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nhs-activated nexterion® slides  (SCHOTT)

 
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    SCHOTT nhs-activated nexterion® slides
    Nhs Activated Nexterion® Slides, supplied by SCHOTT, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/sciflexarrayer+s3+noncontact+microarray+printer/nhs+activated+nexterion++slides/pm35750689-225-6-10
    Average 90 stars, based on 1 article reviews
    nhs-activated nexterion® slides - by Bioz Stars, 2026-09
    90/100 stars

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    Article Title: Divergent Enzymatic Assembly of a Comprehensive 64‐Membered IgG N ‐Glycan Library for Functional Glycomics
    Article Snippet: Microarray procedure [3] All compounds were printed on NHS-activated Nexterion® slides purchased from Schott using a Scienion sciCLEAN 8 non-contact microarray printer.

    Article Title: Heparan sulfate glycomimetics via iterative assembly of “clickable” disaccharides
    Article Snippet: Printing: All compounds were printed on Nexterion Slide H NHS slides (Schott) in 100 mM sodium phosphate buffer pH 8.0.

    Article Title: Cell surface glycan engineering reveals that matriglycan alone can recapitulate dystroglycan binding and function
    Article Snippet: All compounds were printed on NHS-activated Nexterion® slides purchased from Schott using a Scienion sciFLEXARRAYER S3 non-contact microarray printer equipped with a Scienion PDC80 nozzle (Scienion Inc.).

    Article Title: Chemical Resolution of an Epitope Recognized by Blood Group Antibodies Capable of Binding Both A and B Red Blood Cells.
    Article Snippet: The high specificity of human antibodies to blood group A and B antigens is impressive, especially when considering the structural difference between these antigens (tetrasaccharides) is a NHAc versus a hydroxyl group on the terminal monosaccharide residue.. It is well established that in addition to antiA and anti-B there is a third antibody, anti-A,B capable of recognizing both A and B antigens.. To analyze this AB specificity, we synthesized a tetrasaccharide, where the NHAc of the A antigen was replaced with an NH2.

    Article Title: Cell surface glycan engineering reveals that matriglycan alone can recapitulate dystroglycan binding and function.
    Article Snippet: All compounds were printed on NHS-activated Nexterion® slides purchased from Schott using a Scienion sciFLEXARRAYER S3 noncontact microarray printer equipped with a Scienion PDC80 nozzle (Scienion Inc.).

    Article Title: Anti-GalNAcα (anti-Tn) antibody repertoire differs between individuals with blood groups A and B.
    Article Snippet: 1 Shemyakin-Ovchinnikov Institute of Bioorganic Chemistry, Russian Academy of Sciences, 16/10 Miklukho-Maklaya, Moscow 117997, Russian Federation 2 Semiotik LLC, 16/10 Miklukho-Maklaya, Moscow 117997, Russian Federation 3 National Medical Research Center for Obstetrics, Gynecology and Perinatology Named After V.I.. Kulakov of the Ministry of Health Care of the Russian Federation, Moscow, Russian Federation 4 Nantes Université, Inserm, CNRS, Immunology and New Concepts in Immunotherapy, INCIT, Nantes, France Abstract Recently, Breiman et al. (2021) reported that the level of anti-Tn in the blood of healthy donors and COVID-19 patients is significantly lower in individuals of blood group A than B.. This prompted us to look for qualitative differences in the repertoire of anti-Tn like specificity in individuals of different blood groups (BG).

    Article Title: Supporting Information Synthesis of a Systematic 64-Membered Heparan Sulfate Tetrasaccharide Library
    Article Snippet: The solution was spatially arrayed onto NHSactivated slides (Nexterion® Slide H from SCHOTT, Jena, Germany) under ~50% relative humidity at 20oC.

    Modification:

    Article Title: N-glycosylation of mannose receptor (CD206) regulates glycan binding by C-type lectin domains
    Article Snippet: .. All probes were printed onto Nexterion Slide H NHS Slides (Schott AG), using the sciFLEXARRAYER S11 (Scienion US, Inc) equipped with piezo-dispensing capillary nozzles with a modified Type 3 coating (PDC 70 Type 3) as sold by the manufacturer. ..



