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sciflexarrayer s3 noncontact microarray printer  (SCIENION)


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    Structured Review

    SCIENION sciflexarrayer s3 noncontact microarray printer
    Sciflexarrayer S3 Noncontact Microarray Printer, supplied by SCIENION, used in various techniques. Bioz Stars score: 96/100, based on 318 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/sciflexarrayer+s3+noncontact+microarray+printer/sciFLEXARRAYER+S3/pm35750689-225-14-13
    Average 96 stars, based on 318 article reviews
    sciflexarrayer s3 noncontact microarray printer - by Bioz Stars, 2026-10
    96/100 stars

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    Related Articles

    Automated:

    Article Title: A microfluidics platform for studying ion and solute transport by isolated synaptic vesicles.
    Article Snippet: .. Patterns of droplets (400 pL) were spotted on the coverslips using an automated piezo driven, non-contact dispensing system for ultra-low volumes (sciFLEXARRAYER S3, Scienion GmBH, Berlin, Germany). ..

    Article Title: Rapid classification of live bacteria in blood using antimicrobial peptide microarrays.
    Article Snippet: .. Peptide arraying was performed by printing 6 droplets of 280 to 360 pL each per spot using a sciFLEXARRAYER S3 (Scienion, Berlin, Germany) automated piezo-dispenser. ..

    other:

    Article Title: Spatially resolved, multimodal in vivo Perturb-seq using antibody-based cell hashing
    Article Snippet: Once fabricated, the chips were then filled via piezo-driven non-contact liquid dispensing (sciFLEXARRAYER S3, Scienion AG) using a custom program to fill the wells with a specific barcode layout.

    Article Title: Grating-coupled fluorescent plasmonic (GC-FP) biosensor for point-of-care serological detection of Lyme Disease.
    Article Snippet: Thin films of 5 nm titanium (adhesion layer) and 70 nm gold (plasmonic layer) were deposited using an electron beam evaporator in the University at Albany's College of Nanotechnology, Science & Engineering Innovation Lab. All of the mentioned antigens, HSA, human IgG, and human IgM, were deposited onto the chip's surface using a SciFLEXARRAYER S3 from SCIENION GmbH as described previously (Taubner et al., 2024).

    Control:

    Article Title: Label matters: comparing gold nanoparticles and nanoshells with upconversion nanoparticles for quantitative lateral flow immunoassays
    Article Snippet: .. For dip-stick tests with piezo-driven line deposition (Fig. c), antibodies were dispensed onto the test and control lines on the nitrocellulose membrane using the sciFLEXARRAYER S3 (Scienion, Germany), and the membrane was then attached to a plastic support along with an absorbent and a sample pad. ..

    Membrane:

    Article Title: Label matters: comparing gold nanoparticles and nanoshells with upconversion nanoparticles for quantitative lateral flow immunoassays
    Article Snippet: .. For dip-stick tests with piezo-driven line deposition (Fig. c), antibodies were dispensed onto the test and control lines on the nitrocellulose membrane using the sciFLEXARRAYER S3 (Scienion, Germany), and the membrane was then attached to a plastic support along with an absorbent and a sample pad. ..

    Microarray:

    Article Title: Stage-associated immunoproteomic profiling of serum autoantibody-captured retinal antigens in age-related macular degeneration
    Article Snippet: .. Commercially obtained antigens ( ) were spotted in triplicates onto nitrocellulose-coated slides (Oncyte, Grace Bio Labs, USA) using a non-contact microarray spotter (sciFLEXARRAYER S3, Scienion, Germany). ..

    Single Cell:

    Article Title: Minimally invasive monitoring of clonal evolution through integrated single cell and ctDNA analysis
    Article Snippet: .. This single cell suspension was loaded into a contactless piezoelectric dispenser (sciFLEXARRAYER S3, Scienion) and spotted in open nanowell arrays (SmartChip, TakaraBio) preprinted with unique dual index sequencing primer pairs. .. Libraries were sequenced at UBC Biomedical Research Centre (BRC) in Vancouver, British Columbia on the Illumina NextSeq 550 (mid- or high-output, paired-end 150-bp reads), or at the GSC on Illumina HiSeq2500 (paired-end 125- bp reads) and Illumina HiSeqX (paired-end 150-bp reads).

    Suspension:

    Article Title: Minimally invasive monitoring of clonal evolution through integrated single cell and ctDNA analysis
    Article Snippet: .. This single cell suspension was loaded into a contactless piezoelectric dispenser (sciFLEXARRAYER S3, Scienion) and spotted in open nanowell arrays (SmartChip, TakaraBio) preprinted with unique dual index sequencing primer pairs. .. Libraries were sequenced at UBC Biomedical Research Centre (BRC) in Vancouver, British Columbia on the Illumina NextSeq 550 (mid- or high-output, paired-end 150-bp reads), or at the GSC on Illumina HiSeq2500 (paired-end 125- bp reads) and Illumina HiSeqX (paired-end 150-bp reads).

