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Incyte corporation microarrays
Microarrays, supplied by Incyte corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/marathon+argus+microarray+printer/microarrays/pmc06740475-144-11-10
Average 90 stars, based on 1 article reviews
microarrays - by Bioz Stars, 2026-10
90/100 stars

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Gene Expression:

Article Title: Induction of dendritic cell-like phenotype in macrophages during foam cell formation.
Article Snippet: Hyung Jun Cho, Pavel Shashkin, Christian A. Gleissner, Dane Dunson, Nitin Jain, Jae K. Lee, Yury Miller, and Klaus Ley Department of Public Health Sciences, University of Virginia, Charlottesville, Virginia; Department of Statistics, Korea University, Seoul, Korea; Department of Biomedical Engineering, Department of Molecular Physiology and Biological Physics, and Robert M. Berne Cardiovascular Research Center, University of Virginia, Charlottesville, Virginia; and Department of Medicine, University of California, San Diego, California

Article Title: Deep mRNA Sequencing for In Vivo Functional Analysis of Cardiac Transcriptional Regulators
Article Snippet: .. Previous analyses of mRNA expression in the cardiac G q transgenic model used RNA dot blotting4,19–22 or Incyte microarrays,17,18 neither of which provides a comprehensive examination of gene expression. ..

Article Title: The Fuzzy Logic of Physiological Cardiac Hypertrophy
Article Snippet: .. Specific alterations in cardiac gene expression observed in pathological hypertrophy are sufficiently characteristic so that they have become important criteria for its detection and diagnosis.2,31 Our laboratory performed one of the first attempts to delineate the molecular signatures of physiological and pathological forms of cardiac hypertrophy using early murine microarrays and 5 different mouse genetic hypertrophy models.32 In these experiments, more physiological hypertrophy was represented by a mouse in which protein kinase C was specifically activated through transgenic translocation facilitation.33 Pathological hypertrophy was represented by a mouse overexpressing the subunit of Gq,34 and pathological hypertrophy in transition to heart failure was represented by a mouse overexpressing calcineurin.35 Although the study was limited by lack of comprehensive representation of the mouse genome on the Incyte microarrays and by use of specific genetic perturbations rather than physiological stress to create a spectrum of hypertrophy, the results are noteworthy in that they revealed nonoverlap of gene expression between the physiological and pathological hypertrophy phenotypes and described an apoptosis gene expression program in pathological hypertrophy. ..

Microarray:

Article Title: Method for treating cancer using an antibody that inhibits notch4 signaling
Article Snippet: .. Among the vendors of microarrays and microarray technology useage are Affymetrix, Inc. (USA), NimbleGen Systems, Inc. (Madison, Wis., USA), and Incyte Genomics (USA) (producing microarrays for core facilities in large industrial and academic departments); Agilent Technologies (USA) and Grafinity Pharmaceutical Design, GmbH (Germany) (which provide specific services such as printing and fingerprinting arrays designed and used by individual researchers); and CLONTECH Laboratories (Becton Dickinson Bioscience) and BioRobotics, Ltd. (Great Britain) (which provide the basic tools necessary for individual researchers to carry out the entire process of producing microarrays, including printing). ..

Labeling:

Article Title: Methods and systems for facilitating the diagnosis and treatment of schizophrenia
Article Snippet: .. Sample Labeling, Microarrays, Hybridization, and Data Analysis Labeling was performed at Incyte Genomics, Inc. (Fremont, Calif.). ..

Article Title: Functional Genomics and Psychiatric Illness
Article Snippet: .. Labeled probes from pairs of controls and schizophrenics were combined and hybridized to high-density, commercially produced (Incyte Pharmaceuticals) microarrays containing 8700 human cDNAs. ..

Hybridization:

Article Title: Methods and systems for facilitating the diagnosis and treatment of schizophrenia
Article Snippet: .. Sample Labeling, Microarrays, Hybridization, and Data Analysis Labeling was performed at Incyte Genomics, Inc. (Fremont, Calif.). ..

Expressing:

Article Title: Deep mRNA Sequencing for In Vivo Functional Analysis of Cardiac Transcriptional Regulators
Article Snippet: .. Previous analyses of mRNA expression in the cardiac G q transgenic model used RNA dot blotting4,19–22 or Incyte microarrays,17,18 neither of which provides a comprehensive examination of gene expression. ..

Transgenic Assay:

Article Title: Deep mRNA Sequencing for In Vivo Functional Analysis of Cardiac Transcriptional Regulators
Article Snippet: .. Previous analyses of mRNA expression in the cardiac G q transgenic model used RNA dot blotting4,19–22 or Incyte microarrays,17,18 neither of which provides a comprehensive examination of gene expression. ..

