of histological imaging is commercially available through digital analysis software like ZEISS ZEN—Digital Imaging for light-microscopy (Carl Zeiss MicroImaging GmbH, Jena, Germany). ..

    Fluorescence:

    Article Title: Suppressed basal mitophagy drives cellular aging phenotypes that can be reversed by a p62-targeting small molecule.
    Article Snippet: .. Fluorescence images were obtained using an LSM700 confocal microscope (Zeiss) and analysis software (Zen 2011, Zeiss), an inverted DM5500 microscope (Leica) or an inverted DMi8 microscope (Leica) with a Plan-Apochromat 63x/1.40 oil immersion lens, equipped with an ORCA-Flash4v2.0 camera (Hamamatsu). ..

    Article Title: Plastid LPAT1 is an integral inner envelope membrane protein with the acyltransferase domain located in the stroma.
    Article Snippet: Key message The N-terminal transmembrane domain of LPAT1 crosses the inner membrane placing the N terminus in the intermembrane space and the C-terminal enzymatic domain in the stroma.. Abstract Galactolipids monoand di-galactosyl diacylglycerol are the major and vital lipids of photosynthetic membranes.. They are synthesized by five enzymes hosted at different sub-chloroplast locations.

    Microscopy:

    Article Title: Suppressed basal mitophagy drives cellular aging phenotypes that can be reversed by a p62-targeting small molecule.
    Article Snippet: .. Fluorescence images were obtained using an LSM700 confocal microscope (Zeiss) and analysis software (Zen 2011, Zeiss), an inverted DM5500 microscope (Leica) or an inverted DMi8 microscope (Leica) with a Plan-Apochromat 63x/1.40 oil immersion lens, equipped with an ORCA-Flash4v2.0 camera (Hamamatsu). ..

    Article Title: Plastid LPAT1 is an integral inner envelope membrane protein with the acyltransferase domain located in the stroma.
    Article Snippet: Key message The N-terminal transmembrane domain of LPAT1 crosses the inner membrane placing the N terminus in the intermembrane space and the C-terminal enzymatic domain in the stroma.. Abstract Galactolipids monoand di-galactosyl diacylglycerol are the major and vital lipids of photosynthetic membranes.. They are synthesized by five enzymes hosted at different sub-chloroplast locations.

    Confocal Laser Scanning Microscopy:

    Article Title: Plastid LPAT1 is an integral inner envelope membrane protein with the acyltransferase domain located in the stroma.
    Article Snippet: Key message The N-terminal transmembrane domain of LPAT1 crosses the inner membrane placing the N terminus in the intermembrane space and the C-terminal enzymatic domain in the stroma.. Abstract Galactolipids monoand di-galactosyl diacylglycerol are the major and vital lipids of photosynthetic membranes.. They are synthesized by five enzymes hosted at different sub-chloroplast locations.

    Transgenic Assay:

    Article Title: Plastid LPAT1 is an integral inner envelope membrane protein with the acyltransferase domain located in the stroma.
    Article Snippet: Key message The N-terminal transmembrane domain of LPAT1 crosses the inner membrane placing the N terminus in the intermembrane space and the C-terminal enzymatic domain in the stroma.. Abstract Galactolipids monoand di-galactosyl diacylglycerol are the major and vital lipids of photosynthetic membranes.. They are synthesized by five enzymes hosted at different sub-chloroplast locations.