    Sequencing:

    Article Title: Minimally invasive monitoring of clonal evolution through integrated single cell and ctDNA analysis
    Article Snippet: .. This single cell suspension was loaded into a contactless piezoelectric dispenser (sciFLEXARRAYER S3, Scienion) and spotted in open nanowell arrays (SmartChip, TakaraBio) preprinted with unique dual index sequencing primer pairs. .. Libraries were sequenced at UBC Biomedical Research Centre (BRC) in Vancouver, British Columbia on the Illumina NextSeq 550 (mid- or high-output, paired-end 150-bp reads), or at the GSC on Illumina HiSeq2500 (paired-end 125- bp reads) and Illumina HiSeqX (paired-end 150-bp reads).



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    96
    SCIENION noncontact microarray printer
    <t>Microarray</t> analysis reveals the effects of GFAP mAb treatment on key inflammatory mediators in glaucomatous retina. ( A - C ) The expression of TLR4 and S100A8 was significantly downregulated by 25–50 µg GFAP mAb treatment compared to the vehicle. The marker for microglial activation, CD68, was significantly decreased in the 25 µg GFAP mAb treatment group. ( D - F ) Proteins of the inflammasome pathway, NLRP3, GSDMD and Caspase-1 were significantly downregulated in the 25 µg GFAP mAb treatment group. With the 50 µg dose, these protein expressions showed a similar downward trend, but without statistical significance. ( G ) Expression profiling of inflammation-associated mediators showed that 25 µg GFAP mAb significantly decreased pro-inflammatory factors (TNF-α, IL-1β, IL-8, MMP9, and IFN-γ), and increased the anti-inflammatory cytokine IL-10 compared to the vehicle group. In contrast, the 50 µg dose significantly reduced IFN-γ only. ( H ) Representative images of spots showing IL-1β levels in the subarrays for each group. Image analysis was conducted with Imagene software, using the median intensity from each spot to calculate the mean of the triplicate spots for each marker. For CD68 and TLR4, due to significant heterogeneity in SDs, statistical analysis was performed using Welch’s ANOVA followed by the Tamhane T2 post hoc test. All other data were analyzed using one-way ANOVA with Tukey’s post hoc test. Data are presented as the mean ± SD; n = 4 per group; * p < 0.05, ** p < 0.01, *** p < 0.001; ns = not significant. TLR4 – Toll-Like Receptor 4. S100A8 – S100 Calcium Binding Protein A8. NLRP3 – NLR Family Pyrin Domain Containing 3. GSDMD – Gasdermin D
    Noncontact Microarray Printer, supplied by SCIENION, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/sciflexarrayer+s3+noncontact+microarray+printer/sciFLEXARRAYER+S3/pmc12175471-139-7-12
    Average 96 stars, based on 1 article reviews
    noncontact microarray printer - by Bioz Stars, 2026-10
    96/100 stars
      Buy from Supplier

    96
    SCIENION sciflexarrayer s3 noncontact microarray printer
    <t>Microarray</t> analysis reveals the effects of GFAP mAb treatment on key inflammatory mediators in glaucomatous retina. ( A - C ) The expression of TLR4 and S100A8 was significantly downregulated by 25–50 µg GFAP mAb treatment compared to the vehicle. The marker for microglial activation, CD68, was significantly decreased in the 25 µg GFAP mAb treatment group. ( D - F ) Proteins of the inflammasome pathway, NLRP3, GSDMD and Caspase-1 were significantly downregulated in the 25 µg GFAP mAb treatment group. With the 50 µg dose, these protein expressions showed a similar downward trend, but without statistical significance. ( G ) Expression profiling of inflammation-associated mediators showed that 25 µg GFAP mAb significantly decreased pro-inflammatory factors (TNF-α, IL-1β, IL-8, MMP9, and IFN-γ), and increased the anti-inflammatory cytokine IL-10 compared to the vehicle group. In contrast, the 50 µg dose significantly reduced IFN-γ only. ( H ) Representative images of spots showing IL-1β levels in the subarrays for each group. Image analysis was conducted with Imagene software, using the median intensity from each spot to calculate the mean of the triplicate spots for each marker. For CD68 and TLR4, due to significant heterogeneity in SDs, statistical analysis was performed using Welch’s ANOVA followed by the Tamhane T2 post hoc test. All other data were analyzed using one-way ANOVA with Tukey’s post hoc test. Data are presented as the mean ± SD; n = 4 per group; * p < 0.05, ** p < 0.01, *** p < 0.001; ns = not significant. TLR4 – Toll-Like Receptor 4. S100A8 – S100 Calcium Binding Protein A8. NLRP3 – NLR Family Pyrin Domain Containing 3. GSDMD – Gasdermin D
    Sciflexarrayer S3 Noncontact Microarray Printer, supplied by SCIENION, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/sciflexarrayer+s3+noncontact+microarray+printer/sciFLEXARRAYER+S3/pm35750689-225-14-13
    Average 96 stars, based on 1 article reviews
    sciflexarrayer s3 noncontact microarray printer - by Bioz Stars, 2026-10
    96/100 stars
      Buy from Supplier

