glut2 polyclonal antibody (Rockland Immunochemicals)
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Rockland Immunochemicals
glut2 polyclonal antibody
Glut2 Polyclonal Antibody, supplied by Rockland Immunochemicals, used in various techniques. Bioz Stars score: 91/100, based on 8 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/glucose+solution/Glut2+Antibody/pm36249809-62-0-4
Average 91 stars, based on 8 article reviews
Glut2 Polyclonal Antibody, supplied by Rockland Immunochemicals, used in various techniques. Bioz Stars score: 91/100, based on 8 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/glucose+solution/Glut2+Antibody/pm36249809-62-0-4
Average 91 stars, based on 8 article reviews
glut2 polyclonal antibody - by Bioz Stars,
2026-09
91/100 stars
Images
1) Product Images from "Fruits of Hippophaë rhamnoides in human leukocytes and Caco-2 cell monolayer models-A question about their preventive role in lipopolysaccharide leakage and cytokine secretion in endotoxemia."
Article Title: Fruits of Hippophaë rhamnoides in human leukocytes and Caco-2 cell monolayer models-A question about their preventive role in lipopolysaccharide leakage and cytokine secretion in endotoxemia.
Journal: Frontiers in pharmacology
doi: 10.3389/fphar.2022.981874
Figure Legend Snippet: FIGURE 5 Detection of GLUT2 translocation in Caco-2 cells in a fluorescence microscope. HR—aqueous extract from the fruit of Hipopphaë rhamnoides.
Techniques Used: Translocation Assay, Microscopy
Related Articles
Western Blot:Article Title: Loss of function of renal Glut2 reverses hyperglycaemia and normalises body weight in mouse models of diabetes and obesity. Article Snippet: Aims/hypothesis Renal GLUT2 is increased in diabetes, thereby enhancing glucose reabsorption and worsening hyperglycaemia.. Here, we determined whether loss of Glut2 (also known as Slc2a2) specifically in the kidneys would reverse hyperglycaemia and normalise body weight in mouse models of diabetes and obesity.. Methods We used the tamoxifen-inducible CreERT2-Lox system in mice to knockout Glut2 specifically in the kidneys (KsGlut2 KO) to establish the contribution of renal GLUT2 to systemic glucose homeostasis in health and in insulin-dependent as well as non-insulin-dependent diabetes. Staining:Article Title: Normal β-Cell Glut2 Expression Is not Required for Regulating Glucose-Stimulated Insulin Secretion and Systemic Glucose Homeostasis in Mice Article Snippet: .. The tissue sections were then processed for GLUT1, GLUT2, or insulin staining using previously validated antibodies [ , ]: rabbit anti-GLUT1 (1:250 dilution in TBST, ab652 or ab115730, Abcam, Waltham, MA, USA), Article Title: Normal β-Cell Glut2 Expression Is not Required for Regulating Glucose-Stimulated Insulin Secretion and Systemic Glucose Homeostasis in Mice. Article Snippet: .. The tissue sections were then processed for GLUT1, GLUT2, or insulin staining using previously validated antibodies [4,5]: rabbit anti-GLUT1 (1:250 dilution in TBST, ab652 or ab115730, Abcam, Waltham, MA, USA), Incubation:Article Title: Normal β-Cell Glut2 Expression Is not Required for Regulating Glucose-Stimulated Insulin Secretion and Systemic Glucose Homeostasis in Mice Article Snippet: .. The tissue sections were then processed for GLUT1, GLUT2, or insulin staining using previously validated antibodies [ , ]: rabbit anti-GLUT1 (1:250 dilution in TBST, ab652 or ab115730, Abcam, Waltham, MA, USA), Article Title: Loss of function of renal Glut2 reverses hyperglycaemia and normalises body weight in mouse models of diabetes and obesity. Article Snippet: Aims/hypothesis Renal GLUT2 is increased in diabetes, thereby enhancing glucose reabsorption and worsening hyperglycaemia.. Here, we determined whether loss of Glut2 (also known as Slc2a2) specifically in the kidneys would reverse hyperglycaemia and normalise body weight in mouse models of diabetes and obesity.. Methods We used the tamoxifen-inducible CreERT2-Lox system in mice to knockout Glut2 specifically in the kidneys (KsGlut2 KO) to establish the contribution of renal GLUT2 to systemic glucose homeostasis in health and in insulin-dependent as well as non-insulin-dependent diabetes. Article Title: Normal β-Cell Glut2 Expression Is not Required for Regulating Glucose-Stimulated Insulin Secretion and Systemic Glucose Homeostasis in Mice. Article Snippet: .. The tissue sections were then processed for GLUT1, GLUT2, or insulin staining using previously validated antibodies [4,5]: rabbit anti-GLUT1 (1:250 dilution in TBST, ab652 or ab115730, Abcam, Waltham, MA, USA), Article Title: Transcription factor c-Maf deletion improves streptozotocin-induced diabetic nephropathy by directly regulating Sglt2 and Glut2 Article Snippet: .. The samples were then blocked for 1 hour in PBS with 5% bovine serum albumin (BSA) and 10% goat serum, then incubated for 40 hours at 4°C with the following primary antibodies: anti–c-Maf rabbit monoclonal antibody (1:200; M00654; Boster Biological Technology), anti-Sglt2 (1:200; ab85626; Abcam), anti-Glut2 (1:200; 600-401-GN3; ROCKLAND), rabbit Saline:Article Title: Loss of function of renal Glut2 reverses hyperglycaemia and normalises body weight in mouse models of diabetes and obesity. Article Snippet: Aims/hypothesis Renal GLUT2 is increased in diabetes, thereby enhancing glucose reabsorption and worsening hyperglycaemia.. Here, we determined whether loss of Glut2 (also known as Slc2a2) specifically in the kidneys would reverse hyperglycaemia and normalise body weight in mouse models of diabetes and obesity.. Methods We used the tamoxifen-inducible CreERT2-Lox system in mice to knockout Glut2 specifically in the kidneys (KsGlut2 KO) to establish the contribution of renal GLUT2 to systemic glucose homeostasis in health and in insulin-dependent as well as non-insulin-dependent diabetes. Bioprocessing:Article Title: Transcription factor c-Maf deletion improves streptozotocin-induced diabetic nephropathy by directly regulating Sglt2 and Glut2 Article Snippet: .. The samples were then blocked for 1 hour in PBS with 5% bovine serum albumin (BSA) and 10% goat serum, then incubated for 40 hours at 4°C with the following primary antibodies: anti–c-Maf rabbit monoclonal antibody (1:200; M00654; Boster Biological Technology), anti-Sglt2 (1:200; ab85626; Abcam), anti-Glut2 (1:200; 600-401-GN3; ROCKLAND), rabbit |