rnx-plustm kit (CinnaGen Co)
90
Structured Review
CinnaGen Co
rnx-plustm kit
Rnx Plustm Kit, supplied by CinnaGen Co, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rnx-plustm+kit/rnx+plus+kit/pm39496922-42-5-7
Average 90 stars, based on 1 article reviews
Rnx Plustm Kit, supplied by CinnaGen Co, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rnx-plustm+kit/rnx+plus+kit/pm39496922-42-5-7
Average 90 stars, based on 1 article reviews
rnx-plustm kit - by Bioz Stars,
2026-10
90/100 stars
Images
Related Articles
other:Article Title: Identification of lncRNA associated with the SERPINE1 gene in colorectal cancer through TGF-β pathway. Article Snippet: Colorectal cancer (CRC) is a common cancer type which develops due to intricate molecular processes, the Transforming Growth Factor-beta (TGF-β) pathway a role in progression.. This study investigates the immunological functions of SERPINE1 and its interaction with long non-coding RNA (lncRNA) LINC01705 within the TGF-β pathway, aiming to identify novel therapeutic targets for CRC.. We hypothesized that LINC01705 modulates SERPINE1 expression, thereby influencing CRC progression and immune response. Article Title: Protective potential of BM-MSC extracted Exosomes in a rat model of Alzheimer's disease. Article Snippet: RNA of each hippocampal sample was extracted using an Article Title: Comparative study of liver injury protection by Akkermansia muciniphila and Faecalibacterium prausnitzii interventions in live and cell-free supernatant forms via targeting the hepcidin – ferroportin axis in mice with CCl₄-induced liver fibrosis Article Snippet: Total RNA from mice tissues (liver, colon, and brain) was extracted using the Article Title: Investigating the Relationship between FGF2 Gene Expression and Airway Remodeling in Severe Asthma. Article Snippet: The RNA was extracted using 100 μm of processed Article Title: Interferon gamma-induced hub genes and key pathways: A study based on biological network analysis and experimental validation. Article Snippet: By performing a biological network analysis, we identified some hub genes, which were upor down-regulated in the breast cancer (BC) cell line after treatment with IFN-γ.. Moreover, several pathways including cytokinecytokine receptor interaction, TNF signaling pathway, NOD-like receptor signaling pathway, and NF-κB signaling pathway were detected that their activation leads to the antiproliferation, proapoptosis, and antiviral activities.. To validate in silico results, the bioactivity of recombinant human IFN-γ (hIFN-γ) produced in different hosts was analyzed by antiviral and anticancer assays. Article Title: Improving the Accumulation and Solubility of Human Interferon-γ (hIFN-γ) in Escherichia coli: A Fusion Protein-Based Method and Network Pharmacology Analysis. Article Snippet: Using a network pharmacology-based method, some differentially expressed genes (DEGs) were detected in the colon cancer cell line (HCT116) after treatment with the human interferon-γ (hIFN-γ).. Moreover, several pathways including cell cycle, NOD-like receptor signaling pathway, and P53 signaling pathway were identified, indicating the inhibitory effect of IFN-γ on the growth and proliferation of HCT116 cells.. To validate in silico results, the hIFN-γ was first produced in Escherichia coli strain Rosetta and its bioactivity was then analyzed by anticancer assay. RNA Extraction:Article Title: Role of Tau and Amyloid-beta in autophagy gene dysregulation through oxidative stress. Article Snippet: Background: Alzheimer’s disease (AD) is a prevalent neurodegenerative disorder characterized by memory impairment and cognitive decline.. Our previous research has demonstrated that pathological Tau and Amyloidbeta (Aβ) disrupt autophagy gene expression, independently.. Other studies have shown that these pathological aggregates create a vicious cycle with oxidative stress. Article Title: Effect of FLT3 ligand on the gene expression of TIM-3, HIF1-α, and TNF-α in an acute myeloid leukemia cell line. Article Snippet: 1 Department of Immunology, Faculty of Medical Sciences, Tarbiat Modares University, Tehran 14155-114, Iran Abstract Background Acute Myeloid Leukemia (AML) pathogenesis is driven by the dysregulation of various cell signaling pathways, including the FMS-Like Tyrosine Kinase 3 (FLT3) pathway and its ligand (FLT3L).. These pathways play a critical role in promoting cell survival, proliferation, and resistance to apoptosis, contributing to leukemogenesis.. In this study, we investigated the effects of FLT3L on the expression of key genes associated with immune regulation, hypoxia, and inflammation—TIM-3, HIF-1α, and TNF-α—in the THP-1 cell line, a well-established model for AML research. cDNA Synthesis:Article Title: Effect of FLT3 ligand on the gene expression of TIM-3, HIF1-α, and TNF-α in an acute myeloid leukemia cell line. Article Snippet: 1 Department of Immunology, Faculty of Medical Sciences, Tarbiat Modares University, Tehran 14155-114, Iran Abstract Background Acute Myeloid Leukemia (AML) pathogenesis is driven by the dysregulation of various cell signaling pathways, including the FMS-Like Tyrosine Kinase 3 (FLT3) pathway and its ligand (FLT3L).. These pathways play a critical role in promoting cell survival, proliferation, and resistance to apoptosis, contributing to leukemogenesis.. In this study, we investigated the effects of FLT3L on the expression of key genes associated with immune regulation, hypoxia, and inflammation—TIM-3, HIF-1α, and TNF-α—in the THP-1 cell line, a well-established model for AML research. Quantitative RT-PCR:Article Title: Effect of FLT3 ligand on the gene expression of TIM-3, HIF1-α, and TNF-α in an acute myeloid leukemia cell line. Article Snippet: 1 Department of Immunology, Faculty of Medical Sciences, Tarbiat Modares University, Tehran 14155-114, Iran Abstract Background Acute Myeloid Leukemia (AML) pathogenesis is driven by the dysregulation of various cell signaling pathways, including the FMS-Like Tyrosine Kinase 3 (FLT3) pathway and its ligand (FLT3L).. These pathways play a critical role in promoting cell survival, proliferation, and resistance to apoptosis, contributing to leukemogenesis.. In this study, we investigated the effects of FLT3L on the expression of key genes associated with immune regulation, hypoxia, and inflammation—TIM-3, HIF-1α, and TNF-α—in the THP-1 cell line, a well-established model for AML research. |