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Hamamatsu computer system image quest
Computer System Image Quest, supplied by Hamamatsu, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/image+analysis+software+macscope/computer+analyzer+system+imagequest/pm20966610-53-17-21
Average 90 stars, based on 1 article reviews
computer system image quest - by Bioz Stars, 2026-11
90/100 stars

Images

Related Articles

Staining:

Article Title: Imidapril improves L-NAME-exacerbated nephrosclerosis with TGF-beta 1 inhibition in spontaneously hypertensive rats.
Article Snippet: Objective This study was designed to investigate whether chronic angiotensin-converting enzyme (ACE) inhibition prevents hypertensive glomerular injury and inhibits increases in the mRNA levels and immunohistological expression of the apoptosis inducer caspase-3, and transforming growth factor (TGF)1 during prolonged nitric oxide synthase (NOS) inhibition with N-nitro-Larginine methyl ester (L-NAME) in spontaneously hypertensive rats (SHR).

Article Title: Cardiorenal protective effects of year-long antihypertensive therapy with an angiotensin-converting enzyme inhibitor or a calcium channel blocker in spontaneously hypertensive rats.
Article Snippet: The area stained in blue with aniline was quantified in 10 randomly selected high-power fields ( 200) using a computer system (Image Quest, Hamamatsu Photonics; Hamamatsu and MacScope, Mitani Co., Fukui, Japan), and the average percent value was used for comparison.

Article Title: Nipradilol prevents L-NAME-exacerbated nephrosclerosis with decreasing of caspase-3 expression in SHR.
Article Snippet: All microscopic slides were examined using a computer analyzer system (Image Quest, Hamamatsu Photonics, Hama- 434 Hypertens Res Vol.

Article Title: Year-long antihypertensive therapy with candesartan completely prevents development of cardiovascular organ injuries in spontaneously hypertensive rats.
Article Snippet: The area stained blue with aniline was quantified in 10 randomly-selected high-power fields (× 200) using a computer system (Image Quest, Hamamatsu Photonics; Hamamatsu and MacScope, Mitani Co., Fukui, Japan), and the average percent value was used for comparison.

Article Title: Apoptosis and glomerular injury after prolonged nitric oxide synthase inhibition in spontaneously hypertensive rats.
Article Snippet: For the morphometric studies, the glomerular area was determined by computer-assisted image analysis (ImageQuest, Hamamatsu Photonics K.K., Hamamatsu, Japan) as described previously.15,20 In situ detection of apoptotic cells (terminal deoxynucleotidil transferase–mediated dUTP nick end labeling of fragmented DNA, TUNEL) was performed as reported by Gavrieli et al.24 Tissue sections were stained using the In Situ Apoptosis Detection Kit (Oncor, Gathersburg, Md) and analyzed by the dioxigenin-nucleotide method, by which the detected cells show internucleosomal DNA fragmentation.

Article Title: Insertion/deletion angiotensin converting enzyme gene polymorphism affects the microvascular structure of the kidney in patients with nondiabetic renal disease.
Article Snippet: Objective It has been reported that the deletion allele of the insertion/deletion polymorphism of the angiotensin I converting enzyme gene is associated with increased cardiovascular risk and progressive renal disease, including immunoglobulin A nephropathy.. We therefore investigated the relationship between angiotensin converting enzyme polymorphism and intrarenal microvascular structure in 56 patients with nondiabetic renal disease.

Comparison:

Article Title: Imidapril improves L-NAME-exacerbated nephrosclerosis with TGF-beta 1 inhibition in spontaneously hypertensive rats.
Article Snippet: Objective This study was designed to investigate whether chronic angiotensin-converting enzyme (ACE) inhibition prevents hypertensive glomerular injury and inhibits increases in the mRNA levels and immunohistological expression of the apoptosis inducer caspase-3, and transforming growth factor (TGF)1 during prolonged nitric oxide synthase (NOS) inhibition with N-nitro-Larginine methyl ester (L-NAME) in spontaneously hypertensive rats (SHR).

Article Title: Cardiorenal protective effects of year-long antihypertensive therapy with an angiotensin-converting enzyme inhibitor or a calcium channel blocker in spontaneously hypertensive rats.
Article Snippet: The area stained in blue with aniline was quantified in 10 randomly selected high-power fields ( 200) using a computer system (Image Quest, Hamamatsu Photonics; Hamamatsu and MacScope, Mitani Co., Fukui, Japan), and the average percent value was used for comparison.

Article Title: Nipradilol prevents L-NAME-exacerbated nephrosclerosis with decreasing of caspase-3 expression in SHR.
Article Snippet: All microscopic slides were examined using a computer analyzer system (Image Quest, Hamamatsu Photonics, Hama- 434 Hypertens Res Vol.

Article Title: Year-long antihypertensive therapy with candesartan completely prevents development of cardiovascular organ injuries in spontaneously hypertensive rats.
Article Snippet: The area stained blue with aniline was quantified in 10 randomly-selected high-power fields (× 200) using a computer system (Image Quest, Hamamatsu Photonics; Hamamatsu and MacScope, Mitani Co., Fukui, Japan), and the average percent value was used for comparison.

Article Title: Apoptosis and glomerular injury after prolonged nitric oxide synthase inhibition in spontaneously hypertensive rats.
Article Snippet: For the morphometric studies, the glomerular area was determined by computer-assisted image analysis (ImageQuest, Hamamatsu Photonics K.K., Hamamatsu, Japan) as described previously.15,20 In situ detection of apoptotic cells (terminal deoxynucleotidil transferase–mediated dUTP nick end labeling of fragmented DNA, TUNEL) was performed as reported by Gavrieli et al.24 Tissue sections were stained using the In Situ Apoptosis Detection Kit (Oncor, Gathersburg, Md) and analyzed by the dioxigenin-nucleotide method, by which the detected cells show internucleosomal DNA fragmentation.

Article Title: Insertion/deletion angiotensin converting enzyme gene polymorphism affects the microvascular structure of the kidney in patients with nondiabetic renal disease.
Article Snippet: Objective It has been reported that the deletion allele of the insertion/deletion polymorphism of the angiotensin I converting enzyme gene is associated with increased cardiovascular risk and progressive renal disease, including immunoglobulin A nephropathy.. We therefore investigated the relationship between angiotensin converting enzyme polymorphism and intrarenal microvascular structure in 56 patients with nondiabetic renal disease.

In Situ:

Article Title: Imidapril improves L-NAME-exacerbated nephrosclerosis with TGF-beta 1 inhibition in spontaneously hypertensive rats.
Article Snippet: Objective This study was designed to investigate whether chronic angiotensin-converting enzyme (ACE) inhibition prevents hypertensive glomerular injury and inhibits increases in the mRNA levels and immunohistological expression of the apoptosis inducer caspase-3, and transforming growth factor (TGF)1 during prolonged nitric oxide synthase (NOS) inhibition with N-nitro-Larginine methyl ester (L-NAME) in spontaneously hypertensive rats (SHR).

Article Title: Cardiorenal protective effects of year-long antihypertensive therapy with an angiotensin-converting enzyme inhibitor or a calcium channel blocker in spontaneously hypertensive rats.
Article Snippet: The area stained in blue with aniline was quantified in 10 randomly selected high-power fields ( 200) using a computer system (Image Quest, Hamamatsu Photonics; Hamamatsu and MacScope, Mitani Co., Fukui, Japan), and the average percent value was used for comparison.

Article Title: Nipradilol prevents L-NAME-exacerbated nephrosclerosis with decreasing of caspase-3 expression in SHR.
Article Snippet: All microscopic slides were examined using a computer analyzer system (Image Quest, Hamamatsu Photonics, Hama- 434 Hypertens Res Vol.