Article Title: The Fuzzy Logic of Physiological Cardiac Hypertrophy
Article Snippet: .. Specific alterations in cardiac gene expression observed in pathological hypertrophy are sufficiently characteristic so that they have become important criteria for its detection and diagnosis.2,31 Our laboratory performed one of the first attempts to delineate the molecular signatures of physiological and pathological forms of cardiac hypertrophy using early murine microarrays and 5 different mouse genetic hypertrophy models.32 In these experiments, more physiological hypertrophy was represented by a mouse in which protein kinase C was specifically activated through transgenic translocation facilitation.33 Pathological hypertrophy was represented by a mouse overexpressing the subunit of Gq,34 and pathological hypertrophy in transition to heart failure was represented by a mouse overexpressing calcineurin.35 Although the study was limited by lack of comprehensive representation of the mouse genome on the Incyte microarrays and by use of specific genetic perturbations rather than physiological stress to create a spectrum of hypertrophy, the results are noteworthy in that they revealed nonoverlap of gene expression between the physiological and pathological hypertrophy phenotypes and described an apoptosis gene expression program in pathological hypertrophy. ..

other:

Article Title: Systems toxicology: applications of toxicogenomics, transcriptomics, proteomics and metabolomics in toxicology.
Article Snippet: Review 10.1586/14789450.2.5.767 © 2005 Future Drugs Ltd ISSN 1478-9450 767www.future-drugs.com Systems toxicology: applications of toxicogenomics, transcriptomics, proteomics and metabolomics in toxicology Wilbert HM Heijne, Anne S Kienhuis, Ben van Ommen, Rob H Stierum† and John P Groten †Author for correspondence TNO, Physiological Sciences Department , PO Box 360, 3700 AJ Zeist, The Netherlands Tel.. : +31 30 694 4545 Fax: +31 30 696 0264 rob.stierum@tno.nl KEYWORDS: genomics, metabolomics, proteomics, systems biology, toxicogenomics, toxicology, transcriptomics Toxicogenomics can facilitate the identification and characterization of toxicity, as illustrated in this review.. Toxicogenomics, the application of the functional genomics technologies (transcriptomics, proteomics and metabolomics) in toxicology enables the study of adverse effects of xenobiotic substances in relation to structure and activity of the genome.

Clone Assay:

Article Title: Gene and protein expression in the myometrium in pregnancy and labor.
Article Snippet: .. Many sources of clones can be used for array analyses including microarrays purchased from companies (e.g. Incyte, Affymetrix, Clontech), which consist of cDNA clones or oligonucleotides that cover a large percentage of the transcripts (known as ESTs) present in public databases (Juhasz et al. 2002). ..

Produced:

Article Title: Functional Genomics and Psychiatric Illness
Article Snippet: .. Labeled probes from pairs of controls and schizophrenics were combined and hybridized to high-density, commercially produced (Incyte Pharmaceuticals) microarrays containing 8700 human cDNAs. ..

Biomarker Discovery:

Article Title: The Fuzzy Logic of Physiological Cardiac Hypertrophy
Article Snippet: .. Specific alterations in cardiac gene expression observed in pathological hypertrophy are sufficiently characteristic so that they have become important criteria for its detection and diagnosis.2,31 Our laboratory performed one of the first attempts to delineate the molecular signatures of physiological and pathological forms of cardiac hypertrophy using early murine microarrays and 5 different mouse genetic hypertrophy models.32 In these experiments, more physiological hypertrophy was represented by a mouse in which protein kinase C was specifically activated through transgenic translocation facilitation.33 Pathological hypertrophy was represented by a mouse overexpressing the subunit of Gq,34 and pathological hypertrophy in transition to heart failure was represented by a mouse overexpressing calcineurin.35 Although the study was limited by lack of comprehensive representation of the mouse genome on the Incyte microarrays and by use of specific genetic perturbations rather than physiological stress to create a spectrum of hypertrophy, the results are noteworthy in that they revealed nonoverlap of gene expression between the physiological and pathological hypertrophy phenotypes and described an apoptosis gene expression program in pathological hypertrophy. ..

Translocation Assay:

Article Title: The Fuzzy Logic of Physiological Cardiac Hypertrophy
Article Snippet: .. Specific alterations in cardiac gene expression observed in pathological hypertrophy are sufficiently characteristic so that they have become important criteria for its detection and diagnosis.2,31 Our laboratory performed one of the first attempts to delineate the molecular signatures of physiological and pathological forms of cardiac hypertrophy using early murine microarrays and 5 different mouse genetic hypertrophy models.32 In these experiments, more physiological hypertrophy was represented by a mouse in which protein kinase C was specifically activated through transgenic translocation facilitation.33 Pathological hypertrophy was represented by a mouse overexpressing the subunit of Gq,34 and pathological hypertrophy in transition to heart failure was represented by a mouse overexpressing calcineurin.35 Although the study was limited by lack of comprehensive representation of the mouse genome on the Incyte microarrays and by use of specific genetic perturbations rather than physiological stress to create a spectrum of hypertrophy, the results are noteworthy in that they revealed nonoverlap of gene expression between the physiological and pathological hypertrophy phenotypes and described an apoptosis gene expression program in pathological hypertrophy. ..



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