microarray (Broad Institute Inc)
90
Structured Review
Broad Institute Inc
microarray
Microarray, supplied by Broad Institute Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/genesifter%C2%AE+microarray+expression+analysis+software/microarray/pmc03383383-52-0-19
Average 90 stars, based on 1 article reviews
Microarray, supplied by Broad Institute Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/genesifter%C2%AE+microarray+expression+analysis+software/microarray/pmc03383383-52-0-19
Average 90 stars, based on 1 article reviews
microarray - by Bioz Stars,
2026-09
90/100 stars
Images
1) Product Images from "A chemical genetic approach identifies piperazine antipsychotics as promoters of CNS neurite growth on inhibitory substrates"
Article Title: A chemical genetic approach identifies piperazine antipsychotics as promoters of CNS neurite growth on inhibitory substrates
Journal: Molecular and Cellular Neurosciences
doi: 10.1016/j.mcn.2012.04.008
Figure Legend Snippet: Table I
Techniques Used: Microarray, Gene Expression
Related Articles
Microarray:Article Title: Depletion of dAKAP1–protein kinase A signaling islands from the outer mitochondrial membrane alters breast cancer cell metabolism and motility Article Snippet: 7 Pearson's correlation, calculated between each AKAP with quantified mRNA expression values and individual mesenchymal markers (36 total) in either breast cancer ( , A–D ) or any cancer etiology with >3 quantified cell lines ( Fig. S1, A and B ), was represented in a heat map using GraphPad Prism7 (GraphPad Software, La Jolla, CA). .. Clinical sample Article Title: Ascorbic Acid 2-Glucoside Stably Promotes the Primitiveness of Embryonic and Mesenchymal Stem Cells Through Ten-Eleven Translocation- and cAMP-Responsive Element-Binding Protein-1-Dependent Mechanisms. Article Snippet: Page 1 of 58 Antioxidants and Redox Signaling © Mary Ann Liebert, Inc. DOI: 10.1089/ars.2019.7743 1 A n ti o xi d an ts a n d R ed o x Si gn al in g A sc o rb ic a ci d 2 -g lu co si d e st ab ly p ro m o te s th e p ri m it iv e n es s o f e m b ry o n ic a n d m es en ch ym al s te m c el ls t h ro u gh T ET - an d C R EB 1 -d ep en d en t m ec h an is m s ( D O I: 1 0 .1 0 8 9 /a rs .2 0 1 9 .7 7 4 3 ) Th is p ap er h as b e e n p ee r- re vi e w ed a n d a cc ep te d f o r p u b lic at io n , b u t h as y et t o u n d er go c o p ye d it in g an d p ro o f co rr e ct io n . T h e fi n al p u b lis h ed v er si o n m ay d if fe r fr o m t h is p ro o f. Ascorbic acid 2-glucoside stably promotes the primitiveness of embryonic and mesenchymal stem cells through TET- and CREB1- dependent mechanisms Seungun Lee1,2,§, Jisun Lim1,2,§, Ji-Heon Lee3, Hyein Ju1,2, Jinbeom Heo1,2, YongHwan Kim1,2, Sujin Kim1,2, Hwan Yeul Yu1,2, Chae-Min Ryu1,2, So-Yeon Lee1, Jung-Min Han3, Yeon-Mok Oh4, Ho Lee5, Hyonchol Jang5, Tae-Joong Yoon6, Hee‐Sung Ahn7, Kyunggon Kim7, Hwa- Ryeon Kim8, Jae-Seok Roe8, Hyung-Min Chung3, Jaekyoung Son1,*, Jong Soo Kim3,*, Dong- Myung Shin1,2,* 1Department of Biomedical Sciences, Asan Medical Center, University of Ulsan College of Medicine, Seoul, 05505, Korea 2Department of Physiology, University of Ulsan College of Medicine, Seoul, 05505, Korea 3Department of Stem Cell Biology, School of Medicine, Konkuk University, Seoul, 05029, Korea 4Department of Pulmonary and Critical Care Medicine, Asan Medical Center, University of Ulsan College of Medicine, Seoul, 05505, Korea 5Research Institute, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Gyeonggi, 10408, Korea 6JJ.. 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We downloaded normalized Article Title: Transcriptomic and metabolomic profiling of ionic liquid stimuli unveils enhanced secondary metabolism in Aspergillus nidulans Article Snippet: .. Annotation of all genes represented on the Article Title: Tumour-specific Causal Inference Discovers Distinct Disease Mechanisms Underlying Cancer Subtypes Article Snippet: .. For GBM, the Article Title: YAP1 enhances NF-κB-dependent and independent effects on clock-mediated unfolded protein responses and autophagy in sarcoma Article Snippet: .. Fig. 6 YAP1 loss alters sarcoma cell metabolism and initiates differentiation a RNA sequencing:Article Title: Depletion of dAKAP1–protein kinase A signaling islands from the outer mitochondrial membrane alters breast cancer cell metabolism and motility Article Snippet: 7 Pearson's correlation, calculated between each AKAP with quantified mRNA expression values and individual mesenchymal markers (36 total) in either breast cancer ( , A–D ) or any cancer etiology with >3 quantified cell lines ( Fig. S1, A and B ), was represented in a heat map using GraphPad Prism7 (GraphPad Software, La Jolla, CA). .. Clinical sample Expressing:Article Title: Depletion of dAKAP1–protein kinase A signaling islands from the outer mitochondrial membrane alters breast cancer cell metabolism and motility Article Snippet: 7 Pearson's correlation, calculated between each AKAP with quantified mRNA expression values and individual mesenchymal markers (36 total) in either breast cancer ( , A–D ) or any cancer etiology with >3 quantified cell lines ( Fig. S1, A and B ), was represented in a heat map using GraphPad Prism7 (GraphPad Software, La Jolla, CA). .. Clinical sample Article Title: YAP1 enhances NF-κB-dependent and independent effects on clock-mediated unfolded protein responses and autophagy in sarcoma Article Snippet: .. Fig. 6 YAP1 loss alters sarcoma cell metabolism and initiates differentiation a Functional Assay:Article Title: Ascorbic Acid 2-Glucoside Stably Promotes the Primitiveness of Embryonic and Mesenchymal Stem Cells Through Ten-Eleven Translocation- and cAMP-Responsive Element-Binding Protein-1-Dependent Mechanisms. Article Snippet: Page 1 of 58 Antioxidants and Redox Signaling © Mary Ann Liebert, Inc. DOI: 10.1089/ars.2019.7743 1 A n ti o xi d an ts a n d R ed o x Si gn al in g A sc o rb ic a ci d 2 -g lu co si d e st ab ly p ro m o te s th e p ri m it iv e n es s o f e m b ry o n ic a n d m es en ch ym al s te m c el ls t h ro u gh T ET - an d C R EB 1 -d ep en d en t m ec h an is m s ( D O I: 1 0 .1 0 8 9 /a rs .2 0 1 9 .7 7 4 3 ) Th is p ap er h as b e e n p ee r- re vi e w ed a n d a cc ep te d f o r p u b lic at io n , b u t h as y et t o u n d er go c o p ye d it in g an d p ro o f co rr e ct io n . T h e fi n al p u b lis h ed v er si o n m ay d if fe r fr o m t h is p ro o f. Ascorbic acid 2-glucoside stably promotes the primitiveness of embryonic and mesenchymal stem cells through TET- and CREB1- dependent mechanisms Seungun Lee1,2,§, Jisun Lim1,2,§, Ji-Heon Lee3, Hyein Ju1,2, Jinbeom Heo1,2, YongHwan Kim1,2, Sujin Kim1,2, Hwan Yeul Yu1,2, Chae-Min Ryu1,2, So-Yeon Lee1, Jung-Min Han3, Yeon-Mok Oh4, Ho Lee5, Hyonchol Jang5, Tae-Joong Yoon6, Hee‐Sung Ahn7, Kyunggon Kim7, Hwa- Ryeon Kim8, Jae-Seok Roe8, Hyung-Min Chung3, Jaekyoung Son1,*, Jong Soo Kim3,*, Dong- Myung Shin1,2,* 1Department of Biomedical Sciences, Asan Medical