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    Physical attributes of naturally aged mice were enhanced following mGFD‐CD3 treatment. (A) Maximal walking speed, treadmill endurance, hanging endurance, and grip strength of naturally aged mice treated with mCD3 or mGFD‐CD3 were recorded at 6 months after the first infusion ( n = 5 mice per condition). (B) H&E staining of gastrocnemius collected from mice treated with mCD3 or mGFD‐CD3, sampled 6 months after the first infusion. Scale bar, 50 μm. Relative cell areas in the gastrocnemius were quantified using <t>Image</t> J ( n = 5 mice per condition). (C) Representative micro‐CT images of the distal femurs of the mice treated with mCD3 or mGFD‐CD3 at 6 months after the first infusion. The relative bone volume fraction (BV/TV) of cancellous bone in the distal femur was quantified for each sample and analyzed by <t>Imaris</t> image <t>analysis</t> <t>software</t> ( n = 5 mice per condition). (D) The percentages of total lean mass and total adipose mass and the adipose mass/lean mass ratio of naturally aged mice treated with mCD3 or mGFD‐CD3 6 months after the first infusion were recorded using InAlyzer software ( n = 5 mice per condition). (E) (left) Micro‐CT images of SAT and bone of the naturally aged mice treated with mCD3 or mGFD‐CD3 were recorded 6 months after the first infusion. Green, SAT; red, bone. (right) Quantification of the adipose tissue of mice based on the micro‐CT images ( left ). Data were normalized to mouse body weight ( n = 5 mice per condition). (F) Basal glucose levels after 12 h of starvation in mice treated with mCD3 or mGFD‐CD3 at 6 months after the first infusion ( n = 5 mice per condition). (G) ( upper ) Levels of glucose before (0 min) and after the intraperitoneal administration of glucose (2 g per kg body weight) (samples as per in (f)). ( lower ) Area under the curve (AUC) for the results from (g) ( n = 5 mice per condition). The mouse experiments are representative results from two independent experiments. Data are presented as mean ± SD and analyzed by one‐way ANOVA followed by Tukey's multiple comparisons test (A–G). * p ≤ 0.05; ** p ≤ 0.01.
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    Physical attributes of naturally aged mice were enhanced following mGFD‐CD3 treatment. (A) Maximal walking speed, treadmill endurance, hanging endurance, and grip strength of naturally aged mice treated with mCD3 or mGFD‐CD3 were recorded at 6 months after the first infusion ( n = 5 mice per condition). (B) H&E staining of gastrocnemius collected from mice treated with mCD3 or mGFD‐CD3, sampled 6 months after the first infusion. Scale bar, 50 μm. Relative cell areas in the gastrocnemius were quantified using <t>Image</t> J ( n = 5 mice per condition). (C) Representative micro‐CT images of the distal femurs of the mice treated with mCD3 or mGFD‐CD3 at 6 months after the first infusion. The relative bone volume fraction (BV/TV) of cancellous bone in the distal femur was quantified for each sample and analyzed by <t>Imaris</t> image <t>analysis</t> <t>software</t> ( n = 5 mice per condition). (D) The percentages of total lean mass and total adipose mass and the adipose mass/lean mass ratio of naturally aged mice treated with mCD3 or mGFD‐CD3 6 months after the first infusion were recorded using InAlyzer software ( n = 5 mice per condition). (E) (left) Micro‐CT images of SAT and bone of the naturally aged mice treated with mCD3 or mGFD‐CD3 were recorded 6 months after the first infusion. Green, SAT; red, bone. (right) Quantification of the adipose tissue of mice based on the micro‐CT images ( left ). Data were normalized to mouse body weight ( n = 5 mice per condition). (F) Basal glucose levels after 12 h of starvation in mice treated with mCD3 or mGFD‐CD3 at 6 months after the first infusion ( n = 5 mice per condition). (G) ( upper ) Levels of glucose before (0 min) and after the intraperitoneal administration of glucose (2 g per kg body weight) (samples as per in (f)). ( lower ) Area under the curve (AUC) for the results from (g) ( n = 5 mice per condition). The mouse experiments are representative results from two independent experiments. Data are presented as mean ± SD and analyzed by one‐way ANOVA followed by Tukey's multiple comparisons test (A–G). * p ≤ 0.05; ** p ≤ 0.01.
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    Physical attributes of naturally aged mice were enhanced following mGFD‐CD3 treatment. (A) Maximal walking speed, treadmill endurance, hanging endurance, and grip strength of naturally aged mice treated with mCD3 or mGFD‐CD3 were recorded at 6 months after the first infusion ( n = 5 mice per condition). (B) H&E staining of gastrocnemius collected from mice treated with mCD3 or mGFD‐CD3, sampled 6 months after the first infusion. Scale bar, 50 μm. Relative cell areas in the gastrocnemius were quantified using <t>Image</t> J ( n = 5 mice per condition). (C) Representative micro‐CT images of the distal femurs of the mice treated with mCD3 or mGFD‐CD3 at 6 months after the first infusion. The relative bone volume fraction (BV/TV) of cancellous bone in the distal femur was quantified for each sample and analyzed by <t>Imaris</t> image <t>analysis</t> <t>software</t> ( n = 5 mice per condition). (D) The percentages of total lean mass and total adipose mass and the adipose mass/lean mass ratio of naturally aged mice treated with mCD3 or mGFD‐CD3 6 months after the first infusion were recorded using InAlyzer software ( n = 5 mice per condition). (E) (left) Micro‐CT images of SAT and bone of the naturally aged mice treated with mCD3 or mGFD‐CD3 were recorded 6 months after the first infusion. Green, SAT; red, bone. (right) Quantification of the adipose tissue of mice based on the micro‐CT images ( left ). Data were normalized to mouse body weight ( n = 5 mice per condition). (F) Basal glucose levels after 12 h of starvation in mice treated with mCD3 or mGFD‐CD3 at 6 months after the first infusion ( n = 5 mice per condition). (G) ( upper ) Levels of glucose before (0 min) and after the intraperitoneal administration of glucose (2 g per kg body weight) (samples as per in (f)). ( lower ) Area under the curve (AUC) for the results from (g) ( n = 5 mice per condition). The mouse experiments are representative results from two independent experiments. Data are presented as mean ± SD and analyzed by one‐way ANOVA followed by Tukey's multiple comparisons test (A–G). * p ≤ 0.05; ** p ≤ 0.01.
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    Image Search Results


