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nexterion ® slide h microarray slides  (SCHOTT)

 
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    SCHOTT nexterion ® slide h microarray slides
    Nexterion ® Slide H Microarray Slides, supplied by SCHOTT, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/sciflexarrayer+s3+noncontact+microarray/nexterion+slide+h+microarray+slides/pmc06027488-65-5-11
    Average 90 stars, based on 1 article reviews
    nexterion ® slide h microarray slides - by Bioz Stars, 2026-09
    90/100 stars

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    Related Articles

    Modification:

    Article Title: Vaccine and therapeutic compositions comprising antigen-conjugated viral capsids
    Article Snippet: Amine-reactive, NHS-ester modified Nexterion® slide H (Schott, Mainz, Germany) microarray slides were used. .. Amine-reactive, NHS-ester modified Nexterion® slide H (Schott, Mainz, Germany) microarray slides were used. ..

    Microarray:

    Article Title: Vaccine and therapeutic compositions comprising antigen-conjugated viral capsids
    Article Snippet: Amine-reactive, NHS-ester modified Nexterion® slide H (Schott, Mainz, Germany) microarray slides were used. .. Amine-reactive, NHS-ester modified Nexterion® slide H (Schott, Mainz, Germany) microarray slides were used. ..

    Article Title: Micromotion Derived Fluid Shear Stress Mediates Peri‐Electrode Gliosis through Mechanosensitive Ion Channels
    Article Snippet: .. Nexterion slide H microarray slides were acquired from Schott AG (Mainz, Germany). .. CF 555, succinimidyl ester was purchased from Sigma‐Aldrich (SCJ4600022, Dublin, Ireland).

    Article Title: A Tympanic Piezo‐Bioreactor Modulates Ion Channel‐Associated Mechanosignaling to Stabilize Phenotype and Promote Tenogenesis in Human Tendon‐Derived Cells.
    Article Snippet: .. Nexterion slide H microarray slides were purchased from Schott AG (Mainz, Germany). .. Alexa Fluor 555 carboxylic acid succinimidyl ester was obtained from Life Technologies (Carlsbad, CA, USA).

    Article Title: Micromotion derived fluid shear stress mediates peri-electrode gliosis through mechanosensitive ion channels
    Article Snippet: .. Nexterion slide H microarray slides were acquired from Schott AG (Mainz, Germany). .. CFTM 555, succinimidyl ester was purchased from Sigma-Aldrich® (SCJ4600022, Dublin, Ireland).

    Article Title: Micromotion Derived Fluid Shear Stress Mediates Peri-Electrode Gliosis through Mechanosensitive Ion Channels.
    Article Snippet: .. Protein Antibody Microarray: Nexterion slide H microarray slides were acquired from Schott AG (Mainz, Germany). .. CF 555, succinimidyl ester was purchased from Sigma-Aldrich (SCJ4600022, Dublin, Ireland).

    other:

    Article Title: Enrichment of sweat-derived extracellular vesicles of human and bacterial origin for biomarker identification
    Article Snippet: Labeled samples were then cleared by centrifugation (12,000× g for 10 min in RT) and applied onto pre-printed and pre-quenched (with 50 mM ethanolamine) Nexterion H microarray slides (Schott, Germany), and further incubated in a humidified chamber with constant agitation for 2 h in RT.


    Article Title: Lectin microarray profiling demonstrates equivalent global glycosylation for whey protein ingredients enriched with α-lactalbumin and milk fat globule membrane.
    Article Snippet: Human milk fat globule membrane (MFGM) and whey proteins are nutritionally and functionally valuable, with many beneficial bioactivities associated with their glycosylation.. However glycosylation of milk components other than free milk oligosaccharides are underinvestigated.. Whey protein concentrate (WPC) ingredients with various enrichments or depletions are used in infant formula (IF) formulations to contribute to human milk equivalence and bioactivity benefits, but their overall or global glycosylation has not been compared.



