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Eve Technologies Corporation steroid thyroid 6 plex assay multi species array panel
Steroid Thyroid 6 Plex Assay Multi Species Array Panel, supplied by Eve Technologies Corporation, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/multi-array+system/31+array+array+chemokine+cytokine+mouse+plex/pmc13167034-226-5-12
Average 86 stars, based on 1 article reviews
steroid thyroid 6 plex assay multi species array panel - by Bioz Stars, 2026-09
86/100 stars

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Clinical Proteomics:

Article Title: Targeting PAR-2 with a negative allosteric modulator increases tumor antigen presentation and potentiates anti-PD-1 immunotherapy.
Article Snippet: The supernatant was collected, and the total protein concentration was evaluated using the Bradford protein assay (Bio- Rad). .. Samples were diluted to 5 mg/ mL and sent for multiplexed profiling using the mouse cytokine/chemokine 32- Plex Discovery assay (Eotaxin, G- CSF, GM- CSF, IFNγ, IL- 1α, IL- 1β, IL- 2, IL- 3, IL- 4, IL- 5, IL- 6, IL- 7, IL- 9, IL- 10, IL- 12p40, IL- 12p70, IL- 13, IL- 15, IL- 17A, IP- 10, KC, LIF, LIX, MCP- 1, M- CSF, MIG, MIP- 1α, MIP- 1β, MIP- 2, RANTES, TNFα, VEGF- A) at Eve Technologies Corporation (Calgary, AB, Canada). ..

Article Title: Effect of intravenous lipid and glutathione infusions on metabolism, inflammation and immune functions of dairy cows.
Article Snippet: .. Fifteen cytokines—including interferon-γ, interleukin (IL) −1α, IL-1β, IL-4, IL-6, IL-8, IL-10, IL-17A, IL-36 receptor antagonist (IL-36RA), interferon-γ-induced protein 10 (IP-10), monocyte chemoattractant protein-1 (MCP-1), macrophage inflammatory proteins (MIP)-1α, MIP-1β, tumor necrosis factor-α (TNF-α), and vascular endothelial growth factor (VEGF)—were measured in plasma by Eve Technologies Corporation (Calgary, AB, Canada) using Bovine Cytokine 15-Plex Discovery Assay Array (BD15), as per the manufacturer’s instructions for use (MILLIPLEX Bovine Cytokine/Chemokine Magnetic Bead Panel 1 Cat. # BCYT1–33K, MilliporeSigma). .. Eve Technologies Corporation used the Luminex 200 system (Luminex Corporation/DiaSorin) with BioPlex Manager software (Bio-Rad Laboratories Inc.) to complete analysis.

other:

Article Title: Targeting Hyperoxia‐Induced Cellular Senescence in Developing Human Airway Cells: Senomorphics Versus Senolytics Versus Antioxidants
Article Snippet: Supernatants collected at day 8 from normoxia and hyperoxia‐exposed fASM were analyzed for SASP secretion by Eve Technologies Corporation (Calgary, Alberta, Canada) using the Human Cytokine/Chemokine 96‐Plex Discovery Assay Array (HD96) panel.

Cell Culture:

Article Title: Integrated Multi-Omics Identifies Lineage-Dependent Myeloid Cells Recruitment and the APP-CD74 Axis as an Immunoregulatory Target in Pediatric High-Grade Glioma
Article Snippet: .. Cell culture supernatants were collected, centrifuged at 500×g for 5 minutes to remove debris, and submitted to Eve Technologies Corporation (Calgary, Canada) for analysis using the human Cytokine/Chemokine 71-plex Discovery assay ® Array (HD71) and TGFB 3-plex Discovery assay ® multi-species array. ..



