microarray gene expression profiles (IMGM Laboratories GmbH)
90
Structured Review
IMGM Laboratories GmbH
microarray gene expression profiles
Microarray Gene Expression Profiles, supplied by IMGM Laboratories GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/microarray+gene-expression+profile+datasets/microarray+gene+expression+profiles/pm31207605-415-13-9
Average 90 stars, based on 1 article reviews
Microarray Gene Expression Profiles, supplied by IMGM Laboratories GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/microarray+gene-expression+profile+datasets/microarray+gene+expression+profiles/pm31207605-415-13-9
Average 90 stars, based on 1 article reviews
microarray gene expression profiles - by Bioz Stars,
2026-09
90/100 stars
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Related Articles
Microarray:Article Title: TOX reinforces the phenotype and longevity of exhausted T cells in chronic viral infection. Article Snippet: Cytotoxic T cells are essential mediators of protective immunity to viral infection and malignant tumours and are a key target of immunotherapy approaches.. However, prolonged exposure to cognate antigens often attenuates the effector capacity of T cells and limits their therapeutic potential1–4.. This process, known as T cell exhaustion or dysfunction1, is manifested by epigenetically enforced changes in gene regulation that reduce the expression of cytokines and effector molecules and upregulate the expression of inhibitory receptors such as programmed cell-death 1 (PD-1)5–8. Gene Expression:Article Title: TOX reinforces the phenotype and longevity of exhausted T cells in chronic viral infection. Article Snippet: Cytotoxic T cells are essential mediators of protective immunity to viral infection and malignant tumours and are a key target of immunotherapy approaches.. However, prolonged exposure to cognate antigens often attenuates the effector capacity of T cells and limits their therapeutic potential1–4.. This process, known as T cell exhaustion or dysfunction1, is manifested by epigenetically enforced changes in gene regulation that reduce the expression of cytokines and effector molecules and upregulate the expression of inhibitory receptors such as programmed cell-death 1 (PD-1)5–8. |