    Image Search Results


    Microarray analysis reveals the effects of GFAP mAb treatment on key inflammatory mediators in glaucomatous retina. ( A - C ) The expression of TLR4 and S100A8 was significantly downregulated by 25–50 µg GFAP mAb treatment compared to the vehicle. The marker for microglial activation, CD68, was significantly decreased in the 25 µg GFAP mAb treatment group. ( D - F ) Proteins of the inflammasome pathway, NLRP3, GSDMD and Caspase-1 were significantly downregulated in the 25 µg GFAP mAb treatment group. With the 50 µg dose, these protein expressions showed a similar downward trend, but without statistical significance. ( G ) Expression profiling of inflammation-associated mediators showed that 25 µg GFAP mAb significantly decreased pro-inflammatory factors (TNF-α, IL-1β, IL-8, MMP9, and IFN-γ), and increased the anti-inflammatory cytokine IL-10 compared to the vehicle group. In contrast, the 50 µg dose significantly reduced IFN-γ only. ( H ) Representative images of spots showing IL-1β levels in the subarrays for each group. Image analysis was conducted with Imagene software, using the median intensity from each spot to calculate the mean of the triplicate spots for each marker. For CD68 and TLR4, due to significant heterogeneity in SDs, statistical analysis was performed using Welch’s ANOVA followed by the Tamhane T2 post hoc test. All other data were analyzed using one-way ANOVA with Tukey’s post hoc test. Data are presented as the mean ± SD; n = 4 per group; * p < 0.05, ** p < 0.01, *** p < 0.001; ns = not significant. TLR4 – Toll-Like Receptor 4. S100A8 – S100 Calcium Binding Protein A8. NLRP3 – NLR Family Pyrin Domain Containing 3. GSDMD – Gasdermin D

    Journal: Journal of Neuroinflammation

    Article Title: Targeting glial fibrillary acidic protein in glaucoma: a monoclonal antibody approach to modulate glial reactivity and neuroinflammation for neuroprotection

    doi: 10.1186/s12974-025-03482-8

    Figure Lengend Snippet: Microarray analysis reveals the effects of GFAP mAb treatment on key inflammatory mediators in glaucomatous retina. ( A - C ) The expression of TLR4 and S100A8 was significantly downregulated by 25–50 µg GFAP mAb treatment compared to the vehicle. The marker for microglial activation, CD68, was significantly decreased in the 25 µg GFAP mAb treatment group. ( D - F ) Proteins of the inflammasome pathway, NLRP3, GSDMD and Caspase-1 were significantly downregulated in the 25 µg GFAP mAb treatment group. With the 50 µg dose, these protein expressions showed a similar downward trend, but without statistical significance. ( G ) Expression profiling of inflammation-associated mediators showed that 25 µg GFAP mAb significantly decreased pro-inflammatory factors (TNF-α, IL-1β, IL-8, MMP9, and IFN-γ), and increased the anti-inflammatory cytokine IL-10 compared to the vehicle group. In contrast, the 50 µg dose significantly reduced IFN-γ only. ( H ) Representative images of spots showing IL-1β levels in the subarrays for each group. Image analysis was conducted with Imagene software, using the median intensity from each spot to calculate the mean of the triplicate spots for each marker. For CD68 and TLR4, due to significant heterogeneity in SDs, statistical analysis was performed using Welch’s ANOVA followed by the Tamhane T2 post hoc test. All other data were analyzed using one-way ANOVA with Tukey’s post hoc test. Data are presented as the mean ± SD; n = 4 per group; * p < 0.05, ** p < 0.01, *** p < 0.001; ns = not significant. TLR4 – Toll-Like Receptor 4. S100A8 – S100 Calcium Binding Protein A8. NLRP3 – NLR Family Pyrin Domain Containing 3. GSDMD – Gasdermin D

    Article Snippet: Briefly, the microarray was prepared using a noncontact microarray printer (SciFLEXARRAYER S3; Scienion, Berlin, Germany).

    Techniques: Microarray, Expressing, Marker, Activation Assay, Software, Binding Assay