Center, University of Ulsan College of Medicine, Seoul, 05505, Korea 2Department of Physiology, University of Ulsan College of Medicine, Seoul, 05505, Korea 3Department of Stem Cell Biology, School of Medicine, Konkuk University, Seoul, 05029, Korea 4Department of Pulmonary and Critical Care Medicine, Asan Medical Center, University of Ulsan College of Medicine, Seoul, 05505, Korea 5Research Institute, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Gyeonggi, 10408, Korea 6JJ.. MADIN Inc, Seoul, 08512, Korea 7 Department of Convergence Medicine, Asan Medical Center, University of Ulsan College of Medicine, Seoul, 05505, Korea 8Department of Biochemistry, College of Life Science and Biotechnology, Yonsei University, Seoul 03722, Korea §These authors equally contributed to this work.. Short title: Effect of AA2G on preserving primitive stem cells *Corresponding authors: Dong-Myung Shin, Ph.D. Department of Biomedical Sciences, Asan Medical Center, University of Ulsan College of Medicine, Pungnap-2 dong, Songpa-gu, Seoul, 05505, Korea Tel: 82-2-3010-2086; Fax: 82-2-3010-8493; Email: d0shin03@amc.seoul.kr D ow nl oa de d by U cs f L ib ra ry U ni ve rs ity o f C al if or ni a Sa n Fr an ci sc o fr om w w w .li eb er tp ub .c om a t 1 1/ 08 /1 9. Software:Article Title: Ascorbic Acid 2-Glucoside Stably Promotes the Primitiveness of Embryonic and Mesenchymal Stem Cells Through Ten-Eleven Translocation- and cAMP-Responsive Element-Binding Protein-1-Dependent Mechanisms. Article Snippet: Page 1 of 58 Antioxidants and Redox Signaling © Mary Ann Liebert, Inc. DOI: 10.1089/ars.2019.7743 1 A n ti o xi d an ts a n d R ed o x Si gn al in g A sc o rb ic a ci d 2 -g lu co si d e st ab ly p ro m o te s th e p ri m it iv e n es s o f e m b ry o n ic a n d m es en ch ym al s te m c el ls t h ro u gh T ET - an d C R EB 1 -d ep en d en t m ec h an is m s ( D O I: 1 0 .1 0 8 9 /a rs .2 0 1 9 .7 7 4 3 ) Th is p ap er h as b e e n p ee r- re vi e w ed a n d a cc ep te d f o r p u b lic at io n , b u t h as y et t o u n d er go c o p ye d it in g an d p ro o f co rr e ct io n . T h e fi n al p u b lis h ed v er si o n m ay d if fe r fr o m t h is p ro o f. Ascorbic acid 2-glucoside stably promotes the primitiveness of embryonic and mesenchymal stem cells through TET- and CREB1- dependent mechanisms Seungun Lee1,2,§, Jisun Lim1,2,§, Ji-Heon Lee3, Hyein Ju1,2, Jinbeom Heo1,2, YongHwan Kim1,2, Sujin Kim1,2, Hwan Yeul Yu1,2, Chae-Min Ryu1,2, So-Yeon Lee1, Jung-Min Han3, Yeon-Mok Oh4, Ho Lee5, Hyonchol Jang5, Tae-Joong Yoon6, Hee‐Sung Ahn7, Kyunggon Kim7, Hwa- Ryeon Kim8, Jae-Seok Roe8, Hyung-Min Chung3, Jaekyoung Son1,*, Jong Soo Kim3,*, Dong- Myung Shin1,2,* 1Department of Biomedical Sciences, Asan Medical Center, University of Ulsan College of Medicine, Seoul, 05505, Korea 2Department of Physiology, University of Ulsan College of Medicine, Seoul, 05505, Korea 3Department of Stem Cell Biology, School of Medicine, Konkuk University, Seoul, 05029, Korea 4Department of Pulmonary and Critical Care Medicine, Asan Medical Center, University of Ulsan College of Medicine, Seoul, 05505, Korea 5Research Institute, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Gyeonggi, 10408, Korea 6JJ.. MADIN Inc, Seoul, 08512, Korea 7 Department of Convergence Medicine, Asan Medical Center, University of Ulsan College of Medicine, Seoul, 05505, Korea 8Department of Biochemistry, College of Life Science and Biotechnology, Yonsei University, Seoul 03722, Korea §These authors