mult 1 (Proteintech)
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93
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Proteintech
mult 1
Mult 1, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 12 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mult-1/ULBP1+Antibody/pm39849761-166-77-81
Average 93 stars, based on 12 article reviews
Mult 1, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 12 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mult-1/ULBP1+Antibody/pm39849761-166-77-81
Average 93 stars, based on 12 article reviews
mult 1 - by Bioz Stars,
2026-11
93/100 stars
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Western Blot:Article Title: TAK-981 potentiates doxorubicin immunocide in triple-negative breast cancer by IFN I-dependent NK cell stimulation. Article Snippet: Protein bands were visualized using enhanced chemiluminescence (ECL) reagents (Meilun Biotechnology, Dalian, China). .. The antibodies employed in this Western blot analysis included GAPDH (dilution 1:1000, #60004-1- Ig, Proteintech, Wuhan, China), SUMO1 (dilution 1:1000, #ab5316, Abcam, Cambridge, England), JAK1 (dilution 1:1000, #3332,Cell Signaling Technology, Boston, USA), p-JAK1 (dilution 1:1000, #74129,Cell Signaling Technology, Boston, USA), STAT1(dilution 1:1000, #82016-1- RR, Proteintech, Wuhan, China), p-STAT1(dilution 1:800, #AF6300, affinity biosciences, Affinity Biosciences, Ohio, USA), NF-κB(dilution 1:1000, #10745-1-AP, Proteintech, Wuhan, China), p- nf-κB(dilution 1:500, #80379-2-RR, Proteintech, Wuhan, China), IFNβ (dilution 1:200, #27506- 1-AP, Proteintech, Wuhan, China), Article Title: TAK-981 potentiates doxorubicin immunocide in triple-negative breast cancer by IFN I-dependent NK cell stimulation Article Snippet: Protein bands were visualized using enhanced chemiluminescence (ECL) reagents (Meilun Biotechnology, Dalian, China). .. The antibodies employed in this Western blot analysis included GAPDH (dilution 1:1000, #60004-1-Ig, Proteintech, Wuhan, China), SUMO1 (dilution 1:1000, #ab5316, Abcam, Cambridge, England), JAK1 (dilution 1:1000, #3332,Cell Signaling Technology, Boston, USA), p-JAK1 (dilution 1:1000, #74129,Cell Signaling Technology, Boston, USA), STAT1(dilution 1:1000, #82016-1-RR, Proteintech, Wuhan, China), p-STAT1(dilution 1:800, #AF6300, affinity biosciences, Affinity Biosciences, Ohio, USA), NF-κB(dilution 1:1000, #10745-1-AP, Proteintech, Wuhan, China), p- nf-κB(dilution 1:500, #80379-2-RR, Proteintech, Wuhan, China), IFNβ (dilution 1:200, #27506-1-AP, Proteintech, Wuhan, China), other:Article Title: NKG2D overexpression potentiates CAR-T cells through costimulation by tumor NKG2D ligands. Article Snippet: REAGENT or RESOURCE SOURCE IDENTIFIER APC anti-human Tim-3 antibody BioLegend Cat#364804; RRID:AB_2616719 PE anti-human Tim-3 antibody BioLegend Cat#345006; RRID:AB_11124386 APC anti-human ULBP1 antibody R&D Systems Cat#FAB1380A; RRID:AB_10569682 BV421 anti-human ULBP-2/5/6 antibody BD Biosciences Cat#748128; RRID:AB_2872361 APC anti-human ULBP3 antibody R&D Systems Cat#FAB1517A; RRID:AB_10569684 Anti-E-cadherin antibody R&D Systems Cat#AF648; RRID:AB_355504 Anti-CD68 antibody Cell Signaling Technology Cat#97778S; RRID:AB_2799736 Anti-CD3 antibody Abcam Cat#ab135372; RRID:AB_2884903 Anti-myeloperoxidase antibody Abcam Cat#ab208670; RRID:AB_2893155 Anti-Ki67 antibody Abcam Cat#ab16667; RRID:AB_302459 Anti-PLCγ antibody Cell Signaling Technology Cat#2822S; RRID:AB_2163702 Anti-phospho-PLCγ antibody Cell Signaling Technology Cat#2821S; RRID:AB_330855 Anti-ZAP70 antibody Cell Signaling Technology Cat#2705S; RRID:AB_2220023 Anti-phospho-ZAP70 antibody Cell Signaling Technology Cat#2717S; RRID:AB_2220027 Anti-phospho-CD3ζ antibody Cell Signaling Technology Cat#67748S; RRID:AB_2909899 Anti-GAPDH antibody Cell Signaling Technology Cat#2118S; RRID:AB_561053 Anti-phospho-p44/42 MAPK (ERK1/2) antibody Cell Signaling Technology Cat#9101S; RRID:AB_331646 Anti-ERK1 antibody Selleck Chemicals Cat#F1152; RRID:N/A Anti-AKT1 antibody Selleck Chemicals Cat#F0217; RRID:N/A Anti-phospho-AKT (Ser473) antibody Cell Signaling Technology Cat#4060S; RRID:AB_2315049 Anti-STAT3 antibody Cell Signaling Technology Cat#9139S; RRID:AB_331757 Anti-phospho-STAT3 (Tyr705) antibody Cell Signaling Technology Cat#9145S; RRID:AB_2491009 Saline:Article Title: Exosomes Derived from Meningitic Escherichia coli-Infected Brain Microvascular Endothelial Cells Facilitate Astrocyte Activation. Article Snippet: Numerous studies have shown that exosomes play a regulatory role in a variety of biological processes as well as in disease development and progression.. However, exosome-mediated intercellular communication between brain microvascular endothelial cells (BMECs) and astrocytes during meningitic Escherichia coli (E. coli)–induced neuroinflammation remains largely unknown.. Here, by using in vivo and in vitro models, we demonstrate that exosomes derived from meningitic E. coli–infected BMECs can activate the inflammatory response of astrocytes. Incubation:Article Title: Exosomes Derived from Meningitic Escherichia coli-Infected Brain Microvascular Endothelial Cells Facilitate Astrocyte Activation. Article Snippet: Numerous studies have shown that exosomes play a regulatory role in a variety of biological processes as well as in disease development and progression.. However, exosome-mediated intercellular communication between brain microvascular endothelial cells (BMECs) and astrocytes during meningitic Escherichia coli (E. coli)–induced neuroinflammation remains largely unknown.. Here, by using in vivo and in vitro models, we demonstrate that exosomes derived from meningitic E. coli–infected BMECs can activate the inflammatory response of astrocytes. Membrane:Article Title: Exosomes Derived from Meningitic Escherichia coli-Infected Brain Microvascular Endothelial Cells Facilitate Astrocyte Activation. Article Snippet: Numerous studies have shown that exosomes play a regulatory role in a variety of biological processes as well as in disease development and progression.. However, exosome-mediated intercellular communication between brain microvascular endothelial cells (BMECs) and astrocytes during meningitic Escherichia coli (E. coli)–induced neuroinflammation remains largely unknown.. Here, by using in vivo and in vitro models, we demonstrate that exosomes derived from meningitic E. coli–infected BMECs can activate the inflammatory response of astrocytes. |