Review




Structured Review

Promega stem-loop reverse transcript primer
Stem Loop Reverse Transcript Primer, supplied by Promega, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/stem+loop+reverse+transcription/stem+loop+reverse+transcript+primer/pm23466643-67-18-29
Average 90 stars, based on 1 article reviews
stem-loop reverse transcript primer - by Bioz Stars, 2026-09
90/100 stars

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Related Articles

Quantitation Assay:

Article Title: miR-371-5p down-regulates pre mRNA processing factor 4 homolog B (PRPF4B) and facilitates the G1/S transition in human hepatocellular carcinoma cells.
Article Snippet: Increasing evidence has lent support to the notion that miRNAs regulate hepatocellular carcinoma (HCC) cell proliferation by directly targeting cell cycle-related genes.. Among these genes, we identified PRPF4B, a CDK-like kinase, as a new target of miR-371-5p.. Over-expression of miR-371-5p and knockdown of PRPF4B promotes cell growth by accelerating the G1/S transition in HCC cell lines.

Generated:

Article Title: miR-371-5p down-regulates pre mRNA processing factor 4 homolog B (PRPF4B) and facilitates the G1/S transition in human hepatocellular carcinoma cells.
Article Snippet: Increasing evidence has lent support to the notion that miRNAs regulate hepatocellular carcinoma (HCC) cell proliferation by directly targeting cell cycle-related genes.. Among these genes, we identified PRPF4B, a CDK-like kinase, as a new target of miR-371-5p.. Over-expression of miR-371-5p and knockdown of PRPF4B promotes cell growth by accelerating the G1/S transition in HCC cell lines.

Virus:

Article Title: miR-371-5p down-regulates pre mRNA processing factor 4 homolog B (PRPF4B) and facilitates the G1/S transition in human hepatocellular carcinoma cells.
Article Snippet: Increasing evidence has lent support to the notion that miRNAs regulate hepatocellular carcinoma (HCC) cell proliferation by directly targeting cell cycle-related genes.. Among these genes, we identified PRPF4B, a CDK-like kinase, as a new target of miR-371-5p.. Over-expression of miR-371-5p and knockdown of PRPF4B promotes cell growth by accelerating the G1/S transition in HCC cell lines.

Reverse Transcription:

Article Title: miR-371-5p down-regulates pre mRNA processing factor 4 homolog B (PRPF4B) and facilitates the G1/S transition in human hepatocellular carcinoma cells.
Article Snippet: Increasing evidence has lent support to the notion that miRNAs regulate hepatocellular carcinoma (HCC) cell proliferation by directly targeting cell cycle-related genes.. Among these genes, we identified PRPF4B, a CDK-like kinase, as a new target of miR-371-5p.. Over-expression of miR-371-5p and knockdown of PRPF4B promotes cell growth by accelerating the G1/S transition in HCC cell lines.

Real-time Polymerase Chain Reaction:

Article Title: miR-371-5p down-regulates pre mRNA processing factor 4 homolog B (PRPF4B) and facilitates the G1/S transition in human hepatocellular carcinoma cells.
Article Snippet: Increasing evidence has lent support to the notion that miRNAs regulate hepatocellular carcinoma (HCC) cell proliferation by directly targeting cell cycle-related genes.. Among these genes, we identified PRPF4B, a CDK-like kinase, as a new target of miR-371-5p.. Over-expression of miR-371-5p and knockdown of PRPF4B promotes cell growth by accelerating the G1/S transition in HCC cell lines.



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