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positions module  (Carl Zeiss)


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    Carl Zeiss positions module
    Positions Module, supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 93/100, based on 20 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/position+module/ZEN+Module+Tiles+%26+Positions/pmc12966732-189-10-13
    Average 93 stars, based on 20 article reviews
    positions module - by Bioz Stars, 2026-10
    93/100 stars

    Images

    Related Articles

    Software:

    Article Title: Moderate loss of renal mass exacerbates renal damage in a genetic mouse model of high-renin hypertension.
    Article Snippet: 11 donkey secondary antibodies (all 1:200, Jackson Immunoresearch Laboratories) and Hoechst 219 (1:1000, Molecular Probes) were applied for 30 min at RT. .. After washes in PBS, sections 220 were mounted in Fluoromount and visualized with either a LSM980 with Airyscan 2 with 221 ZenBlue software or with a Zeiss AxioObserver with the Zeiss Zen tiles and positions tool. .. 222 ImageJ software was used for quantification of glomerular area, nephrin and αSMA and or 223 visual assessment of KIM1.

    Article Title: The vascular gene Apold1 is dispensable for normal development but controls angiogenesis under pathological conditions
    Article Snippet: .. Composite images were stitched together using the tiles module in the ZEN 2011 imaging software (Zeiss). .. Fiber type composition, vascular density (% IB4/PECAM1+ area) and ERG numbers were quantified using ImageJ software.

    Article Title: Exercise-induced angiogenesis is dependent on metabolically primed ATF3/4 + endothelial cells
    Article Snippet: .. Composite images were stitched together using the tiles module in the ZEN 2011 imaging software (Zeiss). .. Fiber type composition, vascular density (% IB4/CD31 + area) and ERG numbers were quantified within the oxidative and glycolytic areas of the muscle using ImageJ software.

    Article Title: Modulation of Host Proteostasis by Prevotella corporis via Induction of the Heat Shock Response.
    Article Snippet: Fluorescence (GFP) and differential interference contrast (DIC) images were acquired using a Zeiss Axio Observer 7 microscope equipped with a Colibri 7 solid-state light source, Axiocam 503 mono camera, and a 10× EC Plan-Neofluar objective (NA 0.3). .. Images were captured and processed using the ZEN Tiles & Positions module in Zeiss ZenPro software. ..

    Article Title: Modulation of host proteostasis by Prevotella corporis via induction of the heat shock response
    Article Snippet: Fluorescence (GFP) and differential interference contrast (DIC) images were acquired using a Zeiss Axio Observer 7 microscope equipped with a Colibri 7 solid-state light source, Axiocam 503 mono camera, and a 10× EC Plan-Neofluar objective (NA 0.3). .. Images were captured and processed using the ZEN Tiles & Positions module in Zeiss ZenPro software. ..

    Article Title: Live Fluorescence Imaging of F-Actin Organization in Chick Whole Embryo Cultures Using SiR-Actin
    Article Snippet: .. The tiles module of the Zeiss Zen software was used to acquire panoramic images with a 10% overlap of single tiles (ZEN pro 2012, equipped with modules “Tiles/Positions”, “Time Lapse” and “Experiment Designer”, was purchased from Carl Zeiss BV, ZEISS Group, Breda (NL)). ..

    Imaging:

    Article Title: The vascular gene Apold1 is dispensable for normal development but controls angiogenesis under pathological conditions
    Article Snippet: .. Composite images were stitched together using the tiles module in the ZEN 2011 imaging software (Zeiss). .. Fiber type composition, vascular density (% IB4/PECAM1+ area) and ERG numbers were quantified using ImageJ software.

    Article Title: Exercise-induced angiogenesis is dependent on metabolically primed ATF3/4 + endothelial cells
    Article Snippet: .. Composite images were stitched together using the tiles module in the ZEN 2011 imaging software (Zeiss). .. Fiber type composition, vascular density (% IB4/CD31 + area) and ERG numbers were quantified within the oxidative and glycolytic areas of the muscle using ImageJ software.



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    Potentiating SK channels restores normal NMDAR integration in Grin1 Y647S +/− mice (A) Schematic of potential mechanism showing impaired negative feedback in Y647S +/− neurons due to insufficient Ca 2+ influx compared to WT and the effect of <t>NS309</t> in boosting SK channel Ca 2+ sensitivity, thereby restoring negative feedback in Y647S +/− neurons. (B) Average NMDAR plateau potential in Y647S +/− neurons at 70 μA with extended tail indicated by red arrow. Application of 10 μM NS309 to the slice restores normal duration and terminates the NMDAR plateau potential in Y647S +/− (Y647S +/− + NS309). (Inset) Restoration of plateau potential duration by NS309 at increasing stimulus intensities in a Y647S +/− neuron. (C) Total width of the NMDAR plateau potential in WT, Y647S +/− , and Y647S +/− + NS309 neurons (∗∗ p < 0.01, ∗∗∗ p < 0.001, ∗∗∗∗ p < 10 −4 , Tukey’s post hoc. Black stars: WT vs. Y647S +/− , purple stars: Y647S +/− vs. Y647S +/− + NS309). (D) SK2 channel blocker (Leidab7, 100 nM) prevents NS309 from reducing plateau potential duration in Y647S +/− neurons. (E) Normalized NMDAR plateau width in Y647S +/− neurons with the addition of NS309 and NS309+SK2 blockers (∗ p < 0.05, Sidak’s post hoc).
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    Image Search Results


    Potentiating SK channels restores normal NMDAR integration in Grin1 Y647S +/− mice (A) Schematic of potential mechanism showing impaired negative feedback in Y647S +/− neurons due to insufficient Ca 2+ influx compared to WT and the effect of NS309 in boosting SK channel Ca 2+ sensitivity, thereby restoring negative feedback in Y647S +/− neurons. (B) Average NMDAR plateau potential in Y647S +/− neurons at 70 μA with extended tail indicated by red arrow. Application of 10 μM NS309 to the slice restores normal duration and terminates the NMDAR plateau potential in Y647S +/− (Y647S +/− + NS309). (Inset) Restoration of plateau potential duration by NS309 at increasing stimulus intensities in a Y647S +/− neuron. (C) Total width of the NMDAR plateau potential in WT, Y647S +/− , and Y647S +/− + NS309 neurons (∗∗ p < 0.01, ∗∗∗ p < 0.001, ∗∗∗∗ p < 10 −4 , Tukey’s post hoc. Black stars: WT vs. Y647S +/− , purple stars: Y647S +/− vs. Y647S +/− + NS309). (D) SK2 channel blocker (Leidab7, 100 nM) prevents NS309 from reducing plateau potential duration in Y647S +/− neurons. (E) Normalized NMDAR plateau width in Y647S +/− neurons with the addition of NS309 and NS309+SK2 blockers (∗ p < 0.05, Sidak’s post hoc).

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    Figure Lengend Snippet: Potentiating SK channels restores normal NMDAR integration in Grin1 Y647S +/− mice (A) Schematic of potential mechanism showing impaired negative feedback in Y647S +/− neurons due to insufficient Ca 2+ influx compared to WT and the effect of NS309 in boosting SK channel Ca 2+ sensitivity, thereby restoring negative feedback in Y647S +/− neurons. (B) Average NMDAR plateau potential in Y647S +/− neurons at 70 μA with extended tail indicated by red arrow. Application of 10 μM NS309 to the slice restores normal duration and terminates the NMDAR plateau potential in Y647S +/− (Y647S +/− + NS309). (Inset) Restoration of plateau potential duration by NS309 at increasing stimulus intensities in a Y647S +/− neuron. (C) Total width of the NMDAR plateau potential in WT, Y647S +/− , and Y647S +/− + NS309 neurons (∗∗ p < 0.01, ∗∗∗ p < 0.001, ∗∗∗∗ p < 10 −4 , Tukey’s post hoc. Black stars: WT vs. Y647S +/− , purple stars: Y647S +/− vs. Y647S +/− + NS309). (D) SK2 channel blocker (Leidab7, 100 nM) prevents NS309 from reducing plateau potential duration in Y647S +/− neurons. (E) Normalized NMDAR plateau width in Y647S +/− neurons with the addition of NS309 and NS309+SK2 blockers (∗ p < 0.05, Sidak’s post hoc).

    Article Snippet: SK channel positive allosteric modulator NS309 (10 μM, Tocris ) was included in a subset of experiments to restore appropriate duration of dendritic integration in Y647S +/− mice.

    Techniques: