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The effect of submaximally dosed semaglutide therapy on insulin levels and adipocytokines in patients with obesity. <t>INS—insulin;</t> <t>LEPT—leptin;</t> <t>RETN—resistin;</t> *— p < 0.05; **— p < 0.01; ns—not significant.
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The effect of submaximally dosed semaglutide therapy on insulin levels and adipocytokines in patients with obesity. <t>INS—insulin;</t> <t>LEPT—leptin;</t> <t>RETN—resistin;</t> *— p < 0.05; **— p < 0.01; ns—not significant.
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The effect of submaximally dosed semaglutide therapy on insulin levels and adipocytokines in patients with obesity. <t>INS—insulin;</t> <t>LEPT—leptin;</t> <t>RETN—resistin;</t> *— p < 0.05; **— p < 0.01; ns—not significant.
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Comparison of cell-cell communication analysis of γδ T cells in single-cell mRNA sequencing between the pediatric sepsis (sepsis) group and HC group. a Comparison of <t>resistin</t> signaling. b Comparison of interactions between cell surface receptors and cell ligands in resistin signaling. c Comparison of mRNA expression levels of cell surface receptors and cell ligand genes in all cell subtypes during resistin signaling. d Comparison of RNA expression levels of CAP1 gene in γδ T cells between sepsis ( n = 5) and HC samples ( n = 5). e The proportion <t>of</t> <t>IL-17A-positive</t> γδ T cells was detected by flow cytometry in resistin stimulation ( n = 5) and control ( n = 5). f Comparison of the proportion of IL-17A-positive γδ T cells between resistin stimulation ( n = 6) and control ( n = 6) (* p < 0.05).
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Comparison of cell-cell communication analysis of γδ T cells in single-cell mRNA sequencing between the pediatric sepsis (sepsis) group and HC group. a Comparison of <t>resistin</t> signaling. b Comparison of interactions between cell surface receptors and cell ligands in resistin signaling. c Comparison of mRNA expression levels of cell surface receptors and cell ligand genes in all cell subtypes during resistin signaling. d Comparison of RNA expression levels of CAP1 gene in γδ T cells between sepsis ( n = 5) and HC samples ( n = 5). e The proportion <t>of</t> <t>IL-17A-positive</t> γδ T cells was detected by flow cytometry in resistin stimulation ( n = 5) and control ( n = 5). f Comparison of the proportion of IL-17A-positive γδ T cells between resistin stimulation ( n = 6) and control ( n = 6) (* p < 0.05).
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Comparison of cell-cell communication analysis of γδ T cells in single-cell mRNA sequencing between the pediatric sepsis (sepsis) group and HC group. a Comparison of <t>resistin</t> signaling. b Comparison of interactions between cell surface receptors and cell ligands in resistin signaling. c Comparison of mRNA expression levels of cell surface receptors and cell ligand genes in all cell subtypes during resistin signaling. d Comparison of RNA expression levels of CAP1 gene in γδ T cells between sepsis ( n = 5) and HC samples ( n = 5). e The proportion <t>of</t> <t>IL-17A-positive</t> γδ T cells was detected by flow cytometry in resistin stimulation ( n = 5) and control ( n = 5). f Comparison of the proportion of IL-17A-positive γδ T cells between resistin stimulation ( n = 6) and control ( n = 6) (* p < 0.05).
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The effect of submaximally dosed semaglutide therapy on insulin levels and adipocytokines in patients with obesity. INS—insulin; LEPT—leptin; RETN—resistin; *— p < 0.05; **— p < 0.01; ns—not significant.

Journal: Pharmaceuticals

Article Title: Effect of Submaximal Doses of Semaglutide in Patients with Obesity on Metabolic Profile and Serum Levels of Adipocytokines

doi: 10.3390/ph18091364

Figure Lengend Snippet: The effect of submaximally dosed semaglutide therapy on insulin levels and adipocytokines in patients with obesity. INS—insulin; LEPT—leptin; RETN—resistin; *— p < 0.05; **— p < 0.01; ns—not significant.

Article Snippet: The Metabolic Syndrome Array 1 panel including eight biomarkers—c-peptide, ferritin, interleukin-6 (IL-6), resistin, insulin, tumor necrosis factor α (TNF-α), interleukin-1α (IL-1α), leptin and plasminogen activator inhibitor-1 (PAI-1)—was subsequently measured and quantified using biochip array technology (Evidence Investigator, Randox, Crumlin, UK), in accordance with the manufacturer’s protocol.

Techniques:

Comparison of cell-cell communication analysis of γδ T cells in single-cell mRNA sequencing between the pediatric sepsis (sepsis) group and HC group. a Comparison of resistin signaling. b Comparison of interactions between cell surface receptors and cell ligands in resistin signaling. c Comparison of mRNA expression levels of cell surface receptors and cell ligand genes in all cell subtypes during resistin signaling. d Comparison of RNA expression levels of CAP1 gene in γδ T cells between sepsis ( n = 5) and HC samples ( n = 5). e The proportion of IL-17A-positive γδ T cells was detected by flow cytometry in resistin stimulation ( n = 5) and control ( n = 5). f Comparison of the proportion of IL-17A-positive γδ T cells between resistin stimulation ( n = 6) and control ( n = 6) (* p < 0.05).

Journal: Journal of Innate Immunity

Article Title: Single-Cell RNA Sequencing in Pediatric Sepsis: γδ T Cell Exhibits a Differentiation to γδT17 Subtype along with Significantly Enhanced Cell Communication with Neutrophils

doi: 10.1159/000547934

Figure Lengend Snippet: Comparison of cell-cell communication analysis of γδ T cells in single-cell mRNA sequencing between the pediatric sepsis (sepsis) group and HC group. a Comparison of resistin signaling. b Comparison of interactions between cell surface receptors and cell ligands in resistin signaling. c Comparison of mRNA expression levels of cell surface receptors and cell ligand genes in all cell subtypes during resistin signaling. d Comparison of RNA expression levels of CAP1 gene in γδ T cells between sepsis ( n = 5) and HC samples ( n = 5). e The proportion of IL-17A-positive γδ T cells was detected by flow cytometry in resistin stimulation ( n = 5) and control ( n = 5). f Comparison of the proportion of IL-17A-positive γδ T cells between resistin stimulation ( n = 6) and control ( n = 6) (* p < 0.05).

Article Snippet: For IL-17A induction, PBMCs were cultured with resistin (100 ng/mL, MCE) and brefeldin A (10 μg/mL, Sigma-Aldrich) for 4 h, followed by flow cytometry.

Techniques: Comparison, Sequencing, Expressing, RNA Expression, Flow Cytometry, Control