antifade solution (Oncor Inc)
90
Structured Review
Oncor Inc
antifade solution
Antifade Solution, supplied by Oncor Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/antifade+solution/antifade+solution/pmc02816331-33-14-16
Average 90 stars, based on 1 article reviews
Antifade Solution, supplied by Oncor Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/antifade+solution/antifade+solution/pmc02816331-33-14-16
Average 90 stars, based on 1 article reviews
antifade solution - by Bioz Stars,
2026-09
90/100 stars
Images
Related Articles
Concentration Assay:Article Title: Genetic Alterations in Intrahepatic Cholangiocarcinoma as revealed by Degenerate Oligonucleotide Primed PCR-Comparative Genomic Hybridization Article Snippet: .. Metaphase slides were counterstained with DAPI at a concentration of 0.1 μg/mL in an Article Title: Comparative Genomic Hybridization: Synchronous Occurrence of Focal Nodular Hyperplasia and Hepatocellular Carcinoma in the Same Liver Is Not Based on Common Chromosomal Aberrations Article Snippet: .. Slides were counterstained with 4',6 diamidino-2-phenylindole-dihydrochloride at a concentration of 0.1 μg/mL in an Article Title: Chromosomal aberrations in Korean nonsmall cell lung carcinomas: degenerate oligonucleotide primed polymerase chain reaction comparative genomic hybridization studies. Article Snippet: Chromosomal aberrations were investigated in 48 Korean nonsmall cell carcinomas of the lung (NSCLC), by degenerate oligonucleotide primed polymerase chain reaction comparative genomic hybridization.. These included 16 adenocarcinomas, 27 squamous cell carcinomas (SCCs), and 5 large-cell carcinomas.. The common sites of copy number increases were 3q26~qter (23 cases, 48%); 8q23~qter (46%); 20q13.1 (42%); 1q42~qter (38%); 3q25 (38%); 21q22 (38%); and 22q13 (38%). Light Microscopy:Article Title: Effect of four missense mutations in the factor XIII A-subunit gene on protein stability: studies with recombinant proteins. Article Snippet: Four missense mutations in the factor XIII A-subunit gene, Arg260Leu, Ala318Val, Thr398Asn and Gly210Arg, were previously reported by us in patients with severe factor XIII deficiency.. The objective of our study was to discern the effect of all four mutations on the stability and intracellular localization of the factor XIII A-subunit by their expression in COS cells.. In-vitro mutagenesis, transient expression of the mutants in COS cells and subsequent pulse–chase analyses were carried out. Laser-Scanning Microscopy:Article Title: Glanzmann thrombasthenia associated with a 21-amino acid deletion (Leu817-Gln837) in glycoprotein IIb due to abnormal splicing in exon 25. Article Snippet: We report a novel genetic defect in a Japanese patient with type I Glanzmann thrombasthenia.. The glycoprotein (GP) IIb complementary DNA (cDNA) from platelet messenger RNA had a 63–base pair deletion in the 5 boundary of exon 25, resulting in an in-frame deletion of 21 amino acid residues (Leu817-Gln837) in the calf-2 domain.. The deleted region was present in the genomic DNA, but the splice acceptor site (AG) of exon 25 was mutated to AC, leading to the use of an AG sequence in the middle of exon 25 as an abnormal cryptic splice acceptor site. |