equally contributed to this work.. Short title: Effect of AA2G on preserving primitive stem cells *Corresponding authors: Dong-Myung Shin, Ph.D. Department of Biomedical Sciences, Asan Medical Center, University of Ulsan College of Medicine, Pungnap-2 dong, Songpa-gu, Seoul, 05505, Korea Tel: 82-2-3010-2086; Fax: 82-2-3010-8493; Email: d0shin03@amc.seoul.kr D ow nl oa de d by U cs f L ib ra ry U ni ve rs ity o f C al if or ni a Sa n Fr an ci sc o fr om w w w .li eb er tp ub .c om a t 1 1/ 08 /1 9. Gene Expression:Article Title: Combined Mapping of Multiple clUsteriNg ALgorithms (COMMUNAL): A Robust Method for Selection of Cluster Number, K Article Snippet: .. We downloaded normalized Article Title: Tumour-specific Causal Inference Discovers Distinct Disease Mechanisms Underlying Cancer Subtypes Article Snippet: .. For GBM, the Article Title: YAP1 enhances NF-κB-dependent and independent effects on clock-mediated unfolded protein responses and autophagy in sarcoma Article Snippet: .. Fig. 6 YAP1 loss alters sarcoma cell metabolism and initiates differentiation a other:Article Title: Depletion of dAKAP1–protein kinase A signaling islands from the outer mitochondrial membrane alters breast cancer cell metabolism and motility Article Snippet: Microarray, RNAseq, and protein expression data in tissue samples ( , G and H , and S1, C and D ) were downloaded from the Broad Institute–associated browser ( http://prot-shiny-vm.broadinstitute.org:3838/BC2016/ ) 7 ( 32 ). Article Title: Epithelial EP4 plays an essential role in maintaining homeostasis in colon. Article Snippet: Gene expression profiling revealed the potential inflammatory status of EP4 cKO colons. (A) GSEA of transcriptomic data from microarray on EP4 cKO versus Villin-Cre colons. (B) KEGG pathways positively enriched in EP4 cKO mouse colons analyzed with DAVID. (C) Biological processes (GO) positively enriched in EP4 cKO mouse colons analyzed with DAVID. (D) Biological processes (GO) negatively enriched in EP4 cKO mouse colons analyzed with DAVID. (E) GSEA plots of enrichment of the indicated gene signatures in EP4 cKO versus Villin-Cre colons using the “Hallmarks” compilation from Molecular Signature Database (MSigDB, Broad Institute). Quantitative RT-PCR:Article Title: YAP1 enhances NF-κB-dependent and independent effects on clock-mediated unfolded protein responses and autophagy in sarcoma Article Snippet: .. Fig. 6 YAP1 loss alters sarcoma cell metabolism and initiates differentiation a Biomarker Discovery:Article Title: YAP1 enhances NF-κB-dependent and independent effects on clock-mediated unfolded protein responses and autophagy in sarcoma Article Snippet: .. Fig. 6 YAP1 loss alters sarcoma cell metabolism and initiates differentiation a Western Blot:Article Title: YAP1 enhances NF-κB-dependent and independent effects on clock-mediated unfolded protein responses and autophagy in sarcoma Article Snippet: .. Fig. 6 YAP1 loss alters sarcoma cell metabolism and initiates differentiation a Rescue Assay:Article Title: YAP1 enhances NF-κB-dependent and independent effects on clock-mediated unfolded protein responses and autophagy in sarcoma Article Snippet: .. Fig. 6 YAP1 loss alters sarcoma cell metabolism and initiates differentiation a shRNA:Article Title: YAP1 enhances NF-κB-dependent and independent effects on clock-mediated unfolded protein responses and autophagy in sarcoma Article Snippet: .. Fig. 6 YAP1 loss alters sarcoma cell metabolism and initiates differentiation a |
