brca2 (Santa Cruz Biotechnology)
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90
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Santa Cruz Biotechnology
brca2
Brca2, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 90/100, based on 14 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/brca2/BRCA2+siRNA/pm20127002-36-0-5
Average 90 stars, based on 14 article reviews
Brca2, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 90/100, based on 14 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/brca2/BRCA2+siRNA/pm20127002-36-0-5
Average 90 stars, based on 14 article reviews
brca2 - by Bioz Stars,
2026-10
90/100 stars
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Transfection:Article Title: Complementary genetic screens identify the E3 ubiquitin ligase CBLC, as a modifier of PARP inhibitor sensitivity. Article Snippet: .. Each experiment was repeated twice. cell-based assays Cell lines were transfected with SMARTpool siRNAs (Dharmacon, GE Healthcare) targeting Article Title: Inhibition of c‐MET increases the antitumour activity of PARP inhibitors in gastric cancer models Article Snippet: .. For the siRNA transfection experiments, we use siRNA for stable knockdown of c‐MET (sc‐35924), BRCA1 (sc‐29219) and Article Title: Silencing of BRCA2 decreases anoikis and its heterologous expression sensitizes yeast cells to acetic acid-induced programmed cell death. Article Snippet: Adhesion of normal epithelial cells to the extracellular matrix (ECM) is essential for survival.. Cell detachment from ECM induces a specific form of programmed cell death (PCD) termed anoikis.. BRCA2, a tumor suppressor gene whose mutations confer predisposition to cancer, has been implicated in the regulation of DNA repair, transcription, cell proliferation, and apoptosis. other:Article Title: Profilin-1 regulates DNA replication forks in a context-dependent fashion by interacting with SNF2H and BOD1L. Article Snippet: Small interfering RNAs targeting Article Title: Profilin-1 regulates DNA replication forks in a context-dependent fashion by interacting with SNF2H and BOD1L Article Snippet: Small interfering RNAs targeting Control:Article Title: Targeting HR Repair as a Synthetic Lethal Approach to Increase DNA Damage Sensitivity by a RAD52 Inhibitor in BRCA2-Deficient Cancer Cells Article Snippet: .. For transient depletion of BRCA2 in MCF7 cells, we purchased Article Title: Inhibition of c‐MET increases the antitumour activity of PARP inhibitors in gastric cancer models Article Snippet: .. For the siRNA transfection experiments, we use siRNA for stable knockdown of c‐MET (sc‐35924), BRCA1 (sc‐29219) and Knockdown:Article Title: Inhibition of c‐MET increases the antitumour activity of PARP inhibitors in gastric cancer models Article Snippet: .. For the siRNA transfection experiments, we use siRNA for stable knockdown of c‐MET (sc‐35924), BRCA1 (sc‐29219) and Over Expression:Article Title: Interaction of FUS and HDAC1 Regulates DNA Damage Response and Repair in Neurons Article Snippet: For LacR constructs, FUS and HDAC1 were cloned into Cherry-LacRep (Addgene plasmid 18985). .. FUS overexpression constructs were generated by fusing mCherry to the N-terminal of FUS and subcloning this into the Lv-CAG lentiviral vector. siRNAs siRNAs against FUS (sc-40563), Construct:Article Title: Interaction of FUS and HDAC1 Regulates DNA Damage Response and Repair in Neurons Article Snippet: For LacR constructs, FUS and HDAC1 were cloned into Cherry-LacRep (Addgene plasmid 18985). .. FUS overexpression constructs were generated by fusing mCherry to the N-terminal of FUS and subcloning this into the Lv-CAG lentiviral vector. siRNAs siRNAs against FUS (sc-40563), Generated:Article Title: Interaction of FUS and HDAC1 Regulates DNA Damage Response and Repair in Neurons Article Snippet: For LacR constructs, FUS and HDAC1 were cloned into Cherry-LacRep (Addgene plasmid 18985). .. FUS overexpression constructs were generated by fusing mCherry to the N-terminal of FUS and subcloning this into the Lv-CAG lentiviral vector. siRNAs siRNAs against FUS (sc-40563), Subcloning:Article Title: Interaction of FUS and HDAC1 Regulates DNA Damage Response and Repair in Neurons Article Snippet: For LacR constructs, FUS and HDAC1 were cloned into Cherry-LacRep (Addgene plasmid 18985). .. FUS overexpression constructs were generated by fusing mCherry to the N-terminal of FUS and subcloning this into the Lv-CAG lentiviral vector. siRNAs siRNAs against FUS (sc-40563), shRNA:Article Title: Silencing of BRCA2 decreases anoikis and its heterologous expression sensitizes yeast cells to acetic acid-induced programmed cell death. Article Snippet: Adhesion of normal epithelial cells to the extracellular matrix (ECM) is essential for survival.. Cell detachment from ECM induces a specific form of programmed cell death (PCD) termed anoikis.. BRCA2, a tumor suppressor gene whose mutations confer predisposition to cancer, has been implicated in the regulation of DNA repair, transcription, cell proliferation, and apoptosis. Expressing:Article Title: Silencing of BRCA2 decreases anoikis and its heterologous expression sensitizes yeast cells to acetic acid-induced programmed cell death. Article Snippet: Adhesion of normal epithelial cells to the extracellular matrix (ECM) is essential for survival.. Cell detachment from ECM induces a specific form of programmed cell death (PCD) termed anoikis.. BRCA2, a tumor suppressor gene whose mutations confer predisposition to cancer, has been implicated in the regulation of DNA repair, transcription, cell proliferation, and apoptosis. Western Blot:Article Title: Silencing of BRCA2 decreases anoikis and its heterologous expression sensitizes yeast cells to acetic acid-induced programmed cell death. Article Snippet: Adhesion of normal epithelial cells to the extracellular matrix (ECM) is essential for survival.. Cell detachment from ECM induces a specific form of programmed cell death (PCD) termed anoikis.. BRCA2, a tumor suppressor gene whose mutations confer predisposition to cancer, has been implicated in the regulation of DNA repair, transcription, cell proliferation, and apoptosis. |