Article Title: Year-long antihypertensive therapy with candesartan completely prevents development of cardiovascular organ injuries in spontaneously hypertensive rats.
Article Snippet: The area stained blue with aniline was quantified in 10 randomly-selected high-power fields (× 200) using a computer system (Image Quest, Hamamatsu Photonics; Hamamatsu and MacScope, Mitani Co., Fukui, Japan), and the average percent value was used for comparison.

Article Title: Apoptosis and glomerular injury after prolonged nitric oxide synthase inhibition in spontaneously hypertensive rats.
Article Snippet: For the morphometric studies, the glomerular area was determined by computer-assisted image analysis (ImageQuest, Hamamatsu Photonics K.K., Hamamatsu, Japan) as described previously.15,20 In situ detection of apoptotic cells (terminal deoxynucleotidil transferase–mediated dUTP nick end labeling of fragmented DNA, TUNEL) was performed as reported by Gavrieli et al.24 Tissue sections were stained using the In Situ Apoptosis Detection Kit (Oncor, Gathersburg, Md) and analyzed by the dioxigenin-nucleotide method, by which the detected cells show internucleosomal DNA fragmentation.

Article Title: Insertion/deletion angiotensin converting enzyme gene polymorphism affects the microvascular structure of the kidney in patients with nondiabetic renal disease.
Article Snippet: Objective It has been reported that the deletion allele of the insertion/deletion polymorphism of the angiotensin I converting enzyme gene is associated with increased cardiovascular risk and progressive renal disease, including immunoglobulin A nephropathy.. We therefore investigated the relationship between angiotensin converting enzyme polymorphism and intrarenal microvascular structure in 56 patients with nondiabetic renal disease.

End Labeling:

Article Title: Imidapril improves L-NAME-exacerbated nephrosclerosis with TGF-beta 1 inhibition in spontaneously hypertensive rats.
Article Snippet: Objective This study was designed to investigate whether chronic angiotensin-converting enzyme (ACE) inhibition prevents hypertensive glomerular injury and inhibits increases in the mRNA levels and immunohistological expression of the apoptosis inducer caspase-3, and transforming growth factor (TGF)1 during prolonged nitric oxide synthase (NOS) inhibition with N-nitro-Larginine methyl ester (L-NAME) in spontaneously hypertensive rats (SHR).

Article Title: Cardiorenal protective effects of year-long antihypertensive therapy with an angiotensin-converting enzyme inhibitor or a calcium channel blocker in spontaneously hypertensive rats.
Article Snippet: The area stained in blue with aniline was quantified in 10 randomly selected high-power fields ( 200) using a computer system (Image Quest, Hamamatsu Photonics; Hamamatsu and MacScope, Mitani Co., Fukui, Japan), and the average percent value was used for comparison.

Article Title: Nipradilol prevents L-NAME-exacerbated nephrosclerosis with decreasing of caspase-3 expression in SHR.
Article Snippet: All microscopic slides were examined using a computer analyzer system (Image Quest, Hamamatsu Photonics, Hama- 434 Hypertens Res Vol.

Article Title: Year-long antihypertensive therapy with candesartan completely prevents development of cardiovascular organ injuries in spontaneously hypertensive rats.
Article Snippet: The area stained blue with aniline was quantified in 10 randomly-selected high-power fields (× 200) using a computer system (Image Quest, Hamamatsu Photonics; Hamamatsu and MacScope, Mitani Co., Fukui, Japan), and the average percent value was used for comparison.

Article Title: Apoptosis and glomerular injury after prolonged nitric oxide synthase inhibition in spontaneously hypertensive rats.
Article Snippet: For the morphometric studies, the glomerular area was determined by computer-assisted image analysis (ImageQuest, Hamamatsu Photonics K.K., Hamamatsu, Japan) as described previously.15,20 In situ detection of apoptotic cells (terminal deoxynucleotidil transferase–mediated dUTP nick end labeling of fragmented DNA, TUNEL) was performed as reported by Gavrieli et al.24 Tissue sections were stained using the In Situ Apoptosis Detection Kit (Oncor, Gathersburg, Md) and analyzed by the dioxigenin-nucleotide method, by which the detected cells show internucleosomal DNA fragmentation.

Article Title: Insertion/deletion angiotensin converting enzyme gene polymorphism affects the microvascular structure of the kidney in patients with nondiabetic renal disease.
Article Snippet: Objective It has been reported that the deletion allele of the insertion/deletion polymorphism of the angiotensin I converting enzyme gene is associated with increased cardiovascular risk and progressive renal disease, including immunoglobulin A nephropathy.. We therefore investigated the relationship between angiotensin converting enzyme polymorphism and intrarenal microvascular structure in 56 patients with nondiabetic renal disease.

TUNEL Assay:

Article Title: Imidapril improves L-NAME-exacerbated nephrosclerosis with TGF-beta 1 inhibition in spontaneously hypertensive rats.
Article Snippet: Objective This study was designed to investigate whether chronic angiotensin-converting enzyme (ACE) inhibition prevents hypertensive glomerular injury and inhibits increases in the mRNA levels and immunohistological expression of the apoptosis inducer caspase-3, and transforming growth factor (TGF)1 during prolonged nitric oxide synthase (NOS) inhibition with N-nitro-Larginine methyl ester (L-NAME) in spontaneously hypertensive rats (SHR).

Article Title: Cardiorenal protective effects of year-long antihypertensive therapy with an angiotensin-converting enzyme inhibitor or a calcium channel blocker in spontaneously hypertensive rats.
Article Snippet: The area stained in blue with aniline was quantified in 10 randomly selected high-power fields ( 200) using a computer system (Image Quest, Hamamatsu Photonics; Hamamatsu and MacScope, Mitani Co., Fukui, Japan), and the average percent value was used for comparison.

Article Title: Nipradilol prevents L-NAME-exacerbated nephrosclerosis with decreasing of caspase-3 expression in SHR.
Article Snippet: All microscopic slides were examined using a computer analyzer system (Image Quest, Hamamatsu Photonics, Hama- 434 Hypertens Res Vol.

Article Title: Year-long antihypertensive therapy with candesartan completely prevents development of cardiovascular organ injuries in spontaneously hypertensive rats.
Article Snippet: The area stained blue with aniline was quantified in 10 randomly-selected high-power fields (× 200) using a computer system (Image Quest, Hamamatsu Photonics; Hamamatsu and MacScope, Mitani Co., Fukui, Japan), and the average percent value was used for comparison.

Article Title: Apoptosis and glomerular injury after prolonged nitric oxide synthase inhibition in spontaneously hypertensive rats.
Article Snippet: For the morphometric studies, the glomerular area was determined by computer-assisted image analysis (ImageQuest, Hamamatsu Photonics K.K., Hamamatsu, Japan) as described previously.15,20 In situ detection of apoptotic cells (terminal deoxynucleotidil transferase–mediated dUTP nick end labeling of fragmented DNA, TUNEL) was performed as reported by Gavrieli et al.24 Tissue sections were stained using the In Situ Apoptosis Detection Kit (Oncor, Gathersburg, Md) and analyzed by the dioxigenin-nucleotide method, by which the detected cells show internucleosomal DNA fragmentation.

Article Title: Insertion/deletion angiotensin converting enzyme gene polymorphism affects the microvascular structure of the kidney in patients with nondiabetic renal disease.
Article Snippet: Objective It has been reported that the deletion allele of the insertion/deletion polymorphism of the angiotensin I converting enzyme gene is associated with increased cardiovascular risk and progressive renal disease, including immunoglobulin A nephropathy.. We therefore investigated the relationship between angiotensin converting enzyme polymorphism and intrarenal microvascular structure in 56 patients with nondiabetic renal disease.



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