lcmodel (Pfeuffer GmbH)
90
Structured Review
Pfeuffer GmbH
lcmodel
Lcmodel, supplied by Pfeuffer GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/linear+combination+model+program+package+lcmodel/lcmodel/pmc04174428-64-0-41
Average 90 stars, based on 1 article reviews
Lcmodel, supplied by Pfeuffer GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/linear+combination+model+program+package+lcmodel/lcmodel/pmc04174428-64-0-41
Average 90 stars, based on 1 article reviews
lcmodel - by Bioz Stars,
2026-09
90/100 stars
Images
Related Articles
Software:Article Title: Ubiquinol treatment for TBI in male rats: Effects on mitochondrial integrity, injury severity, and neurometabolism. Article Snippet: Funding Information This research was sponsored by the TriService Nursing Research Program, Uniformed Services University of the Health Sciences and The Uniformed Services University of the Health Sciences (USU), grant number: HU0001-14-1-TS03 Abstract Following traumatic brain injury (TBI), there is significant secondary damage to cerebral tissue from increased free radicals and impaired mitochondrial function.. This imbalance between reactive oxygen species (ROS) production and the effectiveness of cellular antioxidant defenses is termed oxidative stress.. Often there are insufficient antioxidants to scavenge ROS, leading to alterations in cerebral structure and function. In Vivo:Article Title: Postnatal Morphine Administration Alters Hippocampal Development in Rats Article Snippet: .. In vivo 1 H NMR spectra were analyzed using Article Title: Ubiquinol treatment for TBI in male rats: Effects on mitochondrial integrity, injury severity, and neurometabolism. Article Snippet: Funding Information This research was sponsored by the TriService Nursing Research Program, Uniformed Services University of the Health Sciences and The Uniformed Services University of the Health Sciences (USU), grant number: HU0001-14-1-TS03 Abstract Following traumatic brain injury (TBI), there is significant secondary damage to cerebral tissue from increased free radicals and impaired mitochondrial function.. This imbalance between reactive oxygen species (ROS) production and the effectiveness of cellular antioxidant defenses is termed oxidative stress.. Often there are insufficient antioxidants to scavenge ROS, leading to alterations in cerebral structure and function. Article Title: High-field proton magnetic resonance spectroscopy reveals metabolic effects of normal brain aging Article Snippet: Spectral fitting and quantification Spectra were analyzed with LCModel ( Provencher, 1993 ). .. Article Title: The metabolomic profile during isoflurane anesthesia differs from propofol anesthesia in the live rodent brain Article Snippet: .. Incorporation of anesthesia-related macromolecules in the basis data set Macromolecule (MM) spectra were acquired separately in vivo and were incorporated in the simulated basis data sets for accurate Article Title: Altered neurochemical profile after traumatic brain injury: 1 H-MRS biomarkers of pathological mechanisms Article Snippet: .. Article Title: Postnatal Morphine Administration Alters Hippocampal Development in Rats Article Snippet: .. The study of a single rat did not exceed 60 min. Quantification of Neurochemicals In vivo 1 H NMR spectra were analyzed using Nuclear Magnetic Resonance:Article Title: Postnatal Morphine Administration Alters Hippocampal Development in Rats Article Snippet: .. In vivo 1 H NMR spectra were analyzed using Article Title: Postnatal Morphine Administration Alters Hippocampal Development in Rats Article Snippet: .. The study of a single rat did not exceed 60 min. Quantification of Neurochemicals In vivo 1 H NMR spectra were analyzed using In Vitro:Article Title: Ubiquinol treatment for TBI in male rats: Effects on mitochondrial integrity, injury severity, and neurometabolism. Article Snippet: Funding Information This research was sponsored by the TriService Nursing Research Program, Uniformed Services University of the Health Sciences and The Uniformed Services University of the Health Sciences (USU), grant number: HU0001-14-1-TS03 Abstract Following traumatic brain injury (TBI), there is significant secondary damage to cerebral tissue from increased free radicals and impaired mitochondrial function.. This imbalance between reactive oxygen species (ROS) production and the effectiveness of cellular antioxidant defenses is termed oxidative stress.. Often there are insufficient antioxidants to scavenge ROS, leading to alterations in cerebral structure and function. Article Title: Altered neurochemical profile after traumatic brain injury: 1 H-MRS biomarkers of pathological mechanisms Article Snippet: .. |