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DNA methylation regulation of LGALS9 and its interaction with inflammatory cytokines in SD (A) LGALS9 locus plots reveal consistent genetic effects across SD GWAS, cis -mQTL, and cis -eQTL datasets ( p < 1 × 10−5). (B) Three-step SMR analysis demonstrated causal relationships between DNA methylation-mediated LGALS9 gene expressions and the onset of SD (P SMR multi < 0.05; HEIDI test p > 0.01, and Cochran’s Q p > 0.05). (C) Locus comparisons of LGALS9 gene expression and inflammatory cytokines <t>(CD5,</t> IL-10, and CST5) were performed via colocalization (PPH4 > 0.5). (D) ELISA validation of serum levels of LGALS9, CD5, IL-10, and CST5 in 16 patients with SD and 16 controls. Statistical analysis: SMR criteria as in . Data sources: eQTLGen and FinnGen R10. Colocalization: PPH4 > 0.5. ELISA: p = 0.0486 for LGALS9; ∗ p < 0.05, ∗∗ p < 0.01 versus controls. Boxplots show the median and IQR.
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Fig. 2. Lymphocytes T with <t>CD5+</t> receptor in rabbit males, females and total throughout the year. Legend: a – statistically significant difference between male and female, b – statistically significant difference between seasons (total),
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Fig. 2. Lymphocytes T with <t>CD5+</t> receptor in rabbit males, females and total throughout the year. Legend: a – statistically significant difference between male and female, b – statistically significant difference between seasons (total),
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DNA methylation regulation of LGALS9 and its interaction with inflammatory cytokines in SD (A) LGALS9 locus plots reveal consistent genetic effects across SD GWAS, cis -mQTL, and cis -eQTL datasets ( p < 1 × 10−5). (B) Three-step SMR analysis demonstrated causal relationships between DNA methylation-mediated LGALS9 gene expressions and the onset of SD (P SMR multi < 0.05; HEIDI test p > 0.01, and Cochran’s Q p > 0.05). (C) Locus comparisons of LGALS9 gene expression and inflammatory cytokines (CD5, IL-10, and CST5) were performed via colocalization (PPH4 > 0.5). (D) ELISA validation of serum levels of LGALS9, CD5, IL-10, and CST5 in 16 patients with SD and 16 controls. Statistical analysis: SMR criteria as in . Data sources: eQTLGen and FinnGen R10. Colocalization: PPH4 > 0.5. ELISA: p = 0.0486 for LGALS9; ∗ p < 0.05, ∗∗ p < 0.01 versus controls. Boxplots show the median and IQR.

Journal: iScience

Article Title: Interferon signaling gene expression, and DNA methylation interactions in Sjögren’s disease via mendelian randomization

doi: 10.1016/j.isci.2026.114950

Figure Lengend Snippet: DNA methylation regulation of LGALS9 and its interaction with inflammatory cytokines in SD (A) LGALS9 locus plots reveal consistent genetic effects across SD GWAS, cis -mQTL, and cis -eQTL datasets ( p < 1 × 10−5). (B) Three-step SMR analysis demonstrated causal relationships between DNA methylation-mediated LGALS9 gene expressions and the onset of SD (P SMR multi < 0.05; HEIDI test p > 0.01, and Cochran’s Q p > 0.05). (C) Locus comparisons of LGALS9 gene expression and inflammatory cytokines (CD5, IL-10, and CST5) were performed via colocalization (PPH4 > 0.5). (D) ELISA validation of serum levels of LGALS9, CD5, IL-10, and CST5 in 16 patients with SD and 16 controls. Statistical analysis: SMR criteria as in . Data sources: eQTLGen and FinnGen R10. Colocalization: PPH4 > 0.5. ELISA: p = 0.0486 for LGALS9; ∗ p < 0.05, ∗∗ p < 0.01 versus controls. Boxplots show the median and IQR.

Article Snippet: Galectin-9 regulates B cell activation by increasing colocalization with the inhibitory co-receptor CD5, thereby modulating TLR4 signal transduction.

Techniques: DNA Methylation Assay, Gene Expression, Enzyme-linked Immunosorbent Assay, Biomarker Discovery

Current studies using chimeric antigen receptor therapies in peripheral T-cell lymphoma.

Journal: Cancers

Article Title: Antibody-Based Therapies for Peripheral T-Cell Lymphoma

doi: 10.3390/cancers16203489

Figure Lengend Snippet: Current studies using chimeric antigen receptor therapies in peripheral T-cell lymphoma.

Article Snippet: NCT06420089 , CD5-deleted Chimeric Antigen Receptor Cells (Senza5 CART5) for T-Cell Non-Hodgkin Lymphoma (NHL) , 1 , Recruiting , Vittoria Biotherapeutics.

Techniques: CRISPR, Expressing, Modification, Injection

Fig. 2. Lymphocytes T with CD5+ receptor in rabbit males, females and total throughout the year. Legend: a – statistically significant difference between male and female, b – statistically significant difference between seasons (total),

Journal: Polish journal of veterinary sciences

Article Title: B and T lymphocytes in rabbits change according to the sex and throughout the year.

doi: 10.24425/pjvs.2020.132746

Figure Lengend Snippet: Fig. 2. Lymphocytes T with CD5+ receptor in rabbit males, females and total throughout the year. Legend: a – statistically significant difference between male and female, b – statistically significant difference between seasons (total),

Article Snippet: In rabbit blood, the percentage of B-cells with CD19+ receptor (Serotec, mouse anti rabbit IgM B cell marker, catalogue no. MCA812GA), T-cells with CD5+ receptor (Serotec, mouse anti rabbit CD5, catalogue no. MCA800), their subpopulations – T-cells with receptors CD4+ (Serotec, mouse anti rabbit CD4, catalogue no. MCA799G), CD8+ (Serotec, mouse anti rabbit CD8, catalogue no. MCA1576G) and CD25+ (Serotec, mouse anti rabbit CD8, catalogue no. MCA1119GA) was determined according to the method described by Deptuła et al. (1998).

Techniques: