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multiphoton laser scanning microscope flouview1000 mpe  (Mason Technology Ltd)

 
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    Mason Technology Ltd multiphoton laser scanning microscope flouview1000 mpe
    Multiphoton Laser Scanning Microscope Flouview1000 Mpe, supplied by Mason Technology Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/multiphoton+microscope/multiphoton+laser+scanning+microscope+flouview1000+mpe/pmc03396659-70-4-10
    Average 90 stars, based on 1 article reviews
    multiphoton laser scanning microscope flouview1000 mpe - by Bioz Stars, 2026-11
    90/100 stars

    Images

    Related Articles

    Laser-Scanning Microscopy:

    Article Title: H 2 O 2 Production Downstream of FLT3 Is Mediated by p22phox in the Endoplasmic Reticulum and Is Required for STAT5 Signalling
    Article Snippet: .. Images were acquired using multiphoton laser scanning microscope Flouview1000 MPE (Mason Technology Dublin, Ireland) with 100x oil immersion objective. ..

    Article Title: TLR-4 Signalling Accelerates Colon Cancer Cell Adhesion via NF-κB Mediated Transcriptional Up-Regulation of Nox-1
    Article Snippet: .. SW480 cells were imaged with a multiphoton laser scanning microscope Flouview1000 MPE (Mason Technology, Dublin, Ireland) with an Infrared red Ti:Sapphire Laser that is mode locked. .. Images were acquired and visualised using an XLPLN 25×WMP water immersion objective (1.05 numerical aperture: Olympus Optical GmbH, Hamburg, Germany) and images were stored with an Olympus flouview1000 software (Mason Technology, Dublin, Ireland).

    Infrared:

    Article Title: TLR-4 Signalling Accelerates Colon Cancer Cell Adhesion via NF-κB Mediated Transcriptional Up-Regulation of Nox-1
    Article Snippet: .. SW480 cells were imaged with a multiphoton laser scanning microscope Flouview1000 MPE (Mason Technology, Dublin, Ireland) with an Infrared red Ti:Sapphire Laser that is mode locked. .. Images were acquired and visualised using an XLPLN 25×WMP water immersion objective (1.05 numerical aperture: Olympus Optical GmbH, Hamburg, Germany) and images were stored with an Olympus flouview1000 software (Mason Technology, Dublin, Ireland).

    other:

    Article Title: Imatinib and Nilotinib inhibit Bcr-Abl-induced ROS through targeted degradation of the NADPH oxidase subunit p22phox.
    Article Snippet: Constitutive expression of the Bcr–Abl kinase in Chronic Myelogenous Leukaemia (CML) is known to produce elevated levels of Reactive Oxygen Species (ROS) which can enhance cell survival as well as generate genomic instability.. Our laboratory has previously demonstrated that NADPH oxidase (Nox) activity contributes to intracellular-ROS levels in Bcr–Abl-positive cells, while inducing increased prosurvival signalling through the PI3K/Akt pathway.. How Bcr–Abl signalling regulates Nox activity still remains to be elucidated.



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    Image Search Results


    Protective effects of hiPSC-EV treatment on structural integrity and viability of hAO during IRI. (A) Experimental timeline. (B-E) Representative bright-field images (4x magnifications, EVOS microscope) of hAO pre-ischemia, post-ischemia (16 hours) and different reperfusion phases, show onset of cellular degeneration (black arrows). The bottom row of each image highlights morphological damage. (F) Label-free imaging of hAO pre-ischemia and different reperfusion phases (6 and 24 hours) performed using multiphoton microscopy. (G) hAO viability assessed by cellular ATP-dependent luminescence (RLU) at each time point. Data are mean ± SD (n = 3). Samples without hiPSC-EV treatment: w/o hiPSC-EV. Samples with hiPSC-EV treatment: with hiPSC-EV. *p < 0.05 versus pre-ischemia (control); # p < 0.05 versus IRI without hiPSC-EV.

    Journal: International Journal of Biological Sciences

    Article Title: Extracellular Vesicles from Human Induced Pluripotent Stem Cells Reduce Lung Ischemia-Reperfusion Injury via Cytoprotective Mechanisms in both 2D Cultures and a Human Airway Organoid Model

    doi: 10.7150/ijbs.134686

    Figure Lengend Snippet: Protective effects of hiPSC-EV treatment on structural integrity and viability of hAO during IRI. (A) Experimental timeline. (B-E) Representative bright-field images (4x magnifications, EVOS microscope) of hAO pre-ischemia, post-ischemia (16 hours) and different reperfusion phases, show onset of cellular degeneration (black arrows). The bottom row of each image highlights morphological damage. (F) Label-free imaging of hAO pre-ischemia and different reperfusion phases (6 and 24 hours) performed using multiphoton microscopy. (G) hAO viability assessed by cellular ATP-dependent luminescence (RLU) at each time point. Data are mean ± SD (n = 3). Samples without hiPSC-EV treatment: w/o hiPSC-EV. Samples with hiPSC-EV treatment: with hiPSC-EV. *p < 0.05 versus pre-ischemia (control); # p < 0.05 versus IRI without hiPSC-EV.

    Article Snippet: Label-free imaging of hAO was also performed using a multiphoton microscopy Leica Stellaris DIVE 8 (Leica Microsystems, Germany), enabling visualization of cellular structures and intrinsic properties while maintaining the native organoid environment.

    Techniques: Microscopy, Label-free Imaging, Control