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dlgap5 antibody  (Affinity Biosciences)
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Analysis of cell trajectory, subpopulation abundance, and gene expression in glioma. A , B Cell trajectory analysis using the CytoTRACE algorithm. The inferred pseudotime map visualizes the differentiation potential of glioma-related subpopulations, with darker shades indicating less differentiated (more stem-like) cells and lighter shades indicating more differentiated states. The trajectory highlights the pivotal position of oligodendrocytes at a branching differentiation point, suggesting their potential role in glioma progression. C Abundance proportions of different cell subpopulations in control and glioma groups, with notable increases in T cells, proliferating cancer cells, and inhibitory neurons, and reductions in oligodendrocytes, astrocytes, and tumor-associated macrophages (TAM) in the glioma group. D Expression patterns of specific genes (CHEK1, <t>DLGAP5,</t> MKI67, and TOP2A) across different cell subpopulations, with high expression observed in proliferating cancer cells
Dlgap5 Antibody, supplied by Affinity Biosciences, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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dlgap5  (Proteintech)
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Analysis of cell trajectory, subpopulation abundance, and gene expression in glioma. A , B Cell trajectory analysis using the CytoTRACE algorithm. The inferred pseudotime map visualizes the differentiation potential of glioma-related subpopulations, with darker shades indicating less differentiated (more stem-like) cells and lighter shades indicating more differentiated states. The trajectory highlights the pivotal position of oligodendrocytes at a branching differentiation point, suggesting their potential role in glioma progression. C Abundance proportions of different cell subpopulations in control and glioma groups, with notable increases in T cells, proliferating cancer cells, and inhibitory neurons, and reductions in oligodendrocytes, astrocytes, and tumor-associated macrophages (TAM) in the glioma group. D Expression patterns of specific genes (CHEK1, <t>DLGAP5,</t> MKI67, and TOP2A) across different cell subpopulations, with high expression observed in proliferating cancer cells
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Analysis of cell trajectory, subpopulation abundance, and gene expression in glioma. A , B Cell trajectory analysis using the CytoTRACE algorithm. The inferred pseudotime map visualizes the differentiation potential of glioma-related subpopulations, with darker shades indicating less differentiated (more stem-like) cells and lighter shades indicating more differentiated states. The trajectory highlights the pivotal position of oligodendrocytes at a branching differentiation point, suggesting their potential role in glioma progression. C Abundance proportions of different cell subpopulations in control and glioma groups, with notable increases in T cells, proliferating cancer cells, and inhibitory neurons, and reductions in oligodendrocytes, astrocytes, and tumor-associated macrophages (TAM) in the glioma group. D Expression patterns of specific genes (CHEK1, <t>DLGAP5,</t> MKI67, and TOP2A) across different cell subpopulations, with high expression observed in proliferating cancer cells
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Analysis of cell trajectory, subpopulation abundance, and gene expression in glioma. A , B Cell trajectory analysis using the CytoTRACE algorithm. The inferred pseudotime map visualizes the differentiation potential of glioma-related subpopulations, with darker shades indicating less differentiated (more stem-like) cells and lighter shades indicating more differentiated states. The trajectory highlights the pivotal position of oligodendrocytes at a branching differentiation point, suggesting their potential role in glioma progression. C Abundance proportions of different cell subpopulations in control and glioma groups, with notable increases in T cells, proliferating cancer cells, and inhibitory neurons, and reductions in oligodendrocytes, astrocytes, and tumor-associated macrophages (TAM) in the glioma group. D Expression patterns of specific genes (CHEK1, <t>DLGAP5,</t> MKI67, and TOP2A) across different cell subpopulations, with high expression observed in proliferating cancer cells
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dlgap5 plasmid  (OriGene)
