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one step tunel apoptosis assay kit (Beyotime)

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Beyotime one step tunel apoptosis assay kit

Fig. 4 AGH improved glutathione metabolism and reduced <t>apoptosis.</t> TA muscles from WT, mdx, and AGH-treatment mdx mice were isolated at 8 weeks of age. The ratio of (A) oxidized glutathione (GSSG) to glutathione (GSH) (GSSG/GSH), (B) the concentration of total GSH, (C) GSSG, and (D) GSH were mea sured per assay kit instructions. The activities of (E) GSH reductase and (F) GSH peroxidase in TA muscle were measured per assay kit instructions. Gene expression of (G) Gpx2, Gpx3, Gpx4, and (J) caspase3 measured using real-time PCR. Values were normalized to 36B4 levels. (H–I) Representative images and quantification of <t>TUNEL-positive</t> nuclei on TA muscle (20× magnification, scale bar = 100 μm). Values are presented as the mean ± standard error of the mean for each experiment (n = 5). * p < 0.05 compared with WT control, # p < 0.05 compared with mdx

One Step Tunel Apoptosis Assay Kit, supplied by Beyotime, used in various techniques. Bioz Stars score: 98/100, based on 3512 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/image+processing+tool+kit/pm39806512-97-9-16?v=Beyotime
Average 98 stars, based on 3512 article reviews

one step tunel apoptosis assay kit - by Bioz Stars, 2026-07

98/100 stars

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1) Product Images from "Aminoguanidine hemisulfate improves mitochondrial autophagy, oxidative stress, and muscle force in Duchenne muscular dystrophy via the AKT/FOXO1 pathway in mdx mice."

Article Title: Aminoguanidine hemisulfate improves mitochondrial autophagy, oxidative stress, and muscle force in Duchenne muscular dystrophy via the AKT/FOXO1 pathway in mdx mice.

Journal: Skeletal muscle

doi: 10.1186/s13395-024-00371-1

Fig. 4 AGH improved glutathione metabolism and reduced apoptosis. TA muscles from WT, mdx, and AGH-treatment mdx mice were isolated at 8 weeks of age. The ratio of (A) oxidized glutathione (GSSG) to glutathione (GSH) (GSSG/GSH), (B) the concentration of total GSH, (C) GSSG, and (D) GSH were mea sured per assay kit instructions. The activities of (E) GSH reductase and (F) GSH peroxidase in TA muscle were measured per assay kit instructions. Gene expression of (G) Gpx2, Gpx3, Gpx4, and (J) caspase3 measured using real-time PCR. Values were normalized to 36B4 levels. (H–I) Representative images and quantification of TUNEL-positive nuclei on TA muscle (20× magnification, scale bar = 100 μm). Values are presented as the mean ± standard error of the mean for each experiment (n = 5). * p < 0.05 compared with WT control, # p < 0.05 compared with mdx

Figure Legend Snippet: Fig. 4 AGH improved glutathione metabolism and reduced apoptosis. TA muscles from WT, mdx, and AGH-treatment mdx mice were isolated at 8 weeks of age. The ratio of (A) oxidized glutathione (GSSG) to glutathione (GSH) (GSSG/GSH), (B) the concentration of total GSH, (C) GSSG, and (D) GSH were mea sured per assay kit instructions. The activities of (E) GSH reductase and (F) GSH peroxidase in TA muscle were measured per assay kit instructions. Gene expression of (G) Gpx2, Gpx3, Gpx4, and (J) caspase3 measured using real-time PCR. Values were normalized to 36B4 levels. (H–I) Representative images and quantification of TUNEL-positive nuclei on TA muscle (20× magnification, scale bar = 100 μm). Values are presented as the mean ± standard error of the mean for each experiment (n = 5). * p < 0.05 compared with WT control, # p < 0.05 compared with mdx

Techniques Used: Muscles, Isolation, Concentration Assay, Gene Expression, Real-time Polymerase Chain Reaction, TUNEL Assay, Control

Fig. 8 Summary diagram of AGH treatment in mdx mice. AGH treatment reduced ROS and apoptosis by restoring mitochondrial autophagy, restored the composition of muscle fiber types, and improved muscle force through the AKT/FOXO1 signaling pathway

Figure Legend Snippet: Fig. 8 Summary diagram of AGH treatment in mdx mice. AGH treatment reduced ROS and apoptosis by restoring mitochondrial autophagy, restored the composition of muscle fiber types, and improved muscle force through the AKT/FOXO1 signaling pathway

Techniques Used:

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one step tunel apoptosis assay kit (Beyotime)

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1) Product Images from "Aminoguanidine hemisulfate improves mitochondrial autophagy, oxidative stress, and muscle force in Duchenne muscular dystrophy via the AKT/FOXO1 pathway in mdx mice."

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