    Physical attributes of naturally aged mice were enhanced following mGFD‐CD3 treatment. (A) Maximal walking speed, treadmill endurance, hanging endurance, and grip strength of naturally aged mice treated with mCD3 or mGFD‐CD3 were recorded at 6 months after the first infusion ( n = 5 mice per condition). (B) H&E staining of gastrocnemius collected from mice treated with mCD3 or mGFD‐CD3, sampled 6 months after the first infusion. Scale bar, 50 μm. Relative cell areas in the gastrocnemius were quantified using Image J ( n = 5 mice per condition). (C) Representative micro‐CT images of the distal femurs of the mice treated with mCD3 or mGFD‐CD3 at 6 months after the first infusion. The relative bone volume fraction (BV/TV) of cancellous bone in the distal femur was quantified for each sample and analyzed by Imaris image analysis software ( n = 5 mice per condition). (D) The percentages of total lean mass and total adipose mass and the adipose mass/lean mass ratio of naturally aged mice treated with mCD3 or mGFD‐CD3 6 months after the first infusion were recorded using InAlyzer software ( n = 5 mice per condition). (E) (left) Micro‐CT images of SAT and bone of the naturally aged mice treated with mCD3 or mGFD‐CD3 were recorded 6 months after the first infusion. Green, SAT; red, bone. (right) Quantification of the adipose tissue of mice based on the micro‐CT images ( left ). Data were normalized to mouse body weight ( n = 5 mice per condition). (F) Basal glucose levels after 12 h of starvation in mice treated with mCD3 or mGFD‐CD3 at 6 months after the first infusion ( n = 5 mice per condition). (G) ( upper ) Levels of glucose before (0 min) and after the intraperitoneal administration of glucose (2 g per kg body weight) (samples as per in (f)). ( lower ) Area under the curve (AUC) for the results from (g) ( n = 5 mice per condition). The mouse experiments are representative results from two independent experiments. Data are presented as mean ± SD and analyzed by one‐way ANOVA followed by Tukey's multiple comparisons test (A–G). * p ≤ 0.05; ** p ≤ 0.01.

    Journal: Aging Cell

    Article Title: uPAR ‐Targeting T Cell Engager Exerts Senolytic Effects in Mice and Non‐Human Primates With Serum Aminotransferase Activity as a Safety Monitor

    doi: 10.1111/acel.70742

    Figure Lengend Snippet: Physical attributes of naturally aged mice were enhanced following mGFD‐CD3 treatment. (A) Maximal walking speed, treadmill endurance, hanging endurance, and grip strength of naturally aged mice treated with mCD3 or mGFD‐CD3 were recorded at 6 months after the first infusion ( n = 5 mice per condition). (B) H&E staining of gastrocnemius collected from mice treated with mCD3 or mGFD‐CD3, sampled 6 months after the first infusion. Scale bar, 50 μm. Relative cell areas in the gastrocnemius were quantified using Image J ( n = 5 mice per condition). (C) Representative micro‐CT images of the distal femurs of the mice treated with mCD3 or mGFD‐CD3 at 6 months after the first infusion. The relative bone volume fraction (BV/TV) of cancellous bone in the distal femur was quantified for each sample and analyzed by Imaris image analysis software ( n = 5 mice per condition). (D) The percentages of total lean mass and total adipose mass and the adipose mass/lean mass ratio of naturally aged mice treated with mCD3 or mGFD‐CD3 6 months after the first infusion were recorded using InAlyzer software ( n = 5 mice per condition). (E) (left) Micro‐CT images of SAT and bone of the naturally aged mice treated with mCD3 or mGFD‐CD3 were recorded 6 months after the first infusion. Green, SAT; red, bone. (right) Quantification of the adipose tissue of mice based on the micro‐CT images ( left ). Data were normalized to mouse body weight ( n = 5 mice per condition). (F) Basal glucose levels after 12 h of starvation in mice treated with mCD3 or mGFD‐CD3 at 6 months after the first infusion ( n = 5 mice per condition). (G) ( upper ) Levels of glucose before (0 min) and after the intraperitoneal administration of glucose (2 g per kg body weight) (samples as per in (f)). ( lower ) Area under the curve (AUC) for the results from (g) ( n = 5 mice per condition). The mouse experiments are representative results from two independent experiments. Data are presented as mean ± SD and analyzed by one‐way ANOVA followed by Tukey's multiple comparisons test (A–G). * p ≤ 0.05; ** p ≤ 0.01.

    Article Snippet: 3D image analysis was performed with Imaris image analysis software (version 10.1.0).

    Techniques: Staining, Micro-CT, Analysis, Software, Tissue