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proteome profiler mouse chemokine array kit 1  (R&D Systems)


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    R&D Systems proteome profiler mouse chemokine array kit 1
    Proteome Profiler Mouse Chemokine Array Kit 1, supplied by R&D Systems, used in various techniques. Bioz Stars score: 95/100, based on 83 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+proteome+profiler+array/Proteome+Profiler+Mouse+Chemokine+Array+Kit/pmc12913046-97-9-17
    Average 95 stars, based on 83 article reviews
    proteome profiler mouse chemokine array kit 1 - by Bioz Stars, 2026-10
    95/100 stars

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    Related Articles

    other:

    Article Title: UHRF1 drives subtype-independent aggressiveness and immune evasion in small cell lung cancer through PRC2 interactions
    Article Snippet: mUhrf1 RNA in situ hybridization was performed with RNAscope Multiplex Fluorescent Reagent Kit (Advanced Cell Diagnostics) per manufacturer’s instructions.

    Article Title: Regulatory B cell CCL3 competency promotes disease resolution and oligodendrogenesis in experimental autoimmune encephalomyelitis
    Article Snippet: Chemokine array was conducted using concentrated B cells or GIFT15-Bregs-conditioned media and analyzed with the Proteome Profiler Mouse Chemokine Array Kit (R&D Systems, ARY020) in accordance with the manufacturer’s protocol.

    Article Title: UHRF1 drives subtype-independent aggressiveness and immune evasion in small cell lung cancer through PRC2 interactions
    Article Snippet: After wash, 3,3-diaminobenzidine 823 DAB Quanto (Epredia) was added to the slides, followed by a hematoxylin counterstain.

    Bradford Assay:

    Article Title: Metabolic Reprogramming of T Cells by Dual UCP2 and IL‐17 Blockade Enhances Immunity Against Pancreatic Cancer
    Article Snippet: Serum IFN‐γ, IL‐17, TNF‐α, IL‐12, IL‐6, and TGF‐β concentrations were quantified using commercial ELISA kits (BD Biosciences; R&D systems; BioLegend) following manufacturer instructions. .. Tumor lysates were quantified by Bradford assay, and 250 μg protein per sample was analyzed using a Proteome Profiler Mouse Chemokine Array Kit (ARY020; R&D Systems) per manufacturer protocols. ..

    Article Title: Metabolic Reprogramming of T Cells by Dual UCP2 and IL-17 Blockade Enhances Immunity Against Pancreatic Cancer.
    Article Snippet: Serum IFN- γ, IL-17, TNF- α, IL-12, IL-6, and TGF- β concentrations were quantified using commercial ELISA kits (BD Biosciences; R&D systems; BioLegend) following manufacturer instructions. .. Tumor lysates were quantified by Bradford assay, and 250 μg protein per sample was analyzed using a Proteome Profiler Mouse Chemokine Array Kit (ARY020; R&D Systems) per manufacturer protocols. ..



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    <t>Cytokine</t> profiling showed significantly elevated levels of (A) CXCL10/IP-10 (1.37-fold increase; p < 0.0001, n = 3) and TIMP-1 (1.35-fold increase; p < 0.0001, n = 3) in the CD38-OE group compared to the CD38-WT group, and (B) ICAM-1, with a 1.15-fold increase compared to the CD38-WT group and a 1.34-fold increase compared to the control group (p < 0.0002, n = 3). No significant changes were observed in SDF-1/CXCL12 expression. (n = 3). These molecules were highlighted for their notable, yet relatively understudied, roles in GBM progression and tumor microenvironment modulation compared to other cytokines in the panel, which have more established functions in cancer biology. One-way ANOVA with Tukey’s post-hoc, *p < 0.0001, **p < 0.0002.
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    <t>Cytokine</t> profiling showed significantly elevated levels of (A) CXCL10/IP-10 (1.37-fold increase; p < 0.0001, n = 3) and TIMP-1 (1.35-fold increase; p < 0.0001, n = 3) in the CD38-OE group compared to the CD38-WT group, and (B) ICAM-1, with a 1.15-fold increase compared to the CD38-WT group and a 1.34-fold increase compared to the control group (p < 0.0002, n = 3). No significant changes were observed in SDF-1/CXCL12 expression. (n = 3). These molecules were highlighted for their notable, yet relatively understudied, roles in GBM progression and tumor microenvironment modulation compared to other cytokines in the panel, which have more established functions in cancer biology. One-way ANOVA with Tukey’s post-hoc, *p < 0.0001, **p < 0.0002.
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    panel a  (R&D Systems)
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    <t>Cytokine</t> profiling showed significantly elevated levels of (A) CXCL10/IP-10 (1.37-fold increase; p < 0.0001, n = 3) and TIMP-1 (1.35-fold increase; p < 0.0001, n = 3) in the CD38-OE group compared to the CD38-WT group, and (B) ICAM-1, with a 1.15-fold increase compared to the CD38-WT group and a 1.34-fold increase compared to the control group (p < 0.0002, n = 3). No significant changes were observed in SDF-1/CXCL12 expression. (n = 3). These molecules were highlighted for their notable, yet relatively understudied, roles in GBM progression and tumor microenvironment modulation compared to other cytokines in the panel, which have more established functions in cancer biology. One-way ANOVA with Tukey’s post-hoc, *p < 0.0001, **p < 0.0002.
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    Image Search Results


    Cytokine profiling showed significantly elevated levels of (A) CXCL10/IP-10 (1.37-fold increase; p < 0.0001, n = 3) and TIMP-1 (1.35-fold increase; p < 0.0001, n = 3) in the CD38-OE group compared to the CD38-WT group, and (B) ICAM-1, with a 1.15-fold increase compared to the CD38-WT group and a 1.34-fold increase compared to the control group (p < 0.0002, n = 3). No significant changes were observed in SDF-1/CXCL12 expression. (n = 3). These molecules were highlighted for their notable, yet relatively understudied, roles in GBM progression and tumor microenvironment modulation compared to other cytokines in the panel, which have more established functions in cancer biology. One-way ANOVA with Tukey’s post-hoc, *p < 0.0001, **p < 0.0002.

    Journal: Translational Oncology

    Article Title: CD38 overexpression drives glioblastoma progression via L1CAM/ICAM1/JAK-STAT-Driven tumor microenvironment rewiring

    doi: 10.1016/j.tranon.2026.102758

    Figure Lengend Snippet: Cytokine profiling showed significantly elevated levels of (A) CXCL10/IP-10 (1.37-fold increase; p < 0.0001, n = 3) and TIMP-1 (1.35-fold increase; p < 0.0001, n = 3) in the CD38-OE group compared to the CD38-WT group, and (B) ICAM-1, with a 1.15-fold increase compared to the CD38-WT group and a 1.34-fold increase compared to the control group (p < 0.0002, n = 3). No significant changes were observed in SDF-1/CXCL12 expression. (n = 3). These molecules were highlighted for their notable, yet relatively understudied, roles in GBM progression and tumor microenvironment modulation compared to other cytokines in the panel, which have more established functions in cancer biology. One-way ANOVA with Tukey’s post-hoc, *p < 0.0001, **p < 0.0002.

    Article Snippet: For cytokine profiling, the Mouse Cytokine Array Panel A (Catalog #ARY006, R&D Systems, USA) was used according to the manufacturer’s instructions (Supplementary Table).

    Techniques: Control, Expressing