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    Microarray analysis reveals the effects of GFAP mAb treatment on key inflammatory mediators in glaucomatous retina. ( A - C ) The expression of TLR4 and S100A8 was significantly downregulated by 25–50 µg GFAP mAb treatment compared to the vehicle. The marker for microglial activation, CD68, was significantly decreased in the 25 µg GFAP mAb treatment group. ( D - F ) Proteins of the inflammasome pathway, NLRP3, GSDMD and Caspase-1 were significantly downregulated in the 25 µg GFAP mAb treatment group. With the 50 µg dose, these protein expressions showed a similar downward trend, but without statistical significance. ( G ) Expression profiling of inflammation-associated mediators showed that 25 µg GFAP mAb significantly decreased pro-inflammatory factors (TNF-α, IL-1β, IL-8, MMP9, and IFN-γ), and increased the anti-inflammatory cytokine IL-10 compared to the vehicle group. In contrast, the 50 µg dose significantly reduced IFN-γ only. ( H ) Representative images of spots showing IL-1β levels in the subarrays for each group. Image analysis was conducted with Imagene software, using the median intensity from each spot to calculate the mean of the triplicate spots for each marker. For CD68 and TLR4, due to significant heterogeneity in SDs, statistical analysis was performed using Welch’s ANOVA followed by the Tamhane T2 post hoc test. All other data were analyzed using one-way ANOVA with Tukey’s post hoc test. Data are presented as the mean ± SD; n = 4 per group; * p < 0.05, ** p < 0.01, *** p < 0.001; ns = not significant. TLR4 – Toll-Like Receptor 4. S100A8 – S100 Calcium Binding Protein A8. NLRP3 – NLR Family Pyrin Domain Containing 3. GSDMD – Gasdermin D

    Journal: Journal of Neuroinflammation

    Article Title: Targeting glial fibrillary acidic protein in glaucoma: a monoclonal antibody approach to modulate glial reactivity and neuroinflammation for neuroprotection

    doi: 10.1186/s12974-025-03482-8

    Figure Lengend Snippet: Microarray analysis reveals the effects of GFAP mAb treatment on key inflammatory mediators in glaucomatous retina. ( A - C ) The expression of TLR4 and S100A8 was significantly downregulated by 25–50 µg GFAP mAb treatment compared to the vehicle. The marker for microglial activation, CD68, was significantly decreased in the 25 µg GFAP mAb treatment group. ( D - F ) Proteins of the inflammasome pathway, NLRP3, GSDMD and Caspase-1 were significantly downregulated in the 25 µg GFAP mAb treatment group. With the 50 µg dose, these protein expressions showed a similar downward trend, but without statistical significance. ( G ) Expression profiling of inflammation-associated mediators showed that 25 µg GFAP mAb significantly decreased pro-inflammatory factors (TNF-α, IL-1β, IL-8, MMP9, and IFN-γ), and increased the anti-inflammatory cytokine IL-10 compared to the vehicle group. In contrast, the 50 µg dose significantly reduced IFN-γ only. ( H ) Representative images of spots showing IL-1β levels in the subarrays for each group. Image analysis was conducted with Imagene software, using the median intensity from each spot to calculate the mean of the triplicate spots for each marker. For CD68 and TLR4, due to significant heterogeneity in SDs, statistical analysis was performed using Welch’s ANOVA followed by the Tamhane T2 post hoc test. All other data were analyzed using one-way ANOVA with Tukey’s post hoc test. Data are presented as the mean ± SD; n = 4 per group; * p < 0.05, ** p < 0.01, *** p < 0.001; ns = not significant. TLR4 – Toll-Like Receptor 4. S100A8 – S100 Calcium Binding Protein A8. NLRP3 – NLR Family Pyrin Domain Containing 3. GSDMD – Gasdermin D

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    Techniques: Microarray, Expressing, Marker, Activation Assay, Software, Binding Assay