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(A) Representative image of multi-unit recordings setup of ex vivo thalamic horizontal slices in a humidified, oxygenated interface chamber. (B) Schematic of multi-unit activity recordings with a <t>Neuronexus</t> <t>16-channel</t> recording electrode positioned in the thalamus following electrical stimulation of the internal capsule. (C) Representative 3-second recording of intrathalamic multi-unit activity evoked by stimulation of the internal capsule (black dot indicates time of stimulation). Only 12 of 16 channels are shown for clarity. (D) Collapsed activity from 12 recording channels in (C). Black dot indicates time of stimulation. (E) Post-stimulus time histogram of instantaneous spiking frequency from multi-unit activity recorded across 16 channels (all 16 channels were analyzed, 12 representative channels are displayed in (C) from 15 slices from 9 hCSF1 (WT) mice, 8 slices from 7 hCSF1-Grn −/− +KOxMG mice, and 12 slices from 9 hCSF1-Grn −/− +WTxMG mice. The grey box denotes early response (0–1.5 s), and the white box denotes delayed response (1.5–3 s) after stimulation. (F) Enlarged instantaneous frequency of the delayed response (1.5–3 s) from (D). (G) Relative probability of eliciting spiking during the direct response (0–1.5 s) after stimulation; p values from Kolmogorov-Smirnov test with Bonferroni correction (* p = 0.0024). (H) Instantaneous frequency of spiking during the early response (0–1.5 s) after stimulation; Kruskal–Wallis test, H(3) = 6.767, p = 0.0797. (I) Relative probability of eliciting spiking during the delayed response (1.5–3 s) after stimulation; p values from Kolmogorov-Smirnov test with Bonferroni correction (**** p < 0.0001). (J) Instantaneous frequency of spiking during the delayed response (1.5–3 s) after stimulation; Kruskal-Wallis test, H(3)=56.6, **** p < 0.0001, and Dunn’s multiple comparisons test (**** p < 0.0001). Data are presented as mean ± SEM. Comparisons not shown are not significant.
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(A) Representative image of multi-unit recordings setup of ex vivo thalamic horizontal slices in a humidified, oxygenated interface chamber. (B) Schematic of multi-unit activity recordings with a Neuronexus 16-channel recording electrode positioned in the thalamus following electrical stimulation of the internal capsule. (C) Representative 3-second recording of intrathalamic multi-unit activity evoked by stimulation of the internal capsule (black dot indicates time of stimulation). Only 12 of 16 channels are shown for clarity. (D) Collapsed activity from 12 recording channels in (C). Black dot indicates time of stimulation. (E) Post-stimulus time histogram of instantaneous spiking frequency from multi-unit activity recorded across 16 channels (all 16 channels were analyzed, 12 representative channels are displayed in (C) from 15 slices from 9 hCSF1 (WT) mice, 8 slices from 7 hCSF1-Grn −/− +KOxMG mice, and 12 slices from 9 hCSF1-Grn −/− +WTxMG mice. The grey box denotes early response (0–1.5 s), and the white box denotes delayed response (1.5–3 s) after stimulation. (F) Enlarged instantaneous frequency of the delayed response (1.5–3 s) from (D). (G) Relative probability of eliciting spiking during the direct response (0–1.5 s) after stimulation; p values from Kolmogorov-Smirnov test with Bonferroni correction (* p = 0.0024). (H) Instantaneous frequency of spiking during the early response (0–1.5 s) after stimulation; Kruskal–Wallis test, H(3) = 6.767, p = 0.0797. (I) Relative probability of eliciting spiking during the delayed response (1.5–3 s) after stimulation; p values from Kolmogorov-Smirnov test with Bonferroni correction (**** p < 0.0001). (J) Instantaneous frequency of spiking during the delayed response (1.5–3 s) after stimulation; Kruskal-Wallis test, H(3)=56.6, **** p < 0.0001, and Dunn’s multiple comparisons test (**** p < 0.0001). Data are presented as mean ± SEM. Comparisons not shown are not significant.

Journal: bioRxiv

Article Title: Transplantation of Human IPSC-derived Microglia Ameliorates Neuropathology and Circuit Dysfunction in Progranulin-Deficient Mice

doi: 10.64898/2026.01.13.699312

Figure Lengend Snippet: (A) Representative image of multi-unit recordings setup of ex vivo thalamic horizontal slices in a humidified, oxygenated interface chamber. (B) Schematic of multi-unit activity recordings with a Neuronexus 16-channel recording electrode positioned in the thalamus following electrical stimulation of the internal capsule. (C) Representative 3-second recording of intrathalamic multi-unit activity evoked by stimulation of the internal capsule (black dot indicates time of stimulation). Only 12 of 16 channels are shown for clarity. (D) Collapsed activity from 12 recording channels in (C). Black dot indicates time of stimulation. (E) Post-stimulus time histogram of instantaneous spiking frequency from multi-unit activity recorded across 16 channels (all 16 channels were analyzed, 12 representative channels are displayed in (C) from 15 slices from 9 hCSF1 (WT) mice, 8 slices from 7 hCSF1-Grn −/− +KOxMG mice, and 12 slices from 9 hCSF1-Grn −/− +WTxMG mice. The grey box denotes early response (0–1.5 s), and the white box denotes delayed response (1.5–3 s) after stimulation. (F) Enlarged instantaneous frequency of the delayed response (1.5–3 s) from (D). (G) Relative probability of eliciting spiking during the direct response (0–1.5 s) after stimulation; p values from Kolmogorov-Smirnov test with Bonferroni correction (* p = 0.0024). (H) Instantaneous frequency of spiking during the early response (0–1.5 s) after stimulation; Kruskal–Wallis test, H(3) = 6.767, p = 0.0797. (I) Relative probability of eliciting spiking during the delayed response (1.5–3 s) after stimulation; p values from Kolmogorov-Smirnov test with Bonferroni correction (**** p < 0.0001). (J) Instantaneous frequency of spiking during the delayed response (1.5–3 s) after stimulation; Kruskal-Wallis test, H(3)=56.6, **** p < 0.0001, and Dunn’s multiple comparisons test (**** p < 0.0001). Data are presented as mean ± SEM. Comparisons not shown are not significant.

Article Snippet: Extracellular multi-unit activity (MUA) recordings were obtained with a linear 16-channel multi-electrode array (Neuronexus) that spanned the nRT and VB thalamic regions.

Techniques: Ex Vivo, Activity Assay