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    Image Search Results


    Microarray analysis reveals the effects of GFAP mAb treatment on key inflammatory mediators in glaucomatous retina. ( A - C ) The expression of TLR4 and S100A8 was significantly downregulated by 25–50 µg GFAP mAb treatment compared to the vehicle. The marker for microglial activation, CD68, was significantly decreased in the 25 µg GFAP mAb treatment group. ( D - F ) Proteins of the inflammasome pathway, NLRP3, GSDMD and Caspase-1 were significantly downregulated in the 25 µg GFAP mAb treatment group. With the 50 µg dose, these protein expressions showed a similar downward trend, but without statistical significance. ( G ) Expression profiling of inflammation-associated mediators showed that 25 µg GFAP mAb significantly decreased pro-inflammatory factors (TNF-α, IL-1β, IL-8, MMP9, and IFN-γ), and increased the anti-inflammatory cytokine IL-10 compared to the vehicle group. In contrast, the 50 µg dose significantly reduced IFN-γ only. ( H ) Representative images of spots showing IL-1β levels in the subarrays for each group. Image analysis was conducted with Imagene software, using the median intensity from each spot to calculate the mean of the triplicate spots for each marker. For CD68 and TLR4, due to significant heterogeneity in SDs, statistical analysis was performed using Welch’s ANOVA followed by the Tamhane T2 post hoc test. All other data were analyzed using one-way ANOVA with Tukey’s post hoc test. Data are presented as the mean ± SD; n = 4 per group; * p < 0.05, ** p < 0.01, *** p < 0.001; ns = not significant. TLR4 – Toll-Like Receptor 4. S100A8 – S100 Calcium Binding Protein A8. NLRP3 – NLR Family Pyrin Domain Containing 3. GSDMD – Gasdermin D

    Journal: Journal of Neuroinflammation

    Article Title: Targeting glial fibrillary acidic protein in glaucoma: a monoclonal antibody approach to modulate glial reactivity and neuroinflammation for neuroprotection

    doi: 10.1186/s12974-025-03482-8

    Figure Lengend Snippet: Microarray analysis reveals the effects of GFAP mAb treatment on key inflammatory mediators in glaucomatous retina. ( A - C ) The expression of TLR4 and S100A8 was significantly downregulated by 25–50 µg GFAP mAb treatment compared to the vehicle. The marker for microglial activation, CD68, was significantly decreased in the 25 µg GFAP mAb treatment group. ( D - F ) Proteins of the inflammasome pathway, NLRP3, GSDMD and Caspase-1 were significantly downregulated in the 25 µg GFAP mAb treatment group. With the 50 µg dose, these protein expressions showed a similar downward trend, but without statistical significance. ( G ) Expression profiling of inflammation-associated mediators showed that 25 µg GFAP mAb significantly decreased pro-inflammatory factors (TNF-α, IL-1β, IL-8, MMP9, and IFN-γ), and increased the anti-inflammatory cytokine IL-10 compared to the vehicle group. In contrast, the 50 µg dose significantly reduced IFN-γ only. ( H ) Representative images of spots showing IL-1β levels in the subarrays for each group. Image analysis was conducted with Imagene software, using the median intensity from each spot to calculate the mean of the triplicate spots for each marker. For CD68 and TLR4, due to significant heterogeneity in SDs, statistical analysis was performed using Welch’s ANOVA followed by the Tamhane T2 post hoc test. All other data were analyzed using one-way ANOVA with Tukey’s post hoc test. Data are presented as the mean ± SD; n = 4 per group; * p < 0.05, ** p < 0.01, *** p < 0.001; ns = not significant. TLR4 – Toll-Like Receptor 4. S100A8 – S100 Calcium Binding Protein A8. NLRP3 – NLR Family Pyrin Domain Containing 3. GSDMD – Gasdermin D

    Article Snippet: Briefly, the microarray was prepared using a noncontact microarray printer (SciFLEXARRAYER S3; Scienion, Berlin, Germany).

    Techniques: Microarray, Expressing, Marker, Activation Assay, Software, Binding Assay