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Dojindo Labs
chemical compound
Chemical Compound, supplied by Dojindo Labs, used in various techniques. Bioz Stars score: 99/100, based on 1953 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/compounds+12/FerroOrange/pmc12875612-32-0-6
Average 99 stars, based on 1953 article reviews
Chemical Compound, supplied by Dojindo Labs, used in various techniques. Bioz Stars score: 99/100, based on 1953 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/compounds+12/FerroOrange/pmc12875612-32-0-6
Average 99 stars, based on 1953 article reviews
chemical compound - by Bioz Stars,
2026-11
99/100 stars
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Incubation:Article Title: Polystyrene nanoplastics trigger ferroptosis-dependent liver injury caused by ACSL4 overaccumulation. Article Snippet: For cellular uptake observation, cells were exposed to fluorescent PS-NPs (BaseLine, China) for 24 h, and cell membranes were labeled with DiI (Beyotime). .. To detect intracellular Fe2+, lipid peroxidation and lysosomal pH, cells were treated for 72 h and subsequently incubated with Article Title: GPR68 Inhibition Induces Ferroptosis in Atypical Teratoid Rhabdoid Tumors Article Snippet: .. DMSO or OGM was then added to the cells and plates were incubated for 24 h. Cells were then stained with Article Title: Angiotensin-(1-7) attenuates collagen-induced arthritis and suppresses ferroptosis in THP-1-derived macrophages: involvement of p53/SLC7A11 signaling. Article Snippet: 1 Department of Rheumatology and Immunology, The Second Affiliated Hospital of Chongqing Medical University, No. 76 Linjiang Road, Yuzhong District, Chongqing 400010, People’s Republic of China Abstract Rheumatoid arthritis (RA) is characterized by persistent synovial inflammation and progressive joint destruction, while ferroptosis has emerged as a potential contributor to inflammatory tissue injury.. This study investigated whether angiotensin-(1–7) [Ang-(1–7)] attenuates arthritis by regulating ferroptosis and explored the involvement of p53/solute carrier family 7 member 11 (SLC7A11) signaling.. Bioinformatic analysis of the GSE55235 dataset was performed to identify ferroptosis-related differentially expressed genes in RA. Analysis:Article Title: NFS1 as a Metabolic Vulnerability in Acute Myeloid Leukemia Through Iron Dysregulation and STAT1/IFIT2-Associated Signaling. Article Snippet: Acute myeloid leukemia (AML) is a metabolically heterogeneous malignancy with poor outcomes and limited durable therapeutic options.. We investigated NFS1, a key enzyme involved in iron‐sulfur cluster biogenesis, as a potential metabolic vulnerability in AML.. NFS1 was overexpressed in AML samples, and higher expression was associated with inferior overall survival in the TCGA‐LAML cohort. ROS Assay:Article Title: NFS1 as a Metabolic Vulnerability in Acute Myeloid Leukemia Through Iron Dysregulation and STAT1/IFIT2-Associated Signaling. Article Snippet: Acute myeloid leukemia (AML) is a metabolically heterogeneous malignancy with poor outcomes and limited durable therapeutic options.. We investigated NFS1, a key enzyme involved in iron‐sulfur cluster biogenesis, as a potential metabolic vulnerability in AML.. NFS1 was overexpressed in AML samples, and higher expression was associated with inferior overall survival in the TCGA‐LAML cohort. Fluorescence:Article Title: IRP2 contributes to COPD-associated hypoxic epithelial injury through regulation of Sestrin2-related stress responses and ferroptosis Article Snippet: Cells were incubated with 10 μM DCFH-DA at 37 °C for 15 min in the dark, washed three times with serum-free medium, and immediately imaged under a fluorescence microscope (Leica, Germany) at excitation/emission wavelengths of 488/525 nm. .. Staining:Article Title: GPR68 Inhibition Induces Ferroptosis in Atypical Teratoid Rhabdoid Tumors Article Snippet: .. DMSO or OGM was then added to the cells and plates were incubated for 24 h. Cells were then stained with Incubator:Article Title: Angiotensin-(1-7) attenuates collagen-induced arthritis and suppresses ferroptosis in THP-1-derived macrophages: involvement of p53/SLC7A11 signaling. Article Snippet: 1 Department of Rheumatology and Immunology, The Second Affiliated Hospital of Chongqing Medical University, No. 76 Linjiang Road, Yuzhong District, Chongqing 400010, People’s Republic of China Abstract Rheumatoid arthritis (RA) is characterized by persistent synovial inflammation and progressive joint destruction, while ferroptosis has emerged as a potential contributor to inflammatory tissue injury.. This study investigated whether angiotensin-(1–7) [Ang-(1–7)] attenuates arthritis by regulating ferroptosis and explored the involvement of p53/solute carrier family 7 member 11 (SLC7A11) signaling.. Bioinformatic analysis of the GSE55235 dataset was performed to identify ferroptosis-related differentially expressed genes in RA. |