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Analysis of cell trajectory, subpopulation abundance, and gene expression in glioma. A , B Cell trajectory analysis using the CytoTRACE algorithm. The inferred pseudotime map visualizes the differentiation potential of glioma-related subpopulations, with darker shades indicating less differentiated (more stem-like) cells and lighter shades indicating more differentiated states. The trajectory highlights the pivotal position of oligodendrocytes at a branching differentiation point, suggesting their potential role in glioma progression. C Abundance proportions of different cell subpopulations in control and glioma groups, with notable increases in T cells, proliferating cancer cells, and inhibitory neurons, and reductions in oligodendrocytes, astrocytes, and tumor-associated macrophages (TAM) in the glioma group. D Expression patterns of specific genes (CHEK1, <t>DLGAP5,</t> MKI67, and TOP2A) across different cell subpopulations, with high expression observed in proliferating cancer cells
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anti dlgap5  (Proteintech)
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Analysis of cell trajectory, subpopulation abundance, and gene expression in glioma. A , B Cell trajectory analysis using the CytoTRACE algorithm. The inferred pseudotime map visualizes the differentiation potential of glioma-related subpopulations, with darker shades indicating less differentiated (more stem-like) cells and lighter shades indicating more differentiated states. The trajectory highlights the pivotal position of oligodendrocytes at a branching differentiation point, suggesting their potential role in glioma progression. C Abundance proportions of different cell subpopulations in control and glioma groups, with notable increases in T cells, proliferating cancer cells, and inhibitory neurons, and reductions in oligodendrocytes, astrocytes, and tumor-associated macrophages (TAM) in the glioma group. D Expression patterns of specific genes (CHEK1, <t>DLGAP5,</t> MKI67, and TOP2A) across different cell subpopulations, with high expression observed in proliferating cancer cells
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dlgap5-specific small interfering rna (sirna)  (Shanghai GenePharma)
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Analysis of cell trajectory, subpopulation abundance, and gene expression in glioma. A , B Cell trajectory analysis using the CytoTRACE algorithm. The inferred pseudotime map visualizes the differentiation potential of glioma-related subpopulations, with darker shades indicating less differentiated (more stem-like) cells and lighter shades indicating more differentiated states. The trajectory highlights the pivotal position of oligodendrocytes at a branching differentiation point, suggesting their potential role in glioma progression. C Abundance proportions of different cell subpopulations in control and glioma groups, with notable increases in T cells, proliferating cancer cells, and inhibitory neurons, and reductions in oligodendrocytes, astrocytes, and tumor-associated macrophages (TAM) in the glioma group. D Expression patterns of specific genes (CHEK1, <t>DLGAP5,</t> MKI67, and TOP2A) across different cell subpopulations, with high expression observed in proliferating cancer cells
Dlgap5 Specific Small Interfering Rna (Sirna), supplied by Shanghai GenePharma, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Analysis of cell trajectory, subpopulation abundance, and gene expression in glioma. A , B Cell trajectory analysis using the CytoTRACE algorithm. The inferred pseudotime map visualizes the differentiation potential of glioma-related subpopulations, with darker shades indicating less differentiated (more stem-like) cells and lighter shades indicating more differentiated states. The trajectory highlights the pivotal position of oligodendrocytes at a branching differentiation point, suggesting their potential role in glioma progression. C Abundance proportions of different cell subpopulations in control and glioma groups, with notable increases in T cells, proliferating cancer cells, and inhibitory neurons, and reductions in oligodendrocytes, astrocytes, and tumor-associated macrophages (TAM) in the glioma group. D Expression patterns of specific genes (CHEK1, DLGAP5, MKI67, and TOP2A) across different cell subpopulations, with high expression observed in proliferating cancer cells

