hybridization oligonucleotide microarray (Agilent technologies)
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Hybridization Oligonucleotide Microarray, supplied by Agilent technologies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/hybridization+oligonucleotide+microarray/pm33300232-52-6-14
Average 90 stars, based on 1 article reviews
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Hybridization:Article Title: Kidney Injury Molecule-1 Is Upregulated in Renal Lipotoxicity and Mediates Palmitate-Induced Tubular Cell Injury and Inflammatory Response Article Snippet: Complementary RNA (cRNA) was generated and fluorescently labeled from 1 μg of total RNA in each reaction using the One-Color Agilent Low RNA Input Fluorescent Linear Amplification Kit (p/n 5188–5339). .. Hybridization was performed following the Article Title: Expanding the phenotype of X-linked SSR4-CDG: Connective tissue implications. Article Snippet: Signal sequence receptor protein 4 (SSR4) is a subunit of the translocon-associated protein (TRAP) complex, which participates in the translocation of proteins across the endoplasmic reticulum membrane, enhancing the efficiency of N-linked glycosylation. Article Title: Genome‐wide DNA methylation profiling of CpG islands in a morpholino anthracycline derivative‐resistant leukemia cell line: p38 α as a novel candidate for resistance Article Snippet: Samples were hybridized to an Agilent 4 × 44k Whole Human Genome microarray (G4112F; Agilent Technologies), which contains 43,376 coding and noncoding sequences from the human genome, in an Agilent G2545A hybridization oven. .. Hybridization and washing conditions were as described by the manufacturer in the protocol for Article Title: Hepatic gene expression in hepatocyte-specific Pten deficient mice showing steatohepatitis without ethanol challenge. Article Snippet: 1 d Hepatocyte-specific Pten deficient (Pten KO) mice possess almost the same hepatic lesions histologically as human NASH and are thought o represent some limited NASH patients.. We analyzed a comprehensive gene expression of hepatocytes derived from 10to 35-week-old ten KO mice using the DNA microarray technology to find out the candidate genes related to development and aggravation of human NASH. pp1, Vnn1, Itga6, Abcd2, Auh, Acox1, Pdk4, Cpt1a, Lcn2, Igfbp2, Gstm6, Socs3, Tgm2, and Aldh9a1 were regarded as the candidate genes elated to inflammation.. The candidate genes of fibrosis were Spp1, Ctgf, and Cyp2c39 and moreover Cidec and Spp1 were regarded as the andidate genes of carcinogenesis. Microarray:Article Title: Kidney Injury Molecule-1 Is Upregulated in Renal Lipotoxicity and Mediates Palmitate-Induced Tubular Cell Injury and Inflammatory Response Article Snippet: Complementary RNA (cRNA) was generated and fluorescently labeled from 1 μg of total RNA in each reaction using the One-Color Agilent Low RNA Input Fluorescent Linear Amplification Kit (p/n 5188–5339). .. Hybridization was performed following the Article Title: Expanding the phenotype of X-linked SSR4-CDG: Connective tissue implications. Article Snippet: Signal sequence receptor protein 4 (SSR4) is a subunit of the translocon-associated protein (TRAP) complex, which participates in the translocation of proteins across the endoplasmic reticulum membrane, enhancing the efficiency of N-linked glycosylation. Article Title: Genome‐wide DNA methylation profiling of CpG islands in a morpholino anthracycline derivative‐resistant leukemia cell line: p38 α as a novel candidate for resistance Article Snippet: Samples were hybridized to an Agilent 4 × 44k Whole Human Genome microarray (G4112F; Agilent Technologies), which contains 43,376 coding and noncoding sequences from the human genome, in an Agilent G2545A hybridization oven. .. Hybridization and washing conditions were as described by the manufacturer in the protocol for Article Title: Hepatic gene expression in hepatocyte-specific Pten deficient mice showing steatohepatitis without ethanol challenge. Article Snippet: 1 d Hepatocyte-specific Pten deficient (Pten KO) mice possess almost the same hepatic lesions histologically as human NASH and are thought o represent some limited NASH patients.. We analyzed a comprehensive gene