Journal: Discover Oncology

Article Title: Identification of key biomarkers and immune microenvironment features in gliomas based on single-cell analysis combined with bioinformatics

doi: 10.1007/s12672-025-03450-x

Figure Lengend Snippet: Analysis of cell trajectory, subpopulation abundance, and gene expression in glioma. A , B Cell trajectory analysis using the CytoTRACE algorithm. The inferred pseudotime map visualizes the differentiation potential of glioma-related subpopulations, with darker shades indicating less differentiated (more stem-like) cells and lighter shades indicating more differentiated states. The trajectory highlights the pivotal position of oligodendrocytes at a branching differentiation point, suggesting their potential role in glioma progression. C Abundance proportions of different cell subpopulations in control and glioma groups, with notable increases in T cells, proliferating cancer cells, and inhibitory neurons, and reductions in oligodendrocytes, astrocytes, and tumor-associated macrophages (TAM) in the glioma group. D Expression patterns of specific genes (CHEK1, DLGAP5, MKI67, and TOP2A) across different cell subpopulations, with high expression observed in proliferating cancer cells

Article Snippet: HRP-conjugated goat anti-rabbit IgG (S0001), TOP2A antibody (AF0793), DLGAP5 antibody (DF6920), and Chk1 antibody (AF6007) were purchased from Affinity Biosciences Ltd (USA).

Techniques: Gene Expression, Control, Expressing

Molecular docking analysis of quercetin (QU) with key target genes. A Chemical structure of QU. B–E Docking interactions between quercetin and the four target genes (CHEK1, DLGAP5, MKI67, and TOP2A), visualized using PyMOL software. All four interactions show binding energies below − 5 kJ/mol, indicating strong binding affinities

Journal: Discover Oncology

Article Title: Identification of key biomarkers and immune microenvironment features in gliomas based on single-cell analysis combined with bioinformatics

doi: 10.1007/s12672-025-03450-x

Figure Lengend Snippet: Molecular docking analysis of quercetin (QU) with key target genes. A Chemical structure of QU. B–E Docking interactions between quercetin and the four target genes (CHEK1, DLGAP5, MKI67, and TOP2A), visualized using PyMOL software. All four interactions show binding energies below − 5 kJ/mol, indicating strong binding affinities

Article Snippet: HRP-conjugated goat anti-rabbit IgG (S0001), TOP2A antibody (AF0793), DLGAP5 antibody (DF6920), and Chk1 antibody (AF6007) were purchased from Affinity Biosciences Ltd (USA).

Techniques: Software, Binding Assay

Effects of quercetin on cell viability and protein expression in glioma cells. A CCK-8 assay showing the viability of U87MG cells treated with increasing concentrations of quercetin (0, 10, 25, 50, 100, 150, 200, 250 µM) for 24 h. Quercetin significantly reduced cell viability in a dose-dependent manner, with approximately 50% inhibition observed at 100 µM. B Western blot analysis of CHEK1, DLGAP5, and TOP2A protein expression in the control group (HEB), model group (U87MG), and QU group (U87MG + 100 µM quercetin). C Quantitative analysis of protein band intensity. Quercetin treatment significantly downregulated the expression of target proteins compared to untreated U87MG cells. β-actin was used as the internal control. Data are presented as mean ± SD from three independent experiments. * p < 0.05, ** p < 0.01, *** p < 0.001

Journal: Discover Oncology

Article Title: Identification of key biomarkers and immune microenvironment features in gliomas based on single-cell analysis combined with bioinformatics

doi: 10.1007/s12672-025-03450-x

Figure Lengend Snippet: Effects of quercetin on cell viability and protein expression in glioma cells. A CCK-8 assay showing the viability of U87MG cells treated with increasing concentrations of quercetin (0, 10, 25, 50, 100, 150, 200, 250 µM) for 24 h. Quercetin significantly reduced cell viability in a dose-dependent manner, with approximately 50% inhibition observed at 100 µM. B Western blot analysis of CHEK1, DLGAP5, and TOP2A protein expression in the control group (HEB), model group (U87MG), and QU group (U87MG + 100 µM quercetin). C Quantitative analysis of protein band intensity. Quercetin treatment significantly downregulated the expression of target proteins compared to untreated U87MG cells. β-actin was used as the internal control. Data are presented as mean ± SD from three independent experiments. * p < 0.05, ** p < 0.01, *** p < 0.001

Article Snippet: HRP-conjugated goat anti-rabbit IgG (S0001), TOP2A antibody (AF0793), DLGAP5 antibody (DF6920), and Chk1 antibody (AF6007) were purchased from Affinity Biosciences Ltd (USA).

Techniques: Expressing, CCK-8 Assay, Inhibition, Western Blot, Control