expression of hepatocytes derived from 10to 35-week-old ten KO mice using the DNA microarray technology to find out the candidate genes related to development and aggravation of human NASH. pp1, Vnn1, Itga6, Abcd2, Auh, Acox1, Pdk4, Cpt1a, Lcn2, Igfbp2, Gstm6, Socs3, Tgm2, and Aldh9a1 were regarded as the candidate genes elated to inflammation.. The candidate genes of fibrosis were Spp1, Ctgf, and Cyp2c39 and moreover Cidec and Spp1 were regarded as the andidate genes of carcinogenesis. other:Article Title: Epigenetic Modifications of the Estrogen Receptor β Gene in Epithelial Ovarian Cancer Cells Article Snippet: For each sample pair, the Article Title: Targeted disruption of the histone lysine 79 methyltransferase Dot1L in nephron progenitors causes congenital renal dysplasia Article Snippet: Genome-wide microarray analysis Fluorescently labelled cRNA was generated from 0.5 μg total RNA in each reaction using the Article Title: Histone deacetylase 1 and 2 regulate Wnt and p53 pathways in the ureteric bud epithelium Article Snippet: qPCR Quantitative real-time PCR (qPCR) was performed using the One-Step Brilliant Quantitative RT-PCR Master Mix Kit (Applied Biosystems). Article Title: Molecular profiling and characterization of luminal‐like and basal‐like in vivo breast cancer xenograft models Article Snippet: Hybridization of tumor and reference RNA was performed following the Labeling:Article Title: Hepatic gene expression in hepatocyte-specific Pten deficient mice showing steatohepatitis without ethanol challenge. Article Snippet: 1 d Hepatocyte-specific Pten deficient (Pten KO) mice possess almost the same hepatic lesions histologically as human NASH and are thought o represent some limited NASH patients.. We analyzed a comprehensive gene expression of hepatocytes derived from 10to 35-week-old ten KO mice using the DNA microarray technology to find out the candidate genes related to development and aggravation of human NASH. pp1, Vnn1, Itga6, Abcd2, Auh, Acox1, Pdk4, Cpt1a, Lcn2, Igfbp2, Gstm6, Socs3, Tgm2, and Aldh9a1 were regarded as the candidate genes elated to inflammation.. The candidate genes of fibrosis were Spp1, Ctgf, and Cyp2c39 and moreover Cidec and Spp1 were regarded as the andidate genes of carcinogenesis. In Situ Hybridization:Article Title: Hepatic gene expression in hepatocyte-specific Pten deficient mice showing steatohepatitis without ethanol challenge. Article Snippet: 1 d Hepatocyte-specific Pten deficient (Pten KO) mice possess almost the same hepatic lesions histologically as human NASH and are thought o represent some limited NASH patients.. We analyzed a comprehensive gene expression of hepatocytes derived from 10to 35-week-old ten KO mice using the DNA microarray technology to find out the candidate genes related to development and aggravation of human NASH. pp1, Vnn1, Itga6, Abcd2, Auh, Acox1, Pdk4, Cpt1a, Lcn2, Igfbp2, Gstm6, Socs3, Tgm2, and Aldh9a1 were regarded as the candidate genes elated to inflammation.. The candidate genes of fibrosis were Spp1, Ctgf, and Cyp2c39 and moreover Cidec and Spp1 were regarded as the andidate genes of carcinogenesis. |
![Association of immune marker signatures with PSA response and OS. (A) The correlations between the 24 non-redundant (ie, those without high Spearman correlations among each other) set of evaluated tumor immune microenvironment gene expression signatures (ρ is shown), (B) OS of the group with tumor immune gene expression signature analysis is not significantly different from the overall cohort of Lu-PSMA therapy treated patients at our institution and (C) the <t>cDNA</t> yield from RNA extracted from tumor samples, best PSA response and relative expression signature level (z-scores, higher values indicate higher signature) of immune-related gene signatures are shown correspondingly to identify patterns of signatures with regard to PSA response. Patients with higher PD-L2 signature (above the median) have longer OS time (D) and greater PSA responses (E). Lu-PSMA, [ 177 Lu]Lutetium-PSMA-617; mo., months; OS, overall survival; PD-L1, programmed death-ligand 1; PSA, prostate-specific antigen.](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_3337/pmc10603337/pmc10603337__jitc-2023-007